Staphylococcus

Specific bacteriostatic activity against Escherichia coli (681, 896, 899), Staphylococcus aureus (681, 896), Cocci (900), Shigella dysenteriae (681), Salmonella ryphi (681), Proteus vulgaris (681), Pseudomonas aeruginosa (681), Streptococcus (889, 901, 902) and Pneumococcus (901-904). [Pg.152]

Other Microbial Systems. In addition to the systems described, gene cloning is routinely performed in several other bacterial strains including Streptoccocus Staphylococcus Brevibacterium BJdodobacter Comyebacterium Glucanobacter Acetobacter and Zanthomonas species. [Pg.249]

This can be a dangerous procedure due to the potential growth of food poisoning bacteria such as Staphylococcus aureus (31). This method of inoculation requites a very strict condition to assure the absence of not only bacteria associated with a health hazard but also those associated with product failure (proteolytic, greening, and gas-forming microorganisms). [Pg.33]

Proper refrigeration prevents the growth of some microorganisms, such as Salmonella and the production of toxins, such as Staphylococcus aureus. The growth of bacteria Tscherichia coli and Bacillus cereus is substantially checked by proper cooling and handling of milk. Table 14 Hsts diseases transmitted by cows to humans. Pasteurization is the best means of prevention. [Pg.364]

Tolypomycin Y (48) shows strong antibacterial activity against gram-positive bacteria and Neisseriagonorrheae. When adininistered by subcutaneous, intraperitoneal, and intravenous routes, tolypomycin Y is effective in mice infected with Staphylococcus aureus Streptococcuspyrogenes and Diplococcuspneumoniae. Cross-resistance is observed with rifampicia but not with other antibiotics. Resistance to tolypomycin Y develops rapidly. The bioactivity of tolypomycin R... [Pg.499]

Staphylococcus (MRSA) = methicillin-resistant Staphylococcus aureus. [Pg.83]

Cross-resistance between lincomycin and clindamycin is complete (64), and co-resistances of lincomycin also apply to clindamycin. However, the inactivation of clindamycin by clinical isolates of Staphylococcus haemolyticus and Staphylococcus aureus is caused by adenylylation at the 4-position to form clindamycin 4-(5 -adenylate) [29752-38-3] (7) in contrast to the lincomycin 3-(5 -adenylate) [117785-83-8] (8) that forms (26). [Pg.89]

All of these derivatives protect mice infected with Staphylococcus aureus, however, presumably because of bio transformation into clindamycin (71—73). [Pg.90]

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