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Hemolytic

Phenothia2ine [58-37-7] (thiodiphenylamine) is used orally against intestinal nematodes of mminants and horses. It is used with occasional gastrointestinal upset, hemolytic processes, and photosensitivity. It is used routinely at low concentrations on horse farms to suppress the egg production of intestinal parasites (strongyles) and thus limit pasture contamination and transmission (4). [Pg.404]

The symptoms of vitamin E deficiency in animals are numerous and vary from species to species (13). Although the deficiency of the vitamin can affect different tissue types such as reproductive, gastrointestinal, vascular, neural, hepatic, and optic in a variety of species such as pigs, rats, mice, dogs, cats, chickens, turkeys, monkeys, and sheep, it is generally found that necrotizing myopathy is relatively common to most species. In humans, vitamin E deficiency can result from poor fat absorption in adults and children. Infants, especially those with low birth weights, typically have a vitamin E deficiency which can easily be corrected by supplements. This deficiency can lead to symptoms such as hemolytic anemia, reduction in red blood cell lifetimes, retinopathy, and neuromuscular disorders. [Pg.147]

Methyldopa is effective in mild, moderate, and severe hypertension but a thiazide-type diuretic is needed to overcome the fluid retaining side effect. Methyldopa has been shown to prevent and induce regression of ventricular hypertrophy in hypertensive patients. The principal side effects are sedation, drowsiness, nasal congestion, fluid retention, and in rare occasions, hemolytic anemia. [Pg.142]

Streptokinase. Streptokinase is a single-chain protein containing 415 amino acids, mol wt of 45,000 to 50,000 (261,284—286). It is produced by P-hemolytic streptococci and is not an enzyme per se. Only after streptokinase combines with plasminogen on a 1 1 basis to form a... [Pg.144]

Mice are utilized for testing antiseptics for appHcation to cuts, wounds, and incisions (339). The test bacteria, type 1 pneumococcus and hemolytic streptococcus, ate appHed to the taHs of anaesthetized mice. The tip of the taH is then dipped into the antiseptic for 2 min, after which one-half inch of the taH is removed and inserted into the peritoneal cavity and the incision is closed. If after 10 days the animals survive, the product is considered satisfactory for use as a skin antiseptic. The blood of dead animals is sampled and streaked on blood agar for confirmation of infection from the test bacteria as the cause of death. Since lack of toxicity is another requirement of a product to be appHed to wounds, this test has been combined with a toxicity test (340). [Pg.140]

Streptokinase. The fibrinolytic activity of streptokinase, isolated from strains of hemolytic Streptococci, was first demonstrated in 1933 (63). Streptokinase is a secreted protein product inasmuch as filtrates free of demonstrable bacteria were found to dissolve fibrin clots with rapidity. [Pg.309]

G. H. Williams, Hemolytic Aromatic Substitution. Pergamon Press, London, 1960. [Pg.132]

An affinity sorbent based on WPA-PG carrying immobilized human IgG was applied to the isolation of the first component of the complement (Cl) from human serum and for its separation into subcomponents Clr, Cls and Clq by the one-step procedure [126,127]. Cl was quantitatively bound to the sorbent at 0 °C. The activities of subcomponents Clq and Clr2r2 in the unbound part of the serum were found to be 0.8% and 3.3% of the initial activities in serum. This fraction, therefore, could be used as a R1 reagent for determining the hemolytic activity of Cl. Apparently, the neighboring macromolecules of immobilized IgG resemble to some extent an immune complex, whereas Cl formation is facilitated due to the mobility of polymer chains with the attached IgG macromolecules (Cl is usually dissociated in serum by 30%). After activation of bound Cl by heating (30 °C, 40 min) the activated subcomponent Clr is eluted from the sorbent. Stepwise elution with 0.05 mol/1 EDTA at pH 7.4 or with 0.05 mol/1 EDTA + 1 mol/1 NaCl at pH 8.5 results in a selective and quantitative elution of the activated subcomponent Cls and subcomponent Clq. [Pg.171]

Hematologic diseases autoimmune hemolytic anemia, idiopathic thrombocytopenic purpura, pernicous anemia Kidney disease Goodpasture syndrom, lipoid nephroses, minimal change glomerulonephritis Diseases of the gastrointestinal tract autoimmune chronic active hepatitis, autoimmune atrophic gastritis, Crohn s disease, ulcerative colitis... [Pg.241]

As the above mentioned studies with high supplementation dosages exemplarily show, there is no known toxicity for phylloquinone (vitamin Kl), although allergic reactions are possible. This is NOT true for menadione (vitamin K3) that can interfere with glutathione, a natural antioxidant, resulting in oxidative stress and cell membrane damage. Injections of menadione in infants led to jaundice and hemolytic anemia and therefore should not be used for the treatment of vitamin K deficiency. [Pg.1300]

Rare cases of hemolytic anemia, including fatalities, have been reported with the administration of the cephalosporins. The patient should be monitored for anemia If a patient experiences anemia within 2 to 3 weeks after the start of cephalosporin therapy, drug-induced anemia should be considered. If hemolytic anemia is suspected, the primary health care provider will discontinue the drug therapy. The patient may require blood transfusions to correct tire anemia. Frequent hematological studies may be required. [Pg.79]

Acetaminophen causes few adverse reactions when used as directed on the label or recommended by the primary health care provider. Adverse reactions associated with the use of acetaminophen usually occur with chronic use or when the recommended dosage is exceeded. Adverse reactions to acetaminophen include skin eruptions, urticaria (hives), hemolytic anemia, pancytopenia (a reduction in all cellular components of the blood), hypoglycemia, jaundice (yellow discoloration of the skin), hepatotoxicily (damage to the liver), and hepatic failure (seen in chronic alcoholics taking the drug). [Pg.153]

Idiopathic or secondary thrombocytopenic purpura, hemolytic anemia, red blood cell anemia, congenital hypoplastic anemia... [Pg.516]

Potassium is contraindicated in patients who are at risk for experiencing hyperkalemia, such as those with renal failure, oliguria, or azotemia (file presence of nitrogen-containing compounds in the blood), anuria, severe hemolytic reactions, untreated Addison s disease (see Chap. 50), acute dehydration, heat cramps, and any form of hyperkalemia Potassium is used cautiously in patients with renal impairment or adrenal insufficiency, heart disease, metabolic acidosis, or prolonged or severe diarrhea. Concurrent use of potassium with... [Pg.641]

Bacterial agents, particularly Staphylococcus aureus, P-hemolytic streptococci, and Escherichia coli may give rise to pyomyositis, a form of acute suppurative... [Pg.333]

Co-enzyme obtained from cultures of various strains of Streptococcus haemolyticus and capable of changing plasminogen into plasmin (complex enzyme mixture of streptokinase, streptodornase and streptolysin 0"). From fermentation liquors of hemolytic streptococci species Streptococcus haemolyticus), e. g. H 46 A. [Pg.1905]

Tab. 1.3 Magainin-mimetic peptoid sequences, and antibacterial and hemolytic activities... Tab. 1.3 Magainin-mimetic peptoid sequences, and antibacterial and hemolytic activities...
Certain of these peptoid antibiotics are also selective for bacterial, rather than mammalian, cells. The selectivity of these peptoids has been measured in terms of their capacity to cause hemolysis of human erythrocytes at or near their MIC (Tab. 1.3). Interestingly, the amount of hemolysis induced by these peptoids correlates well with their hydrophobicity as there is an increasing extent of hemolysis as molecular hydrophobicity increases. These results suggest that highly hydro-phobic compounds of this class are poorly selective antibiotics. The most active antibacterial peptoids, T2-15 and T3-12, have quite low hemolytic activity near their MICs. Although highly antibacterial in vitro, T3-17 is also very hemolytic at its MIC value. [Pg.21]


See other pages where Hemolytic is mentioned: [Pg.310]    [Pg.526]    [Pg.365]    [Pg.497]    [Pg.263]    [Pg.273]    [Pg.261]    [Pg.6]    [Pg.306]    [Pg.299]    [Pg.203]    [Pg.170]    [Pg.33]    [Pg.200]    [Pg.247]    [Pg.264]    [Pg.267]    [Pg.613]    [Pg.61]    [Pg.117]    [Pg.152]    [Pg.576]    [Pg.653]    [Pg.30]    [Pg.301]    [Pg.302]    [Pg.215]    [Pg.216]    [Pg.21]    [Pg.98]    [Pg.99]    [Pg.99]   
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See also in sourсe #XX -- [ Pg.10 , Pg.11 , Pg.12 , Pg.17 , Pg.18 , Pg.29 , Pg.36 , Pg.43 , Pg.48 , Pg.58 , Pg.114 , Pg.129 , Pg.132 , Pg.144 , Pg.148 ]

See also in sourсe #XX -- [ Pg.458 , Pg.459 ]

See also in sourсe #XX -- [ Pg.171 , Pg.174 , Pg.303 , Pg.337 , Pg.452 ]

See also in sourсe #XX -- [ Pg.282 ]




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A hemolytic streptococci

Acetaminophen hemolytic anemia with

Acute hemolytic anemia

Anemia hemolytic, Heinz body

Anemia hemolytic, sulfonamides causing

Anemia, hemolytic megaloblastic

Anemia, hemolytic pernicious

Anemia, hemolytic sideroblastic

Anemia, hemolytic vitamin B6 deficiency

Anemia, hemolytic vitamin C deficiency

Anemia, hemolytic vitamin E deficiency

Anemia, hereditary hemolytic

Anemias hemolytic

Autoimmune hemolytic anemia

Bilirubin Hemolytic jaundice

Biological hemolytic effect

Ceftriaxone hemolytic anemia

Cephalosporins hemolytic anemia with

Ciprofloxacin hemolytic anemia

Ciprofloxacin hemolytic anemia with

Dapsone hemolytic anemia

Dapsone hemolytic anemia with

Drug-induced immune hemolytic anemia

Group A beta hemolytic streptococci

Halistanol sulfate hemolytic activity

Hemolysis hemolytic anemia

Hemolytic Bone marrow

Hemolytic Hemosiderin

Hemolytic Urobilinogen

Hemolytic activities, amphiphilic

Hemolytic activities, amphiphilic polymers

Hemolytic activity

Hemolytic activity of Gypsophila paniculata

Hemolytic activity ofSAPO

Hemolytic anaemia

Hemolytic anemia allergic drug reaction

Hemolytic anemia autoimmune type

Hemolytic anemia clinical findings

Hemolytic anemia dapsone caused

Hemolytic anemia deficiency

Hemolytic anemia diagnosis

Hemolytic anemia drug-induced

Hemolytic anemia drugs associated with

Hemolytic anemia drugs involved

Hemolytic anemia immune

Hemolytic anemia immune response

Hemolytic anemia laboratory findings

Hemolytic anemia mechanisms

Hemolytic anemia menadione toxicity

Hemolytic anemia oxidative

Hemolytic anemia pathophysiology

Hemolytic anemia penicillin-induced

Hemolytic anemia treatment

Hemolytic anemias deficiency causing

Hemolytic anemias glucose-6-phosphate dehydrogenase

Hemolytic anemias primaquine-sensitive

Hemolytic crises

Hemolytic disease

Hemolytic disease of newborn

Hemolytic disease of the newborn

Hemolytic drug reaction

Hemolytic factors

Hemolytic foci

Hemolytic index

Hemolytic jaundice

Hemolytic plaque assay

Hemolytic plasma

Hemolytic streptococci cell proteins

Hemolytic streptococci growth

Hemolytic streptococci proteins

Hemolytic streptococcus infections

Hemolytic substitution

Hemolytic syndromes

Hemolytic transfusion reaction

Hemolytic-uremic syndrome

Hemolytic-uremic syndrome tacrolimus nephrotox

Hemolytic-uremic syndrome valacyclovir

Hereditary Hemolytic Anemia Associated with Red Blood Cell Enzyme Deficiency

Hereditary hemolytic anemia adenosine deaminase overproduction

Hereditary hemolytic anemia glucose-6-phosphate dehydrogenase deficiency

Hereditary hemolytic anemia phosphoglycerate kinase deficiency

Hereditary hemolytic anemia pyruvate kinase deficiency

Indinavir hemolytic anemia with

Infant hemolytic disease

Interferon alfa hemolytic anemia

Interferon therapy hemolytic anemia with

Levodopa hemolytic anemia with

Mefenamic acid hemolytic anemia with

Metformin hemolytic anemia

Methyldopa hemolytic anemia

Methyldopa hemolytic anemia with

Newborn hemolytic disease

Penicillin hemolytic anemia with

Phenacetin hemolytic anemia with

Primaquine hemolytic anemia with

Procainamide hemolytic anemia with

Quinidine hemolytic anemia with

Sickle cell hemolytic transfusion reaction

Streptococci hemolytic, enzymes

Streptococci, group hemolytic

Streptococci, hemolytic

Sulfamethoxazole hemolytic anemia with

Sulfonamides hemolytic anemia with

Sulfonamides, hemolytic anemia

Thrombocytopenic purpura-hemolytic

Thrombocytopenic purpura-hemolytic uremic syndrome

Tolbutamide hemolytic anemia with

Triterpene glycoside hemolytic activity

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