Paint lead-based

Over the past 30 years, many lead-based products have disappeared from use or the use has been greatly diminished. These products include lead-based paints, lead-based anti-knock compounds for automobiles, lead glazes for pottery, lead sealants for wine battles, solder for sealing the seams of steel cans, automobile body solder, solders for potable water systems, solder for sealing copper radiators, chemical tank linings, lead pipes, lead-based metal bearings, lead sealants such as for drain pipes, power and electrical cable sheathing, and ammunition. 

HOW LEAD IN PAINT IS REGULATED Federal Law Regulates Lead in Paint Lead-Based Paint Ban... 

Lead use, much like asbestos use, was curtailed in the mid-1970s, because the EPA identified buildings and facilities at risk built before 1978. The majority of lead exposure can be found in the disturbance of lead-based paint. Lead-based paint was more durable than other paints offered at the time and also held color for a longer period of time. Holding the same qualities as asbestos, lead-based paint is not a danger if not disturbed, or friable. Once disturbed, however, several hazards become apparent. 

Because of the risk of lead poisoning, the exposure of children to lead-based paint is a significant public health concern. The first step in the quantitative analysis of lead in dried paint chips is to dissolve the sample. Corl evaluated several dissolution techniques. " In this study, samples of paint were collected and pulverized with a Pyrex mortar and pestle. Replicate portions of the powdered paint were then taken for analysis. Results for an unknown paint sample and for a standard reference material, in which dissolution was accomplished by a 4-6-h digestion with HNO3 on a hot plate, are shown in the following table. 

The %w/w lead in a lead-based paint Standard Reference Material and in unknown paint chips is determined by atomic absorption using external standards. 

Lead-based paint (qv) in old stmctures has been identified as an environmental risk because the chemical form of lead in paint is readily available... 

For asbestos removal, the provisions of the OSHA Asbestos Standard 29 CFR 1926.1101 are more protective of worker health and safety than are the more general provisions. The HASP therefore provides that the asbestos removal tasks conducted inside the building will be performed in accordance with the OSHA Asbestos Standard. After the asbestos has been removed, the lead-based paint will be removed. Again, the provisions of the OSHA Standard for lead removal are more protective of worker health and safety than are the more general provisions of 29 CFR 1910.120. Therefore, the removal of the lead-based paint inside the building will be performed in full compliance with the OSHA Lead Standard [1]. 

In addition to the hazardous waste standards, there may be a variety of other standards that may apply to any worksite. One standard that seems to surprise PMs is the lead standard. Even projeets that are elean eonstruetion (not HAZWOPER or working with ehemieals) may eome under the lead standard. If new proeess equipment is being installed in an existing plant, any tie-ins, pipe raek, struetural members, and even walls may have been eovered with many layers of lead-based paint. 

As an example, if the work requires that the lead-based paint is to be disturbed by drilling a hole in a beam (the beam that has been painted with lead-based paint), will workers be exposed to lead in the air Wliat should be done under the standard to be sure that workers are not being overexposed Under the standards 29 CER 1910.1025, Oeeupational Exposure to Lead in General Industry, and 29 CER 1926.62, Oeeupational Exposure to Lead in Construetion, the following are some eri-teria that should be applied to determine who should be enrolled in the lead program. 

Founded as the National Asbestos Council, EIA is a resource for professionals in the management and control of health hazards to occupants of buildings, facilities, and industrial sites. It de >elops and disseminates information about asbestos, lead based paint hazards, underground storage tanks (USTs), indoor air quality, solid and hazardous waste. 

Paints used for protecting the bottoms of ships encounter conditions not met by structural steelwork. The corrosion of steel immersed in sea-water with an ample supply of dissolved oxygen proceeds by an electrochemical mechanism whereby excess hydroxyl ions are formed at the cathodic areas. Consequently, paints for use on steel immersed in sea-water (pH 8-0-8-2) must resist alkaline conditions, i.e. media such as linseed oil which are readily saponified must not be used. In addition, the paint films should have a high electrical resistance to impede the flow of corrosion currents between the metal and the water. Paints used on structural steelwork ashore do not meet these requirements. It should be particularly noted that the well-known structural steel priming paint, i.e. red lead in linseed oil, is not suitable for use on ships bottoms. Conventional protective paints are based on phenolic media, pitches and bitumens, but in recent years high performance paints based on the newer types of non-saponifiable resins such as epoxies. 

During the 1960s, Americans lived in a lead-drenched society. They fueled their cars with leaded, antiknock gasoline. They ate food and their babies drank milk from lead-soldered cans. They stored drinking water in lead-lined tanks and transported it through lead or lead-soldered pipes. They squeezed toothpaste from lead-lined tubes and poured wine from bottles sealed with lead-covered corks. They picked fruit sprayed with lead arsenate pesticide and served it on lead-glazed dishes in houses painted and puttied with lead-based compounds. 

U.S. EPA has proposed TSCA standards for the disposal of lead-based paint (LBP) debris to replace RCRA regulations. The new standards would establish disposal standards for LBP debris and identify recycling and incineration activities that would be controlled or prohibited. To avoid duplicative regulation, the waste that is subject to these new standards would not be subject to RCRA hazardous waste determination. 

Sources of lead in dust and soil include lead that falls to the ground from the air, and weathering and chipping of lead-based paint from buildings and other structures. Lead in dust may also come from windblown soil. Disposal of lead in municipal and hazardous waste dump sites may also add lead to soil. Mining wastes that have been used for sandlots, driveways, and roadbeds can also be sources of lead. 

You may also be exposed to lead in the home if you work with stained glass as a hobby, make lead fishing weights or ammunition, or if you are involved in home renovation that involves the removal of old lead-based paint. Chapter 5 contains further information on sources of exposure to lead. 

In some cases children swallow nonfood items such as paint chips these may contain very large amounts of lead, particularly in and around older houses that were painted with lead-based paint. The paint in these houses often chips off and mixes with dust and dirt. Some old paint is 5-40% lead. Also, compared to adults, a bigger proportion of the amount of lead swallowed will enter the blood in children. 

The most important way families can lower exposures to lead is to know about the sources of lead in their homes and avoid exposure to these sources. Some homes or day-care facilities may have more lead in them than others. Families who live in or visit these places may be exposed to higher amounts of lead. These include homes built before 1978 that may have been painted with paint that contains lead (lead-based paint). If you are buying a home that was built before 1978, you may want to know if it contains lead based paint. Federal government regulations require a person selling a home to tell the real estate agent or person buying the home of any known lead-... 

Respiratory Effects. The only information located regarding respiratory effects in humans associated with lead exposure was a case report of a 41-year-old man who was exposed to lead for 6 years while removing old lead-based paint from a bridge. At the time of the initial assessment, his PbB level was 87 pg/dL, and he complained of mild dyspnea for the last 2-3 years. No abnormalities in respiratory function were seen at clinical examination, so it is not possible to conclude that his respiratory symptoms were related to exposure to lead (Pollock and Ibels 1986). 

Cardiovascular effects have been noted in occupationally exposed workers after exposure to high levels of lead following exposure durations of as short as 4 weeks. Construction workers (race not specified) using oxyacetylene torches to cut a metal bridge that had been painted with lead-based paint were reported to exhibit increases in heart rate and blood pressure after 4 weeks of exposure (Marino et al. 

Inhibition of ALAD and stimulation of ALAS result in increased levels of ALA in blood or plasma and in urine. For example, in a case report of a 53-year-old man with an 11-year exposure to lead from removing old lead-based paint from a bridge, a PbB level of 55 pg/dL was associated with elevated urinary ALA (Pollock and Ibels 1986). The results of the Meredith et al. (1978) study on lead workers and controls indicated an exponential relationship between PbB and blood ALA. Numerous studies reported direct correlations between PbB level and log urinary ALA in workers. Some of these studies indicated that correlations can be seen at PbB levels of <40 pg/dL (Lauwerys et al. 1974 Selander and Cramer 1970 Solliway et al. 1996), although the slope may be different (less steep) than at PbB levels of >40 pg/dL. In a study of 98 occupationally exposed subjects (51 pg/dL, mean PbB) and 85 matched controls (20.9 pg/dL. mean PbB) it was found that log ZPP and log ALA in urine correlated well with PbB levels (Gennart et al. 1992a). In the exposed group, the mean ZPP was 4 times higher than in the controls, whereas urinary ALA was increased 2-fold. 

There is suggestive evidence indicating that the changes in NCV associated with lead exposure may be transient. Muijser et al. (1987) investigated the effects of a 5-month exposure to lead during the demolition of a steel structure coated with lead-based paints. The motor and sensory nerve conduction velocities were measured in the median and ulnar nerves of eight exposed workers and compared with unexposed referents as well as themselves at 3 and 15 months after the termination of exposure. The mean PbB levels in the exposed workers were 82.5 18.9 pg/dL at the termination of exposure,... 

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