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Monoamine-oxidase

2 Monoamine oxidase B. In a detailed study, DeRose and co-workers have investigated monoamine oxidase B with EPR and ENDOR spectroscopy at both X- and Q-band. The enzyme is an integral membrane protein of the outer mitochondrial membrane and plays an essential role in the regulation of various neurotransmitter and xenobiotic amines by catalysing the oxidative deamina- [Pg.224]


Patients receiving monoamine oxidase inhibitors (MAOI) as antidepressant therapy have been especially subject to the hypertensive effects of vasoactive amines (52). These dietary amines have also been impHcated as causative agents ia migraine. Other aaturaHy occurring alkaloids (qv) have been recognized for centuries as possessing neurological stimulant and depressant properties. [Pg.478]

The 2-isopropyUiydrazide derivative of 4-CPA is iproclozide [3544-35-2] a pharmaceutical that inhibits monoamine oxidase. [Pg.424]

Metabolism. MetaboHsm of histamine occurs via two principal enzymatic pathways (Fig. 1). Most (50 to 70%) histamine is metabolized to /V-methylhistamine by A/-methyltransferase, and some is metabolized further by monoamine oxidase to /V-methy1imidazo1eacetic acid and excreted in the urine. The remaining 30 to 40% of histamine is metabolized to imidazoleacetic acid by diamine oxidase, also called histaminase. Only 2 to 3% of histamine is excreted unchanged in the urine. [Pg.136]

Vanadium. Vanadium is essential in rats and chicks (85,156). Estimated human intake is less than 4 mg/d. In animals, deficiency results in impaired growth, reproduction, and Hpid metaboHsm (157), and altered thyroid peroxidase activities (112). The levels of coen2yme A and coen2yme Q q in rats are reduced and monoamine oxidase activity is increased when rats are given excess vanadium (157). Vanadium may play a role in the regulation of (NaK)—ATPase, phosphoryl transferases, adenylate cyclase, and protein kinases (112). [Pg.388]

Dopamine. Dopamine (DA) (2) is an intermediate in the synthesis of NE and Epi from tyrosine. DA is localized to the basal ganglia of the brain and is involved in the regulation of motor activity and pituitary hormone release. The actions of DA are terminated by conversion to dihydroxyphenylacetic acid (DOPAC) by monoamine oxidase-A and -B (MAO-A and -B) in the neuron following reuptake, or conversion to homovanillic acid (HVA) through the sequential actions of catechol-0-methyl transferase (COMT) and MAO-A and -B in the synaptic cleft. [Pg.540]

Histamine AND histamine antagonists). It is formed from histidine by the enzyme L-histidine decarboxylase. In the periphery, histamine is stored ia mast cells, basophils, cells of the gastric mucosa, and epidermal cells. In the CNS, histamine is released from nerve cells and acts as a neurotransmitter. The actions of histamine ate terrninated by methylation and subsequent oxidation via the enzymes histamine-/V-methyltransferase and monoamine oxidase. [Pg.554]

Treatment of Major Depression. Dmgs commonly used for the treatment of depressive disorders can be classified heuristicaHy iato two main categories first-generation antidepressants with the tricycHc antidepressants (TCAs) and the irreversible, nonselective monoamine—oxidase (MAO) inhibitors, and second-generation antidepressants with the atypical antidepressants, the reversible inhibitors of monoamine—oxidase A (RIMAs), and the selective serotonin reuptake inhibitors (SSRIs). Table 4 fists the available antidepressants. [Pg.229]

Monoamine—Oxidase Inhibitors. In the mid-1950s, tuberculosis patients with depression being treated with iproniazid (42) were occasionally reported to become euphoric. This observation led to the discovery of irreversible monoamine—oxidase (MAO) inhibiting properties. Hydrazine and nonhydrazine-related MAO inhibitors were subsequentiy shown to be antidepressants (122). Three other clinically effective irreversible MAO inhibitors have been approved for treatment of major depression phenelzine (43), isocarboxazid (44), and tranylcypromine (45). [Pg.230]

Reversible Inhibitors of Monoamine Oxidase. Selective MAO-A inhibitors, which aie leveisible (so-called RIMAs), have also been developed, theiefoie substantially leduciag the potential foi food and dmg iateiactions. Indeed, the tyiamine-potentiating effects of these dmgs is much reduced compared with the irreversible MAO inhibitors. The RIMAs represent effective and safer alternatives to the older MAO inhibitors. The only marketed RIMAs ate toloxatone [29218-27-7] and moclobemide (55). The latter is used widely as an effective, weU-tolerated antidepressant. [Pg.233]

Future Outlook for Antidepressants. Third-generation antidepressants are expected to combine superior efficacy and improved safety, but are unlikely to reduce the onset of therapeutic action in depressed patients (179). Many dmgs in clinical development as antidepressive agents focus on estabhshed properties such as inhibition of serotonin, dopamine, and/or noradrenaline reuptake, agonistic or antagonistic action at various serotonin receptor subtypes, presynaptic tt2-adrenoceptor antagonism, or specific monoamine—oxidase type A inhibition. Examples include buspirone (3) (only... [Pg.233]

Monoamine Oxidase Inhibitors. MAOIs inactivate the enzyme MAO, which is responsible for the oxidative deamination of a variety of endogenous and exogenous substances. Among the endogenous substances are the neurotransmitters, norepinephrine, dopamine, and serotonin. The prototype MAOI is iproniazid [54-92-2] (25), originally tested as an antitubercular dmg and a close chemical relative of the effective antitubercular, isoniazid [54-85-3] (26). Tubercular patients exhibited mood elevation, although no reHef of their tuberculosis, following chronic administration of iproniazid. In... [Pg.465]

Normally, dietary tyramine is broken down in the gastrointestinal tract by MAO and is not absorbed. In the presence of MAOI, however, all of its potent sympathomimetic actions are seen. Other side effects of MAOI include excessive CNS stimulation, orthostatic hypotension, weight gain, and in rare cases hepatotoxicity. Because the monoamine oxidase inhibitors exhibit greater toxicity, yet no greater therapeutic response than other, newer agents, clinical use has been markedly curtailed. The primary use for MAOIs is in the treatment of atypical depressions, eg, those associated with increased appetite, phobic anxiety, hypersomnolence, and fatigues, but not melancholia (2). [Pg.466]

Isoproterenol is given sublingually or by iv. It is metabolized by monoamine oxidase and catechol-0-methyltransferase in brain, Hver, and other adrenergically innervated organs. The pharmacological effects of isoproterenol are transient because of rapid inactivation and elimination. About 60% is excreted unchanged. Adverse effects using isoproterenol therapy include nervousness, hypotension, weakness, dizziness, headache, and tachycardia (86). [Pg.120]

Copper is one of the twenty-seven elements known to be essential to humans (69—72) (see Mineral nutrients). The daily recommended requirement for humans is 2.5—5.0 mg (73). Copper is probably second only to iron as an oxidation catalyst and oxygen carrier in humans (74). It is present in many proteins, such as hemocyanin [9013-32-3] galactose oxidase [9028-79-9] ceruloplasmin [9031 -37-2] dopamine -hydroxylase, monoamine oxidase [9001-66-5] superoxide dismutase [9054-89-17, and phenolase (75,76). Copper aids in photosynthesis and other oxidative processes in plants. [Pg.256]

Pharmacologically useful isoxazoles (B-82MI41600) include antibacterial sulfonamides (614), (615) and (616), semisynthetic penicillins (617), (618), (619) and (620), semisynthetic cephalosporin (621), anabolic steroid (622), the monoamine oxidase inhibitor (623) (used in psychotherapy), antiinflammatory agent (624) and antitumor agent (625). [Pg.127]

Mitochondrial monoamine oxidase, 1, 253 Mitomycin synthesis, 7, 658, 659 Mitomycin-A, 7, 93 Mitomycin-B, 7, 93 Mitomycin-C, 7, 93 as antitumor drug, 4, 374 Mixed function oxidases, 1, 224 Mobam... [Pg.703]

Pargyline hydrochloride (Eutonyl, (V-methyl-n-propargylbenzylamine hydrochloride) [306-07-0] M 195.7, m 154-155 , 155 , pK 6.9. Recrystd from EtOH-Et20 and dried in vacuo. It is very soluble in H2O, in which it is unstable. The free base has b 101-103°/ 1mm. It is a glucuronyl transferase inducer and a monoamine oxidase inhibitor, [von Braun et al. Justus Liebigs Ann Chem 445 205 1928, Yeh and Mitchell Experientia 28 298 1972 Langslrom et al. Science 225 1480 1984.]... [Pg.556]

Monoamine oxidase (MAO) inactivates serotonergic and catecholaimnergic neurotransmitters MAO (A and B) inhibitors exhibit mood elevatmg properties 5-Fluoro-Ot-methyltryptamine 19) is an important MAO A-seleUive inhibitor In the treatment of certam depressive illnesses, 4-fluorotranylcypromine (20b) is 10 tunes more potent than the parent tranylcypromme (TCP, 20a) The enhanced m vivo activity may be due to increased lipophihcity at20b and/or to blockade of metabohc para hydroxylation [52]... [Pg.1017]

Thiadiazolo[3,4-/]quinoline monoamine oxidase inhibitory activities were examined in (91YZ499), but the values were too high to compare with other pentanthrene type of heterocycles. [Pg.223]

Oxazolidinones and dihydrofuranones as inactivators and substrates of monoamine oxidase B, approaches to the design of antiparkinsonian agents 97F343. [Pg.235]

Hydrazides of isonicotinic acid have been used as antidepressant agents by virtue of their monoamine oxidase-inhibiting activity the pyridine ring has been shown to be replaceable by an... [Pg.232]

MAO (monoamine oxidase) inhibitor. An agent that blocks one of the enzymes that deaminates amines. [Pg.453]


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1- Phenylcyclopropylamine, monoamine oxidase

Activation, of monoamine oxidase

Aggression monoamine oxidase inhibitor

Amfetamine Monoamine oxidase inhibitors

Amitriptyline Monoamine oxidase inhibitors

Amphetamines monoamine oxidase inhibitors

Anticonvulsants monoamine oxidase inhibitors

Antidepressant agents monoamine oxidase inhibitors

Antidepressant drugs monoamine oxidase inhibitors

Antidepressants Monoamine oxidase inhibitors Serotonin-selective

Antidepressants monoamine oxidase inhibitors

Antihypertensives Monoamine oxidase inhibitors

Atomoxetine Monoamine oxidase inhibitors

Barbiturates Monoamine oxidase inhibitors

Biotransformation monoamine oxidase

Bupropion Monoamine oxidase inhibitors

Buspirone monoamine oxidase inhibitors

Caffeine Monoamine oxidase inhibitors

Cascade Reactions Involving Monoamine Oxidases

Catalysis (cont monoamine oxidase

Cheese, monoamine oxidase inhibitors

Chlorpromazine monoamine oxidase inhibitors

Citalopram Monoamine oxidase inhibitors

Cocaine monoamine oxidase inhibitors

Depression monoamine oxidase inhibitors

Depressive disorders monoamine oxidase inhibitors

Dextromethorphan monoamine oxidase

Disulfiram Monoamine oxidase inhibitors

Dopamine Monoamine oxidase inhibitors

Enzyme monoamine oxidase

Fenfluramine monoamine oxidase inhibitors

Fluoxetine monoamine oxidase inhibitors

Fluvoxamine Monoamine oxidase inhibitors

Food interactions, monoamine oxidase

Hormonal) Monoamine oxidase inhibitors

Human monoamine oxidases

Hypericum monoamine oxidase inhibitors

Imipramine Monoamine oxidase inhibitors

Inhibition of monoamine oxidase

Inhibitors of monoamine oxidases

Instructions for patients taking nonselective monoamine oxidase inhibitors

Isoniazid monoamine oxidase inhibitors

Linezolid Monoamine oxidase inhibitors

Lithium monoamine oxidase inhibitors

Liver , monoamine oxidase inhibitor interaction

Look up the names of both individual drugs and their drug groups to access full information Monoamine oxidase inhibitors

MAO (monoamine oxidase

MAOI (monoamine oxidase

Medicines) Monoamine oxidase inhibitors

Methadone Monoamine oxidase inhibitors

Methyldopa Monoamine oxidase inhibitors

Methylphenidate Monoamine oxidase inhibitors

Mirtazapine Monoamine oxidase inhibitors

Mitochondrial monoamine oxidase

Monoamin oxidase

Monoamine Oxidase and Phenol

Monoamine Oxidases and their Inhibitors

Monoamine oxidase , presynaptic nerve

Monoamine oxidase , presynaptic nerve terminal

Monoamine oxidase -catalyzed

Monoamine oxidase -catalyzed oxidation

Monoamine oxidase A

Monoamine oxidase A and

Monoamine oxidase A inhibitor

Monoamine oxidase MAO) inhibitors

Monoamine oxidase action

Monoamine oxidase activated

Monoamine oxidase activity

Monoamine oxidase adverse effects

Monoamine oxidase blockers

Monoamine oxidase catalysis

Monoamine oxidase catalyzed deamination reactions

Monoamine oxidase catecholamine inactivation

Monoamine oxidase catecholamine metabolization

Monoamine oxidase catecholamine production

Monoamine oxidase chemistry

Monoamine oxidase comparative studies

Monoamine oxidase deficiency

Monoamine oxidase degradation

Monoamine oxidase distribution

Monoamine oxidase dopamine

Monoamine oxidase drugs acting

Monoamine oxidase effect on dopamine

Monoamine oxidase effect on noradrenaline

Monoamine oxidase effect on serotonin

Monoamine oxidase functions

Monoamine oxidase in outer mitochondrial membran

Monoamine oxidase inactivation

Monoamine oxidase indications

Monoamine oxidase inhibition

Monoamine oxidase inhibitor MAOI)

Monoamine oxidase inhibitor contraindications

Monoamine oxidase inhibitor interaction and

Monoamine oxidase inhibitors

Monoamine oxidase inhibitors MAOIs) interactions

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Monoamine oxidase inhibitors Parkinson disease

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Monoamine oxidase isoenzymes

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Monoamine-oxidase B inhibitors

Morphine Monoamine oxidase inhibitors

Neurotransmitter degradation by monoamine oxidase (MAO

Nicotine monoamine oxidase activity

Of monoamine oxidase

Older Antidepressants Tricyclics and Monoamine Oxidase Inhibitors

Opioids monoamine oxidase inhibitors

Oxidases monoamine oxidase

Oxidases monoamine oxidase

Oxidative reactions monoamine oxidases

Oxidoreductases monoamine oxidase

Panic disorder monoamine oxidase inhibitors

Paroxetine Monoamine oxidase inhibitors

Pharmacokinetics monoamine oxidase inhibitors

Pharmacology monoamine oxidase

Phenothiazines Monoamine oxidase inhibitors

Postural hypotension, with monoamine oxidase

Postural hypotension, with monoamine oxidase inhibitors

Procarbazine monoamine oxidase inhibitors

Prozac Interaction With Monoamine Oxidase Inhibitors and Tryptophan

Pseudoephedrine Monoamine oxidase inhibitors

Psychotropic agents monoamine oxidase inhibitors

RIMAs (Reversible inhibitors of monoamine oxidase

Rasagiline Monoamine oxidase inhibitors

Reserpine Monoamine oxidase inhibitors

Reversible inhibitor of monoamine oxidase A

Reversible inhibitor of monoamine oxidase type A

Reversible inhibitors of monoamine oxidase

Reversible inhibitors of monoamine oxidase type

Reversible monoamine oxidase inhibitors

Reversible monoamine oxidase inhibitors RIMA) type

Selective serotonin reuptake inhibitors monoamine oxidase

Selective serotonin reuptake with monoamine oxidase

Selegiline Monoamine oxidase inhibitors

Serotonin monoamine oxidase

Sertraline Monoamine oxidase inhibitors

Social anxiety disorder monoamine oxidase inhibitors

Tramadol Monoamine oxidase inhibitors

Trazodone monoamine oxidase

Tricyclic antidepressants and monoamine oxidase inhibitors

Triptans Monoamine oxidase inhibitors

Type-B monoamine oxidase

Tyramine, dietary, monoamine oxidase inhibitor

Tyramine, monoamine oxidase inhibitors

Xenobiotic metabolism monoamine oxidase

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