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Gonadotropin

Prosta.te Ca.ncer, Estrogen has an inhibitory effect on the prostate in addition to its suppression of gonadotropin secretion by the pituitary. The three- and five-year survival rates in prostate cancer patients with metastatic disease improved when treated with DES (7) alone or along with castration. However, DES does not improve the survival rates in patients whose carcinoma is confined to the prostate. Small doses of DES (1 mg/d) appear to retard prostate cancer growth and could reduce the cardiovascular complications associated with larger doses (5 mg/d) (135) (see... [Pg.244]

Leucine residues 2, 5, 7, 12, 20, and 24 of the motif are invariant in both type A and type B repeats of the ribonuclease inhibitor. An examination of more than 500 tandem repeats from 68 different proteins has shown that residues 20 and 24 can be other hydrophobic residues, whereas the remaining four leucine residues are present in all repeats. On the basis of the crystal structure of the ribonuclease inhibitor and the important structural role of these leucine residues, it has been possible to construct plausible structural models of several other proteins with leucine-rich motifs, such as the extracellular domains of the thyrotropin and gonadotropin receptors. [Pg.56]

Gonadotropin releasing hormone analogs (goserelin, buserelin, leuprorelin, triptorelin) inhibit gonadotropin release and thus lower testosterone or estrogen levels. They are used to treat breast cancer and prostate cancer. [Pg.155]

In premenopausal women the ovary is the richest source of aromatase and hence estrogen. Aromatase is confined to the granulosa cells and is produced under the influence of gonadotropins (FSH and LH). Despite being a rich source of aromatase, three separate studies have shown that aromatase inhibitors are unable to sufficiently suppress ovarian estrogen production to postmenopausal levels. One explanation for this phenomenon may be a compensatory rise in gonadotrophins which maintains adequate estrogen production, despite the presence of the inhibitor. As such aromatase inhibitors cannot be used in premenopausal breast cancer patients. After menopause, ovarian... [Pg.219]

Chorionic gonadotropin (CG) is produced in the placenta. Together- with the pituitary hormones, luteinizing hormone (LH) and follicle-stimulating hormone (FSH), it constitutes the glycoprotein family of gonadotropins. The actions of CG are mediated by the LH receptor, both belonging to the superfamily of G-protein Coupled Receptors. [Pg.361]


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Anterior pituitary gonadotropin

Anti- Chorionic gonadotropin, human

Cancer Gonadotropins

Chorionic gonadotropin

Chorionic gonadotropin excretion

Chorionic gonadotropin function

Chorionic gonadotropin inactivation

Chorionic gonadotropin, human

Endometriosis gonadotropin-releasing hormone agonists

Estrogens gonadotropin-releasing hormone

Exogenous gonadotropin

GnRH (gonadotropin-releasing

GnRH and Gonadotropins

Gonadotropin Luteinizing hormone

Gonadotropin Release from Anterior Pituitary Cells

Gonadotropin Thyroid-stimulating

Gonadotropin analogs

Gonadotropin analogue

Gonadotropin antagonists

Gonadotropin bases

Gonadotropin biochemistry

Gonadotropin gene expression

Gonadotropin hormone

Gonadotropin hormone antagonists

Gonadotropin humans

Gonadotropin inhibitor

Gonadotropin measurement

Gonadotropin neurons

Gonadotropin ovarian hyperstimulation

Gonadotropin pituitary responsiveness

Gonadotropin plasma levels

Gonadotropin preparations

Gonadotropin receptors

Gonadotropin regulation

Gonadotropin release

Gonadotropin releasing

Gonadotropin releasing hormone GnRH LHRH)

Gonadotropin releasing hormone antagonists

Gonadotropin releasing hormone receptor

Gonadotropin releasing hormone structure

Gonadotropin secretion

Gonadotropin severe

Gonadotropin surge

Gonadotropin synthesis

Gonadotropin tannate

Gonadotropin urine

Gonadotropin-II

Gonadotropin-releasing Factor/Hormone

Gonadotropin-releasing factor

Gonadotropin-releasing hormone

Gonadotropin-releasing hormone (GnRH

Gonadotropin-releasing hormone GnRH) agonists

Gonadotropin-releasing hormone GnRH) analogs

Gonadotropin-releasing hormone GnRH) antagonists

Gonadotropin-releasing hormone NMR spectrum

Gonadotropin-releasing hormone actions

Gonadotropin-releasing hormone agonists

Gonadotropin-releasing hormone analogs

Gonadotropin-releasing hormone analogues

Gonadotropin-releasing hormone antineoplastic

Gonadotropin-releasing hormone stimulation test

Gonadotropin-releasing hormone therapy

Gonadotropin-releasing hormone-associated

Gonadotropin-releasing hormone-dependent

Gonadotropin-releasing-hormon

Gonadotropins GnRH agonists

Gonadotropins cells

Gonadotropins clinical trial

Gonadotropins observational study

Gonadotropins oocyte

Gonadotropins purification

Gonadotropins spermatogenesis

Human Chorionic Gonadotropin (hCG)

Human chorionic gonadotropin hormone

Human chorionic gonadotropin recombinant

Human chorionic gonadotropin stimulation

Human chorionic gonadotropin, use

Human gonadotropin-releasing

Human gonadotropin-releasing hormone

Human menopausal gonadotropin

Hypothalamic gonadotropin releasing

In gonadotropins

P-subunit of human chorionic gonadotropin

Pituitary gonadotropins

Placental chorionic gonadotropin

Pregnancy blood, gonadotropin

Pregnancy chorionic gonadotropin

Protein human chorionic gonadotropin

Reaction of Different Species to Various Gonadotropins

Serum chorionic gonadotropin

Stimulation test human chorionic gonadotropin

Subject gonadotropin-releasing hormon

Synergism between the Anterior Pituitary Lobe and Chorionic Gonadotropin

Urine chorionic gonadotropin

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