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Clots

I6I C. Warfarin baits need contain only 0 025% active principle, and rats are killed after ingesting about 5 doses the bait can be left down and the risk of acute toxicity to man or domestic animals is not serious. In common with other coumarin derivatives, warfarin reduces the clotting power of blood and death is caused by haemorrhages initiated by any slight injury. Warfarin is a vitamin K antagonist, and large oral doses of the vitamin can be given as an antidote. [Pg.425]

Surfaces can be active in inducing blood clotting, and there is much current searching for thromboresistant synthetic materials for use in surgical repair of blood vessels (see Ref. 111). It may be important that a protective protein film be strongly adsorbed [112]. The role of water structure in cell-wall interactions may be quite important as well [113]. [Pg.552]

Another physiologically important quinone is vitamin K Here K stands for koag ulation (Danish) because this substance was first identified as essential for the normal clotting of blood... [Pg.1013]

Physiological responses to prostaglandins encompass a variety of effects Some prostaglandins relax bronchial muscle others contract it Some stimulate uterine con tractions and have been used to induce therapeutic abortions PGEj dilates blood vessels and lowers blood pressure it inhibits the aggregation of platelets and offers promise as a drug to reduce the formation of blood clots... [Pg.1080]

The concentration of t-PA in human blood is 2—5 ng/mL, ie, 2—5 ppb. Plasminogen activation is accelerated in the presence of a clot, but the rate is slow. The dissolution of a clot requites a week or more during normal repair of vascular damage (17). Prevention of irreversible tissue damage during a heart attack requires that a clot, formed by mpture of an atherosclerotic plaque, be dissolved in a matter of hours. This rapid thrombolysis (dissolution of the clot) must be achieved without significant tibrinogenolysis elsewhere in the patient. [Pg.44]

Whole blood is seldom used ia modem blood transfusion. Blood is separated into its components. Transfusion therapy optimizes the use of the blood components, using each for a specific need. Red cell concentrates are used for patients needing oxygen transport, platelets are used for hemostasis, and plasma is used as a volume expander or a source of proteins needed for clotting of the blood. [Pg.519]

Each component of blood has a function ia the body. Red cells transport oxygen and carbon dioxide between the lungs and cells ia the tissues. White cells function as defense of the body. Platelets are important for hemostasis, ie, the maintenance of vascular iategrity. Plasma, an aqueous solution containing various proteias and fatty acids, transports cells, food, and hormones throughout the body. Some proteias ia plasma play a role ia clotting, others are messengers between cells. [Pg.520]

They release adenosine diphosphate [58-64-0 (ADP) and thromboxane [57576-52-0] which results in vascular contraction and, indirectiy, in the formation of fibrin clot. Platelet transfusions are indicated for patients with thrombocytopenia, ie, a shortage of healthy platelets or thrombocytopathy, ie, platelet malignancy associated with spontaneous hemorrhages. [Pg.520]

Plasma Collection. Human plasma is collected from donors either as a plasma donation, from which the red cells and other cellular components have been removed and returned to the donor by a process known as plasmapheresis, or in the form of a whole blood donation. These are referred to as source plasma and recovered plasma, respectively (Fig. 1). In both instances the donation is collected into a solution of anticoagulant (146) to prevent the donation from clotting and to maintain the stabiUty of the various constituents. Regulations in place to safeguard the donor specify both the frequency of donation and the volume that can be taken on each occasion (147). [Pg.531]

Determination of the potency of Factor VIII is also difficult. This is normally measured by the abiUty of the sample to correct the clotting time of plasma deficient in Factor VIII. A number of methods and practices have evolved for this purpose (231), but these give very different results, particularly when activation of products may also occur (232). International standards have been used, but further standardization of the analytical method and harmonization of working standards is underway (233,234) under the auspices of the ISTH and the EC. [Pg.536]

Liver and Gallbladder. High dosages of oral estrogens have been reported to increase the risk for jaundice, cholestatic hepatitis, gallstones, and hepatic vein blood clots. Estrogens promote the development of hepatic neoplasms associated with increased hepatic cell regenerative activity (186,187). [Pg.245]

The calcium ion, necessary for blood-clot formation, stimulates release of bloodclotting factors from platelets (see Blood, coagulants and anticoagulants) (25). Neuromuscular excitabihty also depends on the relative concentrations of Na", Ca ", Mg ", and (26). Upon a decrease in... [Pg.376]

The primary advantages of implantable ports are no maintenance between uses other than periodic flushing with heparinized saline every 28 days to ensure patency, lower incidence of clotting and thrombosis, no dressing changes, insignificant infection incidence, unobtmsive cosmetic appearance, and no restriction on physical activity. [Pg.184]

Patients immediate post-operative pain is lower compared to a standard operation and healing and rehabiUtation more rapid. Patients can resume near-normal activities in just days. In some cases athletes, who are in prime physical condition, can return to challenging athletic activities within a few weeks. CompHcations are rare, but do occur on occasion. Most complications associated with this surgery are infection, phlebitis, excessive swelling or bleeding, blood clots, or damage to blood vessels or nerves. [Pg.190]

Biochemical Reactions. The quinones in biological systems play varied and important roles (21,22). In insects they are used for defense purposes, and the vitamin K family members, eg, vitamin [11104-38-4] (32) and vitamin [11032-49-8] (33), which are based on 2-meth5l-l,4-naphthoquiaone, are blood-clotting agents (see Vitamins, vitamin k). [Pg.406]

The importance of quinones with unsaturated side chains in respiratory, photosynthetic, blood-clotting, and oxidative phosphorylation processes has stimulated much research in synthetic methods. The important alkyl- or polyisoprenyltin reagents, eg, (71) or (72), illustrate significant conversions of 2,3-dimethoxy-5-methyl-l,4-ben2oquinone [605-94-7] (73) to 75% (74) [727-81-1] and 94% (75) [4370-61-0] (71—73). [Pg.412]

Hematology. The functional status of blood and of the blood-forming tissues can be assessed by tests which include red and white blood cell counts, platelet counts, clotting time, coagulation tests, and examination of bone marrow. Such tests, in addition to detecting abnormahties, may also allow differentiation between primary and secondary effects on blood and blood-forming tissues (75). [Pg.236]


See other pages where Clots is mentioned: [Pg.201]    [Pg.343]    [Pg.397]    [Pg.423]    [Pg.403]    [Pg.454]    [Pg.2565]    [Pg.2696]    [Pg.911]    [Pg.1081]    [Pg.457]    [Pg.21]    [Pg.720]    [Pg.406]    [Pg.544]    [Pg.1059]    [Pg.1070]    [Pg.44]    [Pg.44]    [Pg.392]    [Pg.532]    [Pg.233]    [Pg.242]    [Pg.371]    [Pg.503]    [Pg.235]    [Pg.259]    [Pg.386]    [Pg.177]    [Pg.179]    [Pg.474]    [Pg.484]    [Pg.229]   
See also in sourсe #XX -- [ Pg.92 ]




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Activated clotting enzyme

Activated clotting time

Activated clotting time measurement

Affect the Clotting Cascade

Biochemistry blood clotting

Bleeding and Blood Clotting Problems

Blood Clotting Test

Blood clot coagulation cascade

Blood clot coagulation cascade pathway

Blood clot coagulation factor inhibitors

Blood clot coagulation factors

Blood clot drugs

Blood clot embolization

Blood clot foreign surfaces

Blood clot formation

Blood clot hemostasis

Blood clot times

Blood clots

Blood clots, generation

Blood clotting

Blood clotting acetylsalicylic acid

Blood clotting and

Blood clotting anticoagulation systems

Blood clotting cascade mechanism

Blood clotting cofactors

Blood clotting cross-linking

Blood clotting factor

Blood clotting factor VIII

Blood clotting fibrin clot, control

Blood clotting inhibition

Blood clotting intrinsic mechanism

Blood clotting mechanisms

Blood clotting oral contraceptives

Blood clotting proteases

Blood clotting proteins

Blood clotting serine proteases

Blood clotting time

Blood clotting, biological messenger

Blood clotting, effect

Blood clotting, vitamin

Blood coagulation clotting enhancement

Blood oxygenators clotting

Blood-clotting cascade

Blood-clotting enzymes

Blood-clotting factors, binding

Blood-clotting proteins, interaction

Blood-clotting proteins, interaction with surfaces

Cardiovascular system clotting

Chymosin milk-clotting action

Clot Retraction

Clot Types

Clot busters

Clot dissolution—fibrinolysis

Clot evolution

Clot extraction devices

Clot formation, mechanism

Clot fragmentation

Clot lysis

Clot obstruction

Clot retraction, inhibition

Clot strength

Clot, fibrin, formation

Clot-dissolving drugs

Clot-dissolving system

Clot-promoting surfaces

Clots dissolution

Clots formation

Clots prevention

Clotted soap

Clotting

Clotting

Clotting Time on Concentration and pH

Clotting abnormality

Clotting agents

Clotting cascade

Clotting disease

Clotting disease fibrinolytic agent

Clotting disorders

Clotting disorders anticoagulants

Clotting disorders antiplatelet drugs

Clotting disorders drugs used

Clotting extrinsic pathway

Clotting factor VII

Clotting factor concentrates

Clotting factor inhibitors

Clotting factors

Clotting factors heparin affecting

Clotting factors listed

Clotting factors recombinant

Clotting factors reduced production

Clotting factors under Factor

Clotting factors, anticoagulant rodenticides

Clotting factors, deficiency

Clotting factors, surface-activated

Clotting factors, vitamin

Clotting factors, vitamin dependent

Clotting impaired

Clotting inhibitors

Clotting intrinsic pathway

Clotting of blood

Clotting prevention

Clotting process, milk

Clotting regulation

Clotting screens

Clotting system

Clotting time

Clotting time dependence

Clotting time factors affecting

Clotting time incorporation

Clotting time modeling

Clotting time of blood

Clotting zymogen activation

Clotting, cell

Coagulation specific clotting factors

Disseminated intravascular clotting

Drugs Affecting Blood Clotting

Ecarin clotting time

Euglobulin clot lysis time

Fiber clotting

Fiber clotting effect

Fibrin clot

Fibrin clot lysis

Fibrin clot method

Fibrin clot rigidity

Fibrin clot turbidity

Fibrin in clotting

Fibrinogen clots

Fibrinogen clotting reaction

Fibrinogen-fibrin conversion Step 3: Clotting

Fibrinolytic agent in treatment of clotting diseas

Hemostasis blood clotting system

Hemostasis clot formation

Hemostatic wound dressings blood clot

Hepatitis blood clotting factor

Herbicides of blood clotting

Kinetic Clotting Test

Mechanical clot disruption

Metabolism blood clotting

Milk-Clotting Activity

Phenox Clot Retriever

Phospholipids blood clotting

Plasma-thrombin clot methodology

Prevention of Unwanted Blood Clotting

Properties of Fibrin Clots

Prostaglandins blood clotting time

Protease in blood clotting

Proteins blood-clotting, 214 brain

Proteins clotting factors

Prothrombin time test clotting assay

Removal of a Blood Clot

Sedimentation and Clotting

Series reactions blood clotting

Serine proteases blood clotting factors

Serine proteases in blood clotting

Streptokinase clot selectivity

Stroke clot removal

That Inhibit Excessive Clot Formation

That Promote Clotting

Thrombin clots

Thrombin clotting time

Thrombin fibrin clots

Thrombin fibrin clots generation

Thrombin-Induced Clot Formation in Canine Coronary Artery

Thrombin-induced clot formation

Thrombus white clot

Treatment of clotting disorders

Various Properties during Clotting

Vitamin K dependent clotting factor

Vitamin K-Dependent Proteins in Blood Clotting

Vitamin K-dependent clotting

Wounds clotting process

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