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Tumor, Ehrlich ascites

Older investigations have shown that whole RJ and lO-hydroxy-2-decenoic acid (10-HDA) as well as certain closely related dicarboxylic acids could inhibit the development of transplantable AKR leukemia and ascites tumors (Ehrlich ascites cells) when added in vitro just prior to transfer. This activity was associated mainly with 9- and 10-carbon straight-chain monocarboxylic acids, particularly decanoic acid in the ethyl ester form. These compounds showed no activity in vivo [101]. [Pg.284]

Ehrlich ascites tumor agar, collagen glucose 2.4... [Pg.225]

Osterreich, S., Schunck, H., Benndorf, R., Bielka, H. (1991). Cisplatin induces the small heat shock protein hsp25 and thermotolerance in Ehrlich ascites tumor cells. Biochem. Biophys. Res. Comm. 180,243-248. [Pg.458]

Iliakis G. 1984. The mutagenicity of alpha particles in ehrlich ascites tumor cells. Radiat Res 99 52-58. [Pg.243]

ATPase activity of P-glycoprotein related to emergence of drug resistance in Ehrlich ascites tumor cell lines, Biochim. Biophys. Acta 1997,... [Pg.492]

Panniers, R., and Henshaw, E. C. (1983). A GDP/GTP exchange factor essential for eukaryotic initiation factor 2 cycling in Ehrlich ascites tumor cells and its regulation by eukaryotic initiation factor 2 phosphorylation. J. Biol. Chem. 258, 7928—7934. [Pg.50]

Xanthothricin 308 stimulated oxidation of NADH and NAD-linked substrates by rat liver mitochondria, yeast mitochondria, and Ehrlich ascites tumor cells (74MI1, 74MI3). It also stimulated mitochondrial oxidation of succinate, pyruvate, or malate. The antibiotic xanthothricin was obtained... [Pg.261]

Agranofl, B. W., Hajra, A. K. The acyl dihydroxyacetone phosphate pathway for glycerolipid biosynthesis in mouse liver and Ehrlich ascites tumor cells. Proc. Natl. Acad. Sci. U.S. 68, 411-415 (1971). [Pg.68]

A protein highly homologous to the S100 proteins has been isolated from Ehrlich ascites tumor cells it has subsequently been shown to be nearly identical with human calcyclin. The fluorescence intensity from the three tyrosine residues is enhanced on the binding of Ca2+.(l58)... [Pg.35]

In different organs of the rat [128], Ehrlich ascites tumor cells [144], trout testis [127], calf thymus [145], and carp testis [146], H4 is modified mainly as the N -dimethyllysine, while H3 is modified as N -monomethyllysine, N -dimethylly-sine and N -trimethyllysine with the N -dimethyllysine predominating. Pea seedling H4 is not methylated and H3 exits as N -mono- and N -dimethyllysine with N -trimethyllysine not being detectable [147,148]. [Pg.218]

The temporal sequence of H3 and H4 methylation after synthesis has been examined in Ehrlich ascites tumor cells [144] and trout testis [149]. Methylation lagged histone synthesis, and the histone was methylated after being bound to DNA. H4 methylation follows the stepwise acetylations and deacetylations [149]. It was suggested that methylation was involved in final arrangement of H3 and H4 on newly replicated DNA [144] and might be involved in histone interactions with other proteins such as histone kinases [149]. [Pg.218]

Gregory, R.I. et al. (2002) Inhibition of histone deacetylases alters allelic chromatin conformation at the imprinted U2afl-rsl locus in mouse embryonic stem cells. J. Biol. Chem. 277, 11728-11734. Thomas, G., Lange, H.W., and Hempel, K. (1975) Kinetics of histone methylation in vivo and its relation to the cell cycle in Ehrlich ascites tumor cells. Eur. J. Biochem. 51, 609-615. [Pg.305]

Phenanthrenebiguanides possess 384) some activity against cancer. The effect of biguanides on plasma protein surface has been investigated 294, 610) with a view to the possibility of finding anti-tumor ents. Aryl-and naphthyl-biguanides exhibit 625) a slight anti-neoplastic activity in vitro in Ehrlich ascites carcinoma. [Pg.74]

Woerdenbag et al also evaluated the influence of chiral center configurations present in artemisinin (1) structure on the proliferation of Ehrlich ascites tumor (ETA) cells. Compounds 11-hydroxyartemisinin (47) and 11-hydroxy-11-epi-artemisinin (48) (Fig. 4) were synthesized and the... [Pg.321]

Woerdenbag HI, Moskal TA, Pras N, Malingre TM, El-Feraly FS, Kampinga HH, Konings AWT. (1993) Cytotoxicity of artemisinin-related endoperox-ides to Ehrlich ascites tumor cells. J Nat Prod 56 849-856. [Pg.333]

DC059 Majumder, P. K., and M. Gupta. Effect of the seed extract of carrot Daucus carota Linn.) on the growth of Ehrlich ascites tumor in mice. Phytoter Res 1998 12(8) 584-585. [Pg.213]

Antitoxic effect. Sesame oil, adiministered to male Wistar rats, ameliorated hepatic and renal damage in a dose-dependent manner and increased survival in lipopolysaccha-ride-treated rats. It decreased lipid peroxide concentration in serum but not in liver and kidney. Serum nitrite production was unaffected by sesame oil ingestion, and the activity of xanthine oxidase was reduced in the lipopolysaccharide-challenged rats k Anti-tumor activity. Water extract of the dried seed, administered intragastrically to mice at a dose of 50 mg/animal daily for 5 days, was active on CA-Ehrlich-ascites, 18% increase in life-span. Intraperitoneal administration was active on Dalton s lyphoma and CA-Ehrlich-ascites, 19 and 39% increase in life-span, respectively ". Seed oil, administered to rats intraperito-neally with 1,2,5,6-dibenzanthracene or re-tene, was active on sarcoma ". [Pg.493]

Genotoxic Effects. Genotoxic Effects. HDI was demonstrated to be non-mutagenic against some Salmonella typhimurium strains with or without metabolic activation (Anderson et al. 1980). HDI also inhibited the growth of Ehrlich ascites tumor eells in female mice (Moos et al. 1971) and decreased the mutation frequency in Escherichia coli (Kawazoe et al. 1981). Calf thymus DNA incubated in vitro with 10.4 or 52 mol of HDI for 10 or 20 minutes produeed no evidence of intrastrand cross-links or DNA strand breaks (Peel et al. 1997). No studies were located that studied the genotoxic effects of HDI on human cells or that deseribed the ability of prepolymer forms of HDI to induce genotoxicity."... [Pg.107]

In die peripheral blood system, crocetin derivatives prevent an elevation in bilirubin levels [3] and also reduce elevated levels of serum cholesterol and triglyceride [4]. Anti-tumor activity of saffron is observed in mice transplanted with several types of tumor cell lines including sarcoma 180, Ehrlich ascites carcinoma, and Dalton s lymphoma ascites... [Pg.314]

Not only have animals or isolated organs of animals been used to perform pharmacological studies, but also cells in culture. In a study done by Prof. Yamasaki25 at the University of Hiroshima (Figure 16.28), Ehrlich ascites tumor cells have been used to demonstrate the increase of glucose transport into the cell as a function of G115. [Pg.227]

Effect of standardized P. ginseng extract G115 on the glucose transport in Ehrlich ascites tumor cells. [Pg.229]

K. Yamasaki et al., Effects of the standardized ginseng extract G115 on the D-glucose transport by Ehrlich ascites tumor cells, Phytother. Res., 7, 200, 1993. [Pg.234]

Belkin et al. 29> were first to examine various polysaccharide fractions from higher plants for their antitumor activity. They could demonstrate that many of these fractions produced haemorrhagic necrosis in different tumor types. In most cases, the polysaccharides were injected intraperitoneally into mice carrying Sarkoma 37 ascites tumor. The result was a progressive increase in cell volume and in cytoplasmic vacuolization. Osswald 30) found that tragacanth, gum arabic, and CMC reduced tumor cells in Ehrlich ascites carcinoma in female NMRE mice. The effect depended upon the dose, the route of injection, and the molecular size of the polysaccharides administered. [Pg.28]

Other pathways to better resolution and enhanced sensitivity may involve methods used in NMR studies of cellular phosphorus dynamics and humic and fulvic materials of water, soil, and sediments. These techniques include a variety of extractions, ion-pairing reagents, adsorption techniques, and lanthanide-shift reagents. For example, an extraction technique that has greatly enhanced the resolution and sensitivity of 31P FT-NMR spectra of Ehrlich ascites tumor cells (53) and HeLa cells (54) is the addition of 35% perchloric acid and removal of acid-insoluble material, followed by filtration and neutralization with K.2C03 or NaOH. [Pg.192]

It has been found that a similar type of self-regulatory active transport also operates in mammalian cells, in this case in the Ehrlich ascites tumor cell (41). These cells show an active concentrative ability for glucose at low ambient glucose concentrations. This enables the cell to... [Pg.280]

K. 5 -Nudeotidase from Ehrlich Ascites Tumor Cells. . . 348... [Pg.337]


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See also in sourсe #XX -- [ Pg.217 ]




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