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Pulmonary system pneumonia

Cefepime is used for bacterial infections caused by microorganisms that are sensitive to drugs in septicemia, bacteriemia, complicated infections of the upper and lower sections of the urinary system, pneumonia, pulmonary abscesses, emphysema of the pleura, fever in patients with neutropenia, and infected skin and soft tissue wounds. Synonyms of this drug are maxipime, cepim, cepimex, and others. [Pg.462]

Probably the vanadium compound to which people are most likely to be exposed is vanadium pentoxide, V205. Exposure normally occurs via the respiratory route, and the pulmonary system is the most likely to suffer from vanadium toxicity. Bronchitis and bronchial pneumonia are the most common pathological effects of exposure skin and eye irritation may also occur. Severe exposure can also adversely affect the gastrointestinal tract, kidneys, and nervous system. [Pg.231]

However, the excessive intake and accumulation of zinc leads to toxic effects on all living organisms. This is accompanied with oxygen system depression and anemia. The excessive content of zinc in human organisms induces vomiting, nausea, pneumonia and fibrosis of pulmonary system. [Pg.169]

CHRONIC HEALTH RISKS increased risk of lung cancer decreased pulmonary function pneumonia bronchitis asthma liver and kidney damage effects on gastrointestinal and immune systems effects on the blood contact dermatitis skin ulcerations sensitivity nasal itching and soreness complications during pregnancy and childbirth chromate salts are suspected carcinogens. [Pg.507]

Unlabeled Uses Cardiopulmonary bypass surgery hemodialysis pulmonary hypertension associated with acute respiratory distress syndrome, systemic lupus erythematosus, or congenital heart disease refractory CHF severe community-acquired pneumonia... [Pg.441]

There are also several examples of natural surfactants and foams in the human body. The understanding of the pulmonary surfactant system, although discovered in 1929, has only been applied clinically since about 1990 for the treatment of respiratory distress syndrome. Surfactant replacement therapy may also be used in treating other forms of lung disease, such as meconium aspiration syndrome, neonatal pneumonia and congenital diaphragmatic hernia [881]. Lung surfactant, composed of phospholipids and proteins [882,883], is necessary to maintain a low surface tension at the alveolar air-liquid interface. When there is a deficiency of surfac-... [Pg.327]

Fox-Dewhurst, R., Alberts, M., Kajikawa, O., Caldwell, E Johnson, M. C. I., Skerrett, S. J., et al. (1997) Pulmonary and systemic inflammatory responses in rabbits with gram-negative pneumonia. Am. J. Respir. Crit. Care Med. 155, 2030-2040. [Pg.329]

A 72-year-old man with autoimmune thrombocytopenia had taken prednisone (30 mg/day) for 1 year, when he was found to have systemic lupus erythematosus (46). Prednisone was continued and he started to take chloroquine (250 mg/day) and monthly cyclophosphamide (0.75 g/m ). Three weeks after the first bolus of cyclophosphamide, he complained of fever and dyspnea, and chest X-rays showed bilateral pulmonary infiltrates. Despite prompt medical management, he died 5 days after admission with cytomegalovirus-induced interstitial pneumonia. [Pg.1028]

Six cases of complications loosely related to the use of naltrexone pellet implantation during the highly controversial rapid and ultra-rapid opioid detoxification procedures have been reported (22). These included pulmonary edema, prolonged opioid withdrawal states, drug toxicity, withdrawal from cross-dependence to alcohol and benzodiazepines, aspiration pneumonia, and death. The risk of these controversial procedures and of naltrexone in this novel delivery system are high a robust scientifically validated program of research is needed to justify such treatment packages. [Pg.2425]

For the respiratory tract, inhalation can cause spasms, inflammation and edema of the larynx and bronchi, dyspnea, cyanosis, pneumonia, and pulmonary edema. Serious symptoms, such as pulmonary edema and asphyxiation, may not be observed for hours after overexposure. Occasionally, cardiac failure occurs as a complication of severe pulmonary edema. With regards to the cardiovascular system, diphosgene can cause rapid heartbeat and hypotension. Gastrointestinal exposure may cause nausea and vomiting in patients and may be fatal. [Pg.888]

Contact with propylene oxide may cause severe skin and eye irritation. Cases of allergic contact dermatitis and hand eczema have been described. Inhalation of propylene oxide may result in spasm, inflammation, and edema of the larynx and bronchi, as well as pulmonary edema leading to pneumonia. Symptoms of exposure may include burning sensation, coughing, wheezing, headache, nausea, and vomiting. Propylene oxide may cause central nervous system depression and other neurological disorders. [Pg.2132]

ACE angiotensin-converting enzyme CNS central nervous system COPD chronic obstructive pulmonary disease DPIs dry-powder inhalers EDTA ethylenediamine tetraacetic acid EDA Eood and Drug Administration EEVi forced expiratory volume in 1 second HIV human immunodeficiency virus IPS idiopathic pneumonia syndrome NSAIDs nonsteroidal anti-inflammatory drugs... [Pg.588]

Tetanus is a severe acute illness caused by the exotoxin of Clostridium tetani. Sustained muscle contractions are characteristic of tetanus. Tetanus toxin interferes with neurotransmitters that promote muscle relaxation leading to continuous muscle spasms. Death can be due to the tetanus toxin itself or secondary to a complication such as aspiration pneumonia, dysregulation of the autonomic nervous system, or pulmonary embolism. [Pg.2236]


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See also in sourсe #XX -- [ Pg.619 ]




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