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Clinical microbiology

Stokes E.J. Ridgway G.L. (1993) Clinical Microbiology, Ttii edn. London Edward Arnold. [Pg.33]

The most advanced PDF inhibitor to emerge thus far from this collaboration is LBM-415 (12) (also called NVP PDF-713 or VIC-104959), an V-formyl-V-hydroxylamine compound still containing a proline residue at P2. The activity, PK properties, and in vivo efficacy data of (12) were presented at the 14th European Congress of Clinical Microbiology and Infectious Diseases (ECCMID) (2004) and the structure of this... [Pg.125]

Jones, R.N., Fritsche, T.R. and Sadar, H.S. (2004) 14th European Congress of Clinical Microbiology and Infectious Diseases, Prague, Czech Republic, Poster P914. [Pg.141]

Bergeron, M. G. Ouellette, M. Preventing antibiotic resistance through rapid genotypic identification of bacteria and of their antibiotic resistance genes in the clinical microbiology laboratory. J. Clin. Microbiol. 1998, 36, 2169-2172. [Pg.14]

Goodfellow, M. Freeman, R. Sisson, R R. Curie-point pyrolysis mass spectrometry as a tool in clinical microbiology. Zbl. Bakt. 1997, 285,133-156. [Pg.336]

Carter, D.A., Burt, A., Taylor, J.W., Koenig, G.L. and White, T.J. (1996) Clinical isolates of Histoplasma capsulatum from Indianapolis have a recombining population structure. Journal of Clinical Microbiology 34, 2577-2584. [Pg.80]

ICPs should ensure that hospitals have current telephone numbers for notification of both internal (ICPs, epidemiologists, infectious diseases specialists, administrators, and public affairs officials) and external (state and local health departments, Federal Bureau of Investigation field office, and CDC Emergency Response office) contacts and that they are distributed to the appropriate personnel.9 ICPs should work with clinical microbiology laboratories, on- or off-site, that receive specimens for testing from their facility to ensure that cultures from suspicious cases are evaluated appropriately. [Pg.373]

Murray, P.R., ed.. Manual of Clinical Microbiology, ASM Press, Washington, 1995. [Pg.234]

Chapin, K., and M. Musgnug. Evaluation of Three Rapid Methods for the Direct Identification of Staphylococcus aureus from Positive Blood Cultures. Journal of Clinical Microbiology 41 no. 9 (2003) 4324-4327. [Pg.163]

Spicer WJ. Clinical microbiology and infectious diseases an illustrated colour text. 2nd ed. Philadelphia (PA) and London Churchill Livingstone 2007. [Pg.433]

Acar J., M. Casewell, J. Freeman, H. Goossens, and C. Friis (2000). Avoparcin and virginia-mycin as animal growth promoters A plea for science in decision making. Clinical Microbiology and Infections 6 477-482. [Pg.251]

Chambers H.P. (1997). MethicilUn resistance in staphylococci Molecular and biochemical basis and clinical implications. Clinical Microbiology Reviews 10 781-791. [Pg.256]

Hayes J.R., A.C. McIntosh, S. Qai3mmi, J.A. Johnson, L.L. Enghsh, L.E. Carr, D.W. Wagner, and S.W. Joseph (2001). High-frequency recovery of quinupristin-dalfopristin-resistant Enterococcus faecium isolates from poultry production environment. Journal of Clinical Microbiology 39 2298-2299. [Pg.265]

Kiimmeier K. and A. Henninger (2004). Promoting resistance by the emission of antibiotics from households into effluent. Clinical Microbiology and Infection 9 1203-1214. [Pg.272]

Ruoff K.L., D.R. Kuritzkes, J.S. Wolfson, and M.J. Eerraro (1988). Vancomycin-resistant Gram-positive bacteria isolated from human sources. Journal of Clinical Microbiology 26 2064-2068. [Pg.282]

Van Leeuwen W., H. Verbmgh, J. van der Velden, N. van Leeuwen, M. Heck, and A. van Belkum (1999). Validation of binary typing for Staphylococcus aureus strains. Journal of Clinical Microbiology 37 664-674. [Pg.289]

Werner G., I. Klare, and W. Witte (1998). Association between quinupristin/dalfopristin resistance in gylcopeptide-resistant Enterococcus faecium and the use of additives in animal feed. European Journal of Clinical Microbiology and Infectious Diseases 17 401-402. [Pg.290]

Zadoks R., W. van Leeuwen, H. Berkema, O. Sampimon, H. Verbrugh, Y.H. Schukken, and A. van Belkum (2000). Application of pulsed-field gel electrophoresis and binary typing as tools in veterinary clinical microbiology and molecular epidermiologic analysis of... [Pg.291]

Chapman, A., Vallejo, V., Mossie, K.G., Ortiz, D., Agabian, N. and Flisser, A. (1995) Isolation and characterization of species-specific DNA probes from Taenia solium and Taenia saginata and their use in an egg detection assay, journal of Clinical Microbiology 33, 1283-1 288. [Pg.69]

Dinkel, A., von Nickisch-Rosenegk, M., Bilger, B., Merli, M., Lucius, R. and Romig, T. (1998) Detection of Echinococcus multilocularis in the definitive host coprodiagnosis by PCR as an alternative to necropsy. Journal of Clinical Microbiology 36, 1 871 -1 876. [Pg.93]

Zhang, W., Li, J. and McManus, D.P. (2003) Concepts in immunology and diagnosis of hydatid disease. Clinical Microbiology Reviews 16, 18-36. [Pg.95]

Geerts, S. and Gryseels, B. (2000) Drug resistance in human helminths current situation and lessons from livestock. Clinical Microbiology Reviews 13, 207-222. [Pg.266]

Clinical microbiology laboratory methods to evaluate the drug s effectiveness... [Pg.49]

Drobniewski FA, Watterson SA, Wilson SM, Harris GS. A clinical, microbiological and economic analysis of a national service for the rapid molecular diagnosis of tuberculosis and rifampicin resistance in Mycobacterium tuberculosis. J Med Microbiol 2000 49 271-278. [Pg.49]

Green, L.C., LeBlanc, P.J. and Didier, E.S. (2000) Discrimination between viable and dead encephalitozoon cuniculi (microsporidian) spores by dual staining with Sytox Green and Calcofluor White M2R. Journal of Clinical Microbiology 38, 3811-3814. [Pg.20]

Moraxella catarrhalis is also known in the literature as Branhamella catarrhalis and type strains are available from the ATCC or clinical microbiology laboratories may provide clinical isolates. It is strongly recommended that a biohazard safety hood is used when handling large quantities of the bacterium (see Note 3). [Pg.307]

This patient was treated empirically with vancomycin alone because his physicians suspected that he had endocarditis, caused by Streptococcus of the viridans group. The history indicated that the origin of the infection was the patient s mouth. Three days later the results from the clinical microbiology laboratory showed that three blood cultures, taken at the time of admission, grew Enterococcus faecalis. susceptible to ampicillin, vancomycin, gentamicin, and streptomycin. [Pg.446]

Cahoon-Young, B., Chandler, A., Livermore, T., Gaudino, J., and Benjamin, R. (1989). Sensitivity and specificity of pooled versus individual sera in a human immunodeficiency virus antibody prevalence study. Journal of Clinical Microbiology, 27, 1893-1895. [Pg.65]


See other pages where Clinical microbiology is mentioned: [Pg.10]    [Pg.1093]    [Pg.146]    [Pg.333]    [Pg.451]    [Pg.9]    [Pg.184]    [Pg.316]    [Pg.186]    [Pg.186]    [Pg.189]    [Pg.192]    [Pg.215]    [Pg.537]    [Pg.292]    [Pg.534]    [Pg.13]    [Pg.165]    [Pg.213]    [Pg.251]   
See also in sourсe #XX -- [ Pg.209 , Pg.210 ]




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