Myocardial infarction alcohol

E (1999) Type of alcoholic beverage and risk of myocardial infarction , J Cardiol,... 

Father 64 years of age history of alcohol abuse, hypertension, and myocardial infarction... 

COX-2 inhibitors such as celecoxib are associated with adverse effects such as nephrotoxicity and a potential increased risk of myocardial infarction (see Chaps. 55 and 15 for additional information). Combination of COX-2 inhibitors with alcohol may increase GI adverse effects. All NSAIDs should be used with caution in patients with aspirin-induced asthma.31... 

Hines LM, Stampfer MJ, Ma J et al. Genetic variation in alcohol dehydrogenase and the beneficial effect of moderate alcohol consumption on myocardial infarction. N Engl J Med 2001 344 549-555. 

Most of the human experience with disulfiram has come from its use as an avoidance therapy for alcoholism. Metabolites of disulfiram inhibit aldehyde dehydrogenase, resulting in elevated levels of acetaldehyde after ethanol ingestion. Side effects include flushing of the face, tachycardia, severe headache, apprehension, hyperpnea, hypotension, dizziness, nausea, vomiting, and fainting. Severe reactions may include convulsions, myocardial infarction, and marked respiratory depression. ... 

Genetic Polymorphisms of Alcohol Dehydrogenase Modulates Alcohol Disposition and May Relate to Increased Risk of Myocardial Infarction... 

Abacavir NRTT1 300 mg bid Testing to rule out the presence of the HI A-B 5701 allele is recommended prior to the initiation of therapy Rash, hypersensitivity reaction, nausea. Possible increase in myocardial infarction Avoid alcohol... 

Didanosine (ddl) NRTT1 Tablets, 400 mg daily,3 adjusted for weight. 30 min before or 2 h after meals. Separate dosing from fluoroquinolones and tetracyclines by 2 h Peripheral neuropathy, pancreatitis, diarrhea, nausea, hyperuricemia. Possible increase in myocardial infarction Avoid concurrent neuropathic drugs (eg, stavudine, zalcitabine, isoniazid), ribavirin, and alcohol. Do not administer with tenofovir... 

Reactive Loss (adverse life events). Physical illness (myocardial infarct, cancer). Drugs (antihypertensives, alcohol, hormones). Other psychiatric disorders (senility). More than 60% of all depressions. Core depressive syndrome depression, anxiety, bodily complaints, tension, guilt. May respond spontaneously or to a variety of ministrations. 

Beta (/3)-blockers (e.g., propranolol, atenolol, oxyprenolol, pindolol) are used for treating hypertension, cardiac arrhythmias, angina pectoris, and myocardial infarction. These drugs have proven important in the management of alcohol withdrawal and hypothyroidism.56 -blockers also are used as prophylactics in... 

Disulfiram is an aversive therapy that works by inhibiting acetaldehyde dehydrogenase. Interactions between disulfiram and alcohol can result in potentially severe reactions, such as myocardial infarction, congestive heart failure, respiratory depression and death. Patients taking disulfiram should be warned of the possible presence of alcohol in liquid medicines, tonics, foods and even in toiletries and mouthwashes. Patient adherence to disulfiram is poor and there is a lack of strong evidence for its effectiveness, thus it is not routinely recommended. 

GGT is fouud particularly iu hepatocytes aud biliary epithelial cells. GGT serum levels may be high iu liver disease, but it is particularly a feature of biliary outflow obstruction more so than hepatocellular damage. GGT serum measuremeut provides a very sensitive indicator of the presence or absence of hepatobiliary disease. However, raised GGT levels have also been reported in a variety of other clinical conditions, including pancreatic disease, myocardial infarction, chronic obstructive pulmonary disease, renal failure, diabetes, obesity and alcoholism. It is also a sensitive indicator of liver damage through alcohol iugestion. 

The application of antibodies in cardiovascular targeting in vivo originated with the experimental demonstration of the feasibility of using radiolabeled antimyosin antibody for diagnosis of acute myocardial infarction in 1976. Since then, the use of antibodies in the cardiovascular system has encompassed imaging of myocarditis,heart transplant rejection, dilated cardiomyopathy, alcohol induced cardiomyopathy,adriamycin cardiotoxicity, various other cardiomyopathies, vascular clots, atherosclerotic lesions,and even certain cancers such as soft tissue sarcomas.f Yet the best characterized and studied antibody for cardiovascular diagnostic targeting is monoclonal antimyosin Fab for its exquisite specificity... 

Plasma protein concentration Dehydration Acute myocardial infarction Alcoholism ... 

A 38-year-old white man with a history of coronary artery disease, myocardial infarction, coronary artery by-pass, alcoholism, and depression took a combined massive overdose of diltiazem and atenolol (24). He underwent cardiopulmonary resuscitation because of cardiac arrest bradycardia, hypotension, and oliguria followed and were resistant to intravenous pacing and multiple pharmacological interventions, including intravenous fluids, calcium, dopamine, dobutamine, adrenaline, prenalterol, and glucagon. Adequate mean arterial pressure and urine output were restored only after the addition of phenylephrine and transvenous pacing. He survived despite myocardial infarction and pneumonia. 

Moderate alcohol intake, increased levels of high-density lipoprotein and its subimetions, and decreased risk of myocardial infarction. Hew England Journal of Medicine, 329, 1829-1834. 

The determination of alanine aminotransferase and aspartate aminotransferase (ALAT and ASAT, formerly GPT and GOT) in clinical chemistry is as important as that of glucose. The normal ranges are between 5 and 24 U/l for ALAT and between 5 and 20 UA for ASAT. The enzyme activities can rise up to 1000-fold in acute hepatitis, myocardial infarction, or alcoholic insult. 

A series of article abstracts about various magnesium-deficiency symptoms, syndromes, conditions, or diseases are supplied at http //www.mgwa-ter.com/abstract.shtml. These include the following alcohol-related hypertension and strokes, alcohol-induced contraction of cerebral arteries, amyofrophic lateral sclerosis and aluminum deposition in the central nervous system, cardiac arrhythmias, asthma therapy, attention deficit disorder (ADD), cerebral artery disorders, constipation, diabetes, heart muscle disorders or myocardial infarction, hypertension, HIV, kidney stones, menopause, migraine, multiple sclerosis, osteoporosis, and premenstrual syndrome. In all cases, an increase in magnesium levels had beneficial effects. 

---