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Heart transplant rejection

Horuk R, Clayberger C, Krensky AM, et al. A non-peptide functional antagonist of the CCR1 chemokine receptor is effective in rat heart transplant rejection. J Biol Chem 2001 276 4199-4204. [Pg.152]

Ruschitzka F, Meier PJ, Turina M, Luscher TF, Noll G. (2000). Acute heart transplant rejection due to Saint John s wort. Lancet. 355(9203) 548-49. [Pg.515]

Unlabeled Uses Treatment of livertransplantation rejection, mild heart transplant rejection, moderate to severe psoriasis... [Pg.830]

Ruschitzka, F. Meier, P.J. Purina, M. Luscher, T.F. Noll, G. Acute heart transplant rejection due to Saint John s Wort. Lancet 2000, 555, 548-549. [Pg.82]

The application of antibodies in cardiovascular targeting in vivo originated with the experimental demonstration of the feasibility of using radiolabeled antimyosin antibody for diagnosis of acute myocardial infarction in 1976. Since then, the use of antibodies in the cardiovascular system has encompassed imaging of myocarditis,heart transplant rejection, dilated cardiomyopathy, alcohol induced cardiomyopathy,adriamycin cardiotoxicity, various other cardiomyopathies, vascular clots, atherosclerotic lesions,and even certain cancers such as soft tissue sarcomas.f Yet the best characterized and studied antibody for cardiovascular diagnostic targeting is monoclonal antimyosin Fab for its exquisite specificity... [Pg.1150]

Hypericum perforatum (St. John s wort), which is used for mild depression, is an inducer of cytochrome P-450. It caused a rapid dramatic fall in ciclosporin blood concentrations, resulting in acute heart transplant rejection in two patients aged 61 and 63 years (262). It produces an average 50% reduction in ciclosporin blood concentrations (263). At least three other case reports have clearly confirmed that St. John s wort is dangerous in transplant patients, because it produced a rapid and dramatic reduction in blood ciclosporin concentration and resulted in acute organ rejection in two patients (264—266). Induction of CYP3A4 and/or P-glycoprotein was the most likely mechanism. [Pg.759]

Fig. 1.3 An enzyme induction interaction. Rifampicin (600 mg daily plus isoniazid) increased the metabolism of ciclosporin in this patient thereto reducing the trough serum levels. He subsequently died because his heart transplant rejected (after Tronsp/ont Proc, 16, Van Buren D, Wideman CA, Ried M, Gibbons S, n Buren CT, Jarowenko M, Flechner SM, Frazier OH, Cool DA, Kahan BD. The antagonistic effect of ri mpidn upon cyclosporine bioavailability. 1642—5, Copyright Els ier (1984)). Fig. 1.3 An enzyme induction interaction. Rifampicin (600 mg daily plus isoniazid) increased the metabolism of ciclosporin in this patient thereto reducing the trough serum levels. He subsequently died because his heart transplant rejected (after Tronsp/ont Proc, 16, Van Buren D, Wideman CA, Ried M, Gibbons S, n Buren CT, Jarowenko M, Flechner SM, Frazier OH, Cool DA, Kahan BD. The antagonistic effect of ri mpidn upon cyclosporine bioavailability. 1642—5, Copyright Els ier (1984)).
Tc-HYMC annexin V Heart transplant rejection Narula et al. (27)... [Pg.337]

Acute Heart Transplant Rejection Due to Saint John s Wort. [Pg.406]

See other pages where Heart transplant rejection is mentioned: [Pg.794]    [Pg.43]    [Pg.116]    [Pg.1156]    [Pg.1156]    [Pg.1157]    [Pg.479]    [Pg.36]    [Pg.143]    [Pg.400]    [Pg.67]    [Pg.397]    [Pg.355]    [Pg.49]   


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