Pulmonary infarct

Pulmonary embolism Pulmonary infarction Tissue injury Burns... 

Vascular phenomena, major arterial emboli, septic pulmonary infarcts, mycotic aneurysm, intracranial hemorrhage, conjunctival hemorrhages, and Janeway s lesions... 

Pulmonary embolism A disorder of thrombus formation causing obstruction of a pulmonary artery or one of its branches may result in pulmonary infarction. 

Increased LDH levels are found in patients suffering from diseases related to liver and renal functions, cancer and pulmonary infarction,... 

Drug Treatment. Because of the extensive nature of the pulmonary infarction, a thrombolytic agent was used to attempt to resolve the clot. An initial dosage of 250,000 units of streptokinase (Streptase) was administered intravenously within 2 hours after the onset of symptoms. Streptokinase was continued via intravenous infusion at a rate of 100,000 units/hr for 24 hours after the initial dose. 

The only available information regarding lethal effects in humans after inhalation exposure to acrolein was provided by Gosselin et al. (1979), who described a case of a 4-year-old boy exposed to smoke containing acrolein from an overheated fryer for 2 hours the boy s 2-year-old brother also died however, no details were reported. After 24 hours, death occurred by asphyxia. The autopsy revealed massive cellular desquamation of the bronchial lining and miscellaneous debris in the bronchial lumen. Also, multiple pulmonary infarcts were observed. The information provided by this case report must be considered qualitative only, since smoke components other than acrolein may have contributed to the pathology. 

A 23-year-old woman developed pulmonary infarction associated with the use of crack (105). 

Delaney K, Hoffman RS. Pulmonary infarction associated with crack cocaine use in a previously healthy 23-year-old woman. Am J Med 1991 91(l) 92-4. 

Normal commensals of the upper respiratory tract proliferate in damaged lungs especially following viral infections, pulmonary congestion or pulmonary infarction. Mixed infection is therefore common, and since Haemophilus influenzae and Streptococcus pneumoniae are often the pathogens, amoxicillin or trimethoprim are reasonable choices, but if... 

Pneumonia is often caused by aspiration of material from the oropharynx, or due to the presence of other limg pathology such as pulmonary infarction or bronchogenic carcinoma. As well as conventional microbial causes, the pathogens include anaerobic and aerobic streptococci, Bacteroides spp. and Puso-bacterium, and the diagnosis may be missed unless anaerobic cultures of fresh material are performed. Treatment for several weeks with cefuroxime plus metronidazole may be needed to prevent relapse. 

Jaundice from pulmonary infarction, from large haema-toma, from repeated blood transfusions, occasionally in postoperative icterus... 

Pulmonary infarction jaundice following extensive haemorrhagic pulmonary infarction with haemolysis of the erythrocytes which have passed into the alveoli. 

Elevation of LDH-2 Platelet-related disorders Pulmonary infarction... 

D15. Dijkman, J. H., and Kloppenborg, P. W. C., Increased serum alkaline phosphatase activity in pulmonary infarction. Acta Med. Scand. 180, 273-281 (1966). 

N21. Nikkila, E. A., Serum alkaline phosphatase activity in pulmonary infarction. Scand. J. Clin. Lab. Invest. 11, 405-406 (1959). 

Lung Emphysematous changes pulmonary infarcts reduced bacterial clearance muscle atrophy Pneumonia decreases in functional residual capacity, vital capacity, and maximum breathing capacity depressed hypoxic/hypercarbic drives... 

There is substantial evidence (much from the literature on drug abuse) indicating that particulate matter (undissolved substances) is a health hazard in parenteral products [21], The precise hazards depend very much on the physical and biological properties of the particles, and their site of lodgement in the vascular system. Phlebitis and pulmonary infarctions are the most significani problems associated with particulate matter. Particulate matter is an unwanted and unnecessary addition to parenteral therapy. 

Follitropin Hu follicle stimulating hormone Infertility Ovarian enlargement, ovarian hyperstimulation syndrome, pulmonary embolism, pulmonary infarction... 

Rare complications as have been reported in literature are aortic and bronchial necrosis [58], bronchial stenosis [59], unilateral diaphragmatic paralysis [60], pulmonary infarction (especially in patients who have suffered pulmonary artery embolism), left main bronchial-esophageal fistula [61], and non-target embolization (colon, coronary and cerebral circulation) [62]. Especially the newer spherical embolic materials (tris-acryl gelatin) can traverse from the bronchial into the pulmonary circulation, and then through unoccluded pulmonary arteriovenous malformations into the systemic circulation [41]. 

Complications following embolization of PAVMs have in general been infrequent and self-limited, particularly in experienced hands [20, 40]. Most of the reported complications are minor and self-limited, most of these only require symptomatic treatment [20]. Pleuritic chest pain occurring in the first 24 h after embolization is the most frequent complication encountered in up to 13% of treated patients [11, 20, 40, 55, 77]. The incidence seems higher in patients presenting with large PAVMs [20, 36]. Pleural effusion has been reported in up to 12% of patients [40]. Pulmonary infarction has been observed in 3% of patients and most likely was related to occlusion of normal pulmonary arterial branches [55,71,76]. The... 

Some preventive measures against DVT after ACL reconstmction have been recommended, such as use of elastic stockings and prophylactic use of low molecular weight heparin, but no consensus has been reached [63, 64]. When DVT occurs, anticoagulant therapy, thrombolytic therapy, and vascular interventional radiology performed by specialists should be started promptly. In addition, lung contrast CT and other tests should be performed to detect pulmonary infarction. 

In two cases, an asymptomatic pulmonary embolism was detected. In both cases, paravertebral venous opacification was observed. To avoid major pulmonary infarction, the cement should be injected slowly during its pasty polymerization phase under fluoroscopy control and the injection should immediately be stopped if venous leakage is observed (Amar etal.2001). 

Parkinson, J. E., and A. Sunshine Angiokeratoma corporis diffusum universale (Fabry). Presenting as suspected myocardial infarction and pulmonary infarcts. Am. J. Med. 31, 951 (1961). 

Pleuritic involvement is uncommon in BD. Pleural effusion is thought to be secondary to superior vena cava thrombosis and pulmonary infarction. Very rarely can the pleura be involved by vasculitis, which can manifest as inflammation and effusions. Hydropneumothorax can result from rupture of peripheral subpleural opacities into the pleural space (24,25). Other rare thoracic manifestations include fibrosing mediastinitis. 

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