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Nicotine treatment

Grenhoff J., Janson A. M., Svensson T. H., Fuxe K. (1991). Chronic continuous nicotine treatment causes decreased burst firing of nigral dopamine neurons in rats partially hemitransected at the meso-diencephalic junction. Brain Res. 562, 347-51. [Pg.212]

Benwell, M.E., Balfour, D J. The effects of acute and repeated nicotine treatment on nucleus accum-bens dopamine and locomotor activity. Br. J. Pharmacol. 105 849, 1992. [Pg.33]

Brunzell, D.H., Russell, D.S., Picciotto, M.R. In vivo nicotine treatment regulates mesocorticolimbic CREB and ERK signaling in C57B1/6J mice. J. Neurochem. 84 1431, 2003. [Pg.36]

Ke, L., Eisenhour, C., Bencherif, M., Lukas, R. Effects of chronic nicotine treatment on expression of diverse nicotinic acetylcholine receptor subtypes. I. Dose- and time-dependent effects of nicotine treatment. J. Pharmacol. Exp. Ther. 286 825, 1998. [Pg.47]

Marks, M., Pauly, J., Gross, S. et al. Nicotine binding and nicotinic receptor subunit RNA after chronic nicotine treatment. J. Neurosci. 2765, 1992. [Pg.48]

Harkness, P., Millar, N. Changes in conformation and subcellular distribution of a4b2 nicotinic acetylcholine receptors revealed by chronic nicotine treatment and expression of subunit chimeras. J. Neurosci. 22 10172, 2002. [Pg.48]

Wang, F., Nelson, M., Kuryatov, A. et al. Chronic nicotine treatment upregulates human a3 32 but not a3P4 acetylcholine receptors stably transfected in human embryonic kidney cells. J. Biol. Chem. 1998 28721, 1998. [Pg.48]

Pietila, K., Lahde, T., Attila, M., Ahtee, L., Nordberg, A. Regulation of nicotinic receptors in the brain of mice withdrawn from chronic oral nicotine treatment. Naunyn-Schmiedeberg s Arch. Pharmacol. 357 176, 1998. [Pg.48]

Magata, Y., Kitano, H., Shiozaki, T. et al. Effect of chronic (-) nicotine treatment on rat cerebral benzodiazepine receptors. Nuclear Med. Biol. 27 57, 2000. [Pg.50]

Boyadjieva, N.I.S.D. The secretory response of hypothalamic beta-endorphin neurons to acute and chronic nicotine treatments and following nicotine withdrawal. Life Sci. 61 PL59, 1997. [Pg.50]

Nicotinic receptors are widely distributed in the brain and are involved in a number of physiological processes. The prefrontal cortex is a major site for the cognitive effects of nicotine (Vidal 1996). Some of the cognitive benefits of nicotine may occur after chronic treatment, resulting in part from nicotinic receptor up-regulation in frontal, entorhinal, and dorsal hippocampal regions (Abdulla et al. 1996). Chronic nicotine treatment also increases the sensitivity of muscarinic receptors (Wang et al. 1996). [Pg.109]

Wang H, Cui WY, Liu CG. (1996). Regulatory effects of acutely repeated nicotine treatment towards central muscarinic receptors. Life Sci. 59(17) 1415-21. [Pg.467]

Joshi M, Tyndale RF. 2006. Induction and Recovery Time Course of Rat Brain CYP2E1 following Nicotine treatment. Drug Metab Dispos 34 647-652. [Pg.85]

Lee AM, Miksys S, Palmour R, Tyndale RF 2006. CYP2B6 is expressed in African Green monkey brain and is induced by chronic nicotine treatment. Neuropharmacology 50 441-450. [Pg.86]

Schneider NG, Olmstead RE, Franzon MA, Lunell E (2001) The nicotine inhaler clinical pharmacokinetics and comparison with other nicotine treatments, Chn Pharmacokinet 40(9) 661-684 Schoedel KA, Sellers EM, Palmour R, Tyndale RF (2003) Down-regulation of hepatic nicotine metabolism and a CYP2A6-like enzyme in African green monkeys after long-term nicotine administration. Mol Pharmacol 63(1) 96-104... [Pg.59]

Marks Ml, Campbell SM, Romm E, Collins AC (1991) Genotype influences the development of tolerance to nicotine in the mouse. J Pharmacol Exp Ther 259 392 02 Marks MJ, Pauly JR, Gross SD, Deneris ES, Hermans-Borgmeyer I, Heinemann SF, Collins AC (1992) Nicotine binding and nicotinic receptor subunit RNA after chronic nicotine treatment. J Neurosci 12 2765-2784... [Pg.109]

Pauly JR, Ullman EA, CoUins AC (1990b) Strain differences in adrenalectomy-induced alterations in nicotine sensitivity in the mouse, Pharmacol Biochem Behav 35 171-179 Pauly JR, Marks MJ, Gross SD, Collins AC (1991) An autoradiographic analysis of cholinergic receptors in mouse brain after chronic nicotine treatment, J Pharmacol Exp Ther 258 1127-1136 Perry DC, KeUar KJ (1995) [ H]epibatidine labels nicotinic receptors in rat brain an autoradiographic study. J Pharmacol Exp Ther 275 1030-1034 Perry WLM, Talesnik J (1953) The role of acetylchoUne in synaptic transmission at parasympathetic ganglia. J Physiol 119 455 69... [Pg.110]

Fucile S (2004) Ca + permeability of nicotinic acetylcholine receptors. Cell Calcium 35 1-8 Gaddnas H, Pietila K, Ahtee L (2000) Effects of chronic oral nicotine treatment and its withdrawal on locomotor activity and brain monoamines in mice. Behav Brain Res 113 65-72 Geisler S, Derst C, Veh RW, Zahm DS (2007) Glutamatergic afferents of the ventral tegmental area in the rat. J Neurosci 27 5730-5743... [Pg.199]

Quik M, Parameswaran N, McCallum SE, Bordia T, Bao S, McCormack A, Kim A, Tyndale RF, Langston JW, Di Monte DA (2006) Chronic oral nicotine treatment protects against striatal degeneration in MPTP-treated primates. J Neurochem 98 1866-1875 Raiteri L, Raiteri M (2000) Synaptosomes still viable after 25 years of superfusion, Neurochem Res 25 1265-1274... [Pg.204]

Adolescent nicotine exposure results in pronounced and persistent nicotinic cholinergic receptor upregulation in male rats and hippocampal cell damage in females (Trauth et al. 1999). During and following nicotine treatment as adolescents, female rats show impaired rearing and locomotor activity, whereas males are unaffected. On the other hand, improved performance was observed in passive avoidance (Trauth et al. 2000). [Pg.272]

Overall, a limited number of experiments on rodents suggest that the upregulation of nicotinic receptors by nicotine treatment is observed in males but not females. On the other hand, nicotine self-administration has the same upregulation effect on both male and female rats. In human smokers, DA and 5-HT transporter availabihty appears to be higher in females than males although the difference is not substantial. [Pg.274]

Nicotine is an addictive substance with rewarding and reinforcing properties. On the other hand, the autonomic responses following an acute nicotine treatment and the bitter taste of nicotine may cause aversion. This aversion may impact conditioned effects to nicotme. Rinker et al. (2008) studied possible sex differences in taste aversion mduced by nicotine in rats systemic nicotine or saline injections were paired wim oral saccharine. Although nicotme did produce a weak taste aversion, no sex differences were observed, excluding the possible contribution of the aversive properties of nicotine on sexually dimorphic responses to nicotine. The authors conclude that sex differences may arise from differences in the rewardmg properties of the drug. [Pg.278]

Nisell M, Nomikos GG, et al (1996) Condition-independent sensitization of locomotor stimulation and mesocortical dopamine release following chronic nicotine treatment in the rat. Synapse 22(4) 369-381... [Pg.288]

See other pages where Nicotine treatment is mentioned: [Pg.449]    [Pg.44]    [Pg.534]    [Pg.36]    [Pg.93]    [Pg.107]    [Pg.191]    [Pg.191]    [Pg.192]    [Pg.193]    [Pg.195]    [Pg.197]    [Pg.199]    [Pg.201]    [Pg.201]    [Pg.206]    [Pg.206]    [Pg.229]    [Pg.254]    [Pg.255]    [Pg.272]    [Pg.273]    [Pg.274]    [Pg.276]    [Pg.294]    [Pg.404]   
See also in sourсe #XX -- [ Pg.55 , Pg.59 , Pg.115 , Pg.148 ]



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