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Helicobacter

Helicobacter pylori is a spiral shaped bacterium that lives in the stomach and duodenum (the section of intestine just below the stomach). Most bacteiia cannot live in the stomach since the environment is too acidic to survive. However, Helicobacter pylori has large amounts of urease, which converts urea to ammonia and carbon dioxide. Ammonia is used to maintain the periplasmic and cytoplasmic pH of the bacterium at neutrality, so this bacterium can survive. Helicobacter pylori causes peptic... [Pg.578]

Helicobacter Pylori Helix Bundle Helix-loop-helix Motif Helper T Cells Hemangioblast Hematopoiesis... [Pg.1493]

The proton pump inhibitors are particularly important in the treatment of Helicobacter pylori in patients with active duodenal ulcers. Helicobacter pylori (H. pylori) has been implicated as a causative organism in a type of chronic gastritis and in a large number of cases of peptic and duodenal ulcers. [Pg.476]

A breath test, used to determine the presence of Helicobacter pylori, associated with stomach ulcers, is an example of a diagnostic product involving separate drug and device components. To perform the test, a patient swallows some isotopically (e.g. [Pg.19]

The pathogenesis of rosacea is multifactorial. Vascular hyperactivity is the primary phenomenon that is complicated by inflammatory changes. Endocrine, psychological, pharmacological, immunological, infectious, thermal and alimentary factors contribute to produce vascular instability and tissue damage (Fig. 17.1). The role of Helicobacter pylori is still being discussed. [Pg.185]

Detection and identification of Helicobacter pylori and Demodex folliculorum (Fig. 17.13). [Pg.191]

Schilling CH, Covert MW, Famili I, Church GM, Edwards JS, Palsson BO. Genome-scale metabolic model of Helicobacter pylori 26695. J Bacteriol 2002 184 4582-93. [Pg.527]

Excessive secretion of gastric acid, associated with Helicobacter pylori infection, can result in the development of gastric and duodenal ulcers small changes in the composition of bile can result in crystallization of cholesterol as gallstones failure of exocrine pancreatic secretion (as in cystic fibrosis) leads to undernutrition... [Pg.474]

This genus, originally grouped with the Campylobacters (section 6.4.16), is now considered a separate genus. Helicobacter pylori is of interest as a cause of peptic ulcer. [Pg.31]

FAHEY J W, HARISTOY X, DOLAN P M, KENSLER T W, SCHOLTUS I, STEPHENSON K K, TALALAY p and LOZNIEWSKI a (2002) Sulforaphane inhibits extracellular, intracellular and antibiotic-resistant strains of Helicobacter pylori and prevents benzo[a]pyrene-induced stomach tumors , Proc Natl Acad Sci USA, 99 7610-15. [Pg.61]

Yamashita reported anti-inflammatory effect of astaxanthin when administered with aspirin. An oral preparation has been developed by Alejung and Wadstroem for the treatment of Helicobacter infections of the mammalian gastrointestinal tract. Strong evidence suggested that astaxanthin modulated the humoral and non-humoral immune systems. It enhanced the release of interleukin-1 and tumor necrosis factor-... [Pg.407]

Alejung, P. and Wadstroem, T., Oral preparation for treatment of Helicobacter sp. infections comprises xanthophylls, especially astaxanthin esterified with a fatty acid and derived from the alga Haematococcus species, World Patent 9837874, 1998. [Pg.424]

Nagata K, H Yu, M Nishikawa, M Kashiba, A Nakamura, EE Sato, T Tamura, M Inoue (1998) Helicobacter pylori generates superoxide radicals and modulates nitric oxide metabolism. J Biol Chem 273 14071-14073. [Pg.160]

Helicobacter pylori is associated with chronic gastritis, peptic ulceration and possibly involved in the pathogenesis of gastric carcinoma (Correa and Ruiz, 1992 Dixon,... [Pg.144]

Correa, P. and Ruiz, B. (1992). Helicobacter pylori and gastric cancer. In Helicobacter pylori and Gastroduodenal disease (eds. B.J. Rathbone and R.V. Heatley) pp. 158-164. Black-well Scientific Publications, Oxford. [Pg.162]

Davies, G.R., Simmonds, N.J., Stevens, T.R.J., Grandison, A. and Rampton, D.S. (1991). Enhanced production of reactive oxygen species by gastric mucosa infected with Helicobacter pyirri. Gut 32, A564. [Pg.162]


See other pages where Helicobacter is mentioned: [Pg.467]    [Pg.198]    [Pg.108]    [Pg.128]    [Pg.96]    [Pg.578]    [Pg.578]    [Pg.774]    [Pg.859]    [Pg.1030]    [Pg.1034]    [Pg.1035]    [Pg.85]    [Pg.48]    [Pg.1673]    [Pg.151]    [Pg.21]    [Pg.31]    [Pg.125]    [Pg.52]    [Pg.182]    [Pg.539]    [Pg.257]    [Pg.296]    [Pg.296]    [Pg.144]    [Pg.144]    [Pg.144]    [Pg.159]   
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See also in sourсe #XX -- [ Pg.128 ]

See also in sourсe #XX -- [ Pg.270 ]

See also in sourсe #XX -- [ Pg.120 ]




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Anti-Helicobacter agents

Anti-Helicobacter pylori activitie of gancaonol

Anti-Helicobacter pylori activitie of licochalcone

Anti-Helicobacter pylori activity

Eradication of Helicobacter pylori (C)

Gancaonol anti-Helicobacter pylori activity

Gastric Helicobacter pylori

Helicobacter antibiotics

Helicobacter atrophic gastritis

Helicobacter duodenal ulcer

Helicobacter gastric cancer

Helicobacter gastritis

Helicobacter infection

Helicobacter infection clarithromycin effect

Helicobacter mustelae

Helicobacter peptic ulcer

Helicobacter pylori

Helicobacter pylori adhesins

Helicobacter pylori eradication

Helicobacter pylori growth

Helicobacter pylori in peptic ulcer disease

Helicobacter pylori infection

Helicobacter pylori infection and peptic ulcer disease

Helicobacter pylori inhibition

Helicobacter pylori inhibition by chrysophanol

Helicobacter pylori inhibition by emodin

Helicobacter pylori inhibition by rhein

Helicobacter pylori strains

Helicobacter pylori treatment

Helicobacter pylori urease

Helicobacter pylori virulence factors

Helicobacter pylori, and

Helicobacter pyroli

Helicobacter spp

Helicobacter therapy

Helicobacter vaccines

Licochalcone anti-Helicobacter pylori activity

Reduction in Helicobacter pylori

The Heptoglycan Domain in Helicobacter pylori Lipopolysaccharides

The Lipopolysaccharides of Helicobacter Species from Nonhuman Primates

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