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Helicobacter infection

Yamashita reported anti-inflammatory effect of astaxanthin when administered with aspirin. An oral preparation has been developed by Alejung and Wadstroem for the treatment of Helicobacter infections of the mammalian gastrointestinal tract. Strong evidence suggested that astaxanthin modulated the humoral and non-humoral immune systems. It enhanced the release of interleukin-1 and tumor necrosis factor-... [Pg.407]

Losec (omeprazole) Duodenal ulcer Gastro-oesophageal reflux Helicobacter infections 5.7 1.9 1989 - UK and US Once daily, except when used as part of combination therapy... [Pg.135]

Helicobacter infections are of great concern worldwide due to its association with peptic ulcer disease and gastric cancer. Resistance to available antimicrobials is on the rise, not only in the US but also in the developing countries. This increase in resistance has opened the door for research into... [Pg.492]

Matysiak-Budnik, T., van Niel, G., Megraud, F. et al. 2003. Gastric Helicobacter infection inhibits development of oral tolerance to food antigens in mice. Infect Immun 71 5219-5224. [Pg.38]

Losec (omeprazole) Duodenal ulcer Gastroesophageal reflux Helicobacter infections 5.7... [Pg.164]

Helicobacter infection is a common cause of gastritis and its eradication may be associated with cure of peptic ulcers. Clarithromycin has been shown to be an... [Pg.368]

Maggio-Piice, L., Treuting, R, Zeng, W., Tsang, M., Bielefeldt-Ohmaim, H., and Mtani, B.M. (2006) Helicobacter infection is required for inflammation and colon cancer in SMAD3-deficient mice. Cancer Res. 66, 828-838. [Pg.293]

Crowe, S.E., Helicobacter infection, chronic inflammation, and the development of malignancy. Curr. Opin. Gastroenterol, 21, 32-38 (2005). [Pg.405]

Excessive secretion of gastric acid, associated with Helicobacter pylori infection, can result in the development of gastric and duodenal ulcers small changes in the composition of bile can result in crystallization of cholesterol as gallstones failure of exocrine pancreatic secretion (as in cystic fibrosis) leads to undernutrition... [Pg.474]

Alejung, P. and Wadstroem, T., Oral preparation for treatment of Helicobacter sp. infections comprises xanthophylls, especially astaxanthin esterified with a fatty acid and derived from the alga Haematococcus species, World Patent 9837874, 1998. [Pg.424]

Davies, G.R., Simmonds, N.J., Stevens, T.R.J., Grandison, A. and Rampton, D.S. (1991). Enhanced production of reactive oxygen species by gastric mucosa infected with Helicobacter pyirri. Gut 32, A564. [Pg.162]

Rathbone, B.J. and Heatley, R.V. (1992). Possible pathogenic mechanisms in Helicobacter pylori infection. In Helicobacter pylori and Gastroduodenal Disease (eds. B.J. Rathbone and R.V. Heatley) pp. 217-223. Blackwell Scientific Pubhcations, Oxford. [Pg.170]

Sobala, G.M., Crabtree, J., Dixon, M.F., Schorah, C.J., Taylor, J.D., Rathbone, B.J., Heatley, R.V. and Axon, A.T.R. (1991a). Acute Helicobacter pylori infection clinical features, local and systemic immune response, gastric mucosal histology and gastric juice ascorbic acid concentrations. Gut 32, 1415-1418. [Pg.171]

Helicobacter pylori normally resides in the human stomach and is transmitted via the fecal-oral route or through ingestion of fecal-contaminated water or food. Infection with HP is more common in developing countries because of crowded conditions and the presence of contaminated food and water. HP colonization does not necessarily reflect an active infection since the organism can attach itself to the gastric epithelium... [Pg.270]

Helicobacter pylori infection and NSAID use account for most cases of PUD. The relatively high incidence of PUD in the elderly maybe due to higher NSAID use. Although hospitalizations related to PUD have decreased over the past two decades, the incidence of PUD-related complications such as bleeding and perforation remain unchanged. [Pg.270]

Chan FKL, Leung WK. Peptic ulcer disease. Lancet 2002 360 933-941. Howden CW, Hunt RH. Guidelines for the management of Helicobacter pylori infection. Ad hoc committee on practice parameters of the American College of Gastroenterology. Am J Gastroenterol 1998 93 2330-2338. [Pg.280]

Laheij RJ, Rossum LG, fanen fB, et al. Evaluation of treatment regimens to cure Helicobacter pylori infection a meta analysis. Aliment Pharmacol Ther 1999 13 857-864. [Pg.280]

Megraud F, Lamouliatte H. The treatment of refractory Helicobacter pylori infection. Aliment Pharmacol Ther 2003 17 1333-1343. Silverstein FE, Graham DY, Senior IR, et al. Misoprostol reduces serious gastrointestinal complications in patients with rheumatoid arthritis receiving non-steroidal anti-inflammatory drugs a randomized, double-blind, placebo-controlled trial. Ann Intern Med 1995 123 241-249. [Pg.280]

Suerbaum S, Micchetti P. Helicobacter pylori infection. N Engl I Med 2002 347 1175-1186. [Pg.280]

Fox, J.G., Beck, P., Dangler, C.A., Whary, M.T., Wang, T.C., Shi, H.N. and Nagler-Anderson, C. (2000) Concurrent enteric helminth infection modulates inflammation and gastric immune responses and reduces helicobacter-induced gastric atrophy. Nature Medicine 6, 536-542. [Pg.398]

L. inocua ferritin site however, is the first described so far that has ligands belonging to two different subunits, and is not contained within a four-helix bundle. Recently it has been suggested that the neutrophil-activating protein of Helicobacter pylori, the major antigen of the immune response in infected individuals, is also a dodecameric ferritin, capable of binding up to 500 iron atoms per oligomer (Tonello et ah, 1999). [Pg.187]

Furuta T, Shirai N, Takashima M et al. Effect of genotypic differences in CYP2C19 on cure rates for Helicobacter pylori infection by triple therapy with a proton pump inhibitor, amoxicillin, and clarithromycin. Clin Pharmacol Ther 2001 69[3] 158—168. [Pg.35]

Mahalanabis D, Rahman MM, Sarker SA, Bardhan PK, Hildebrand P, Beglinger C, et al Helicobacter pylori infection in the young in Bangladesh Prevalence, socioeconomic and nutritional aspects. Int J Epidemiol 1996 25 894-898. [Pg.19]

Lindkvist P, Asrat D, Nilsson I, Tsega E, Ols-son GL, Wretlind B, et al Age at acquisition of Helicobacter pylori infection Comparison of a high and a low prevalence country. Scand J Infect Dis 1996 28 181-184. [Pg.19]

Kawaguchi H, Haruma K, Komoto K, Yoshi-hara M, Sumii K, Kajiyama G Helicobacter pylori infection is the major risk factor for atrophic gastritis. Am J Gastroenterol 1996,91 959-962. [Pg.19]

Haruma K, Kamada T, Kawaguchi H, Okamo-to S, Yoshihara M, Sumii K, et al Effect of age and Helicobacter pylori infection on gastric acid secretion. J Gastroenterol Hepatol 2000 15 277-283. [Pg.19]

Rifaximin Rifamycin Antibiotic Gut bacteria Enteric infection Diarrhea, infectious Hepatic encephalopathy Small intestine bacterial overgrowth Inflammatory bowel disease Colonic diverticular disease Irritable bowel syndrome Constipation Clostridium difficile infection Helicobacter pylori infection Colorectal surgery Bowel decontamination, selective Pancreatitis, acute Bacterial peritonitis, spontaneous Nonsteroidal anti-inflammatory drug enteropathy... [Pg.36]


See other pages where Helicobacter infection is mentioned: [Pg.476]    [Pg.164]    [Pg.334]    [Pg.336]    [Pg.357]    [Pg.497]    [Pg.476]    [Pg.164]    [Pg.334]    [Pg.336]    [Pg.357]    [Pg.497]    [Pg.108]    [Pg.128]    [Pg.96]    [Pg.1035]    [Pg.85]    [Pg.125]    [Pg.182]    [Pg.144]    [Pg.144]    [Pg.159]    [Pg.1373]    [Pg.394]    [Pg.300]    [Pg.36]   


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