Helicobacter pylori

THE GLYCOTYPE FAMILIES OF Helicobacter pylori LIPOPOLYSACCHARIDES EXPRESSING HISTO-BLOOD GROUPS STRUCTURE, BIOSYNTHESIS, AND ROLE IN PATHOGENESIS  

Institute for Biological Sciences, National Research Council, 100 Sussex Drive, Ottawa, Ontario K1A 0R6, Canada 

The Chemical Structure ot Helicobacter pylori Type Strain Lipopolysaccharide  

The Lewis Y and Sialyl Lewis X in Helicobacter pylori Lipopolysaccharides. 118 

Semi Rough-Fonn and Smooth-Form Lipopolysaccharides in Helicobacter pylori  

The stomachs of about half of the people in the world are colonized by H. pylori, a Gram-negative orgaiusm that is assigned as a Class 1 carcinogen. While most 

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Helicobacter pylori is a spiral shaped bacterium that lives in the stomach and duodenum (the section of intestine just below the stomach). Most bacteiia cannot live in the stomach since the environment is too acidic to survive. However, Helicobacter pylori has large amounts of urease, which converts urea to ammonia and carbon dioxide. Ammonia is used to maintain the periplasmic and cytoplasmic pH of the bacterium at neutrality, so this bacterium can survive. Helicobacter pylori causes peptic... 

Helicobacter Pylori Helix Bundle Helix-loop-helix Motif Helper T Cells Hemangioblast Hematopoiesis... 

The proton pump inhibitors are particularly important in the treatment of Helicobacter pylori in patients with active duodenal ulcers. Helicobacter pylori (H. pylori) has been implicated as a causative organism in a type of chronic gastritis and in a large number of cases of peptic and duodenal ulcers. 

A breath test, used to determine the presence of Helicobacter pylori, associated with stomach ulcers, is an example of a diagnostic product involving separate drug and device components. To perform the test, a patient swallows some isotopically (e.g. 

The pathogenesis of rosacea is multifactorial. Vascular hyperactivity is the primary phenomenon that is complicated by inflammatory changes. Endocrine, psychological, pharmacological, immunological, infectious, thermal and alimentary factors contribute to produce vascular instability and tissue damage (Fig. 17.1). The role of Helicobacter pylori is still being discussed. 

Detection and identification of Helicobacter pylori and Demodex folliculorum (Fig. 17.13). 

Schilling CH, Covert MW, Famili I, Church GM, Edwards JS, Palsson BO. Genome-scale metabolic model of Helicobacter pylori 26695. J Bacteriol 2002 184 4582-93. 

Excessive secretion of gastric acid, associated with Helicobacter pylori infection, can result in the development of gastric and duodenal ulcers small changes in the composition of bile can result in crystallization of cholesterol as gallstones failure of exocrine pancreatic secretion (as in cystic fibrosis) leads to undernutrition... 

This genus, originally grouped with the Campylobacters (section 6.4.16), is now considered a separate genus. Helicobacter pylori is of interest as a cause of peptic ulcer. 

FAHEY J W, HARISTOY X, DOLAN P M, KENSLER T W, SCHOLTUS I, STEPHENSON K K, TALALAY p and LOZNIEWSKI a (2002) Sulforaphane inhibits extracellular, intracellular and antibiotic-resistant strains of Helicobacter pylori and prevents benzo[a]pyrene-induced stomach tumors , Proc Natl Acad Sci USA, 99 7610-15. 

Nagata K, H Yu, M Nishikawa, M Kashiba, A Nakamura, EE Sato, T Tamura, M Inoue (1998) Helicobacter pylori generates superoxide radicals and modulates nitric oxide metabolism. J Biol Chem 273 14071-14073. 

Helicobacter pylori is associated with chronic gastritis, peptic ulceration and possibly involved in the pathogenesis of gastric carcinoma (Correa and Ruiz, 1992 Dixon,... 

Correa, P. and Ruiz, B. (1992). Helicobacter pylori and gastric cancer. In Helicobacter pylori and Gastroduodenal disease (eds. B.J. Rathbone and R.V. Heatley) pp. 158-164. Black-well Scientific Publications, Oxford. 

Davies, G.R., Stevens, T.R.J., Lawrenson, L, Banatvala, N., Powell, M. and Rampton, D.S. (1992c). Activation of human jDeripheral blood neutrophils by Helicobacter pylori in vitro. Gut 33, S68. 

Sobala, G.M., Crabtree, J., Dixon, M.F., Schorah, C.J., Taylor, J.D., Rathbone, B.J., Heatley, R.V. and Axon, A.T.R. (1991a). Acute Helicobacter pylori infection clinical features, local and systemic immune response, gastric mucosal histology and gastric juice ascorbic acid concentrations. Gut 32, 1415-1418. 

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