Candida albicans strain

Perea S, JL Lopez-Ribot, WR Kirkpatrick, RK McAtee, RA Santillan, M Martinez, D Calabrese, D Sanglard, TP Patterson (2001) Prevalence of molecular mechanisms of resistance to azole antifungal agents in Candida albicans strains displaying high-level fluconazole resistance isolated from human immunodeficiency virus-infected patients. Antibicrob Agents Chemother 45 2676-2684. [Pg.179]

L. Millon, A. Manteaux, G. Reboux, C. Drobacheff, M. Monod, T. Barale, and Y. Michel-Briand, Fluconazole-resistant recurrent oral candidiasis in human immunodeficiency virus-positive patients persistence of Candida albicans strains with the same genotype,. Clin. Microbiol., 32, 1115, 1994. [Pg.116]

Donovan M, Schumuke JJ, Fonzi WA, Bonar SL, Gheesling-Mullis K, Jakob GS, Davisson VJ, Dotson SB Virulence of a phosphoribosylaminoimidazole carboxylase-deficient Candida albicans strain in an iimnimosuppressed murine model of systemic candidiasis. Infect Immun 2001 69 2542-2548. [Pg.124]

Lay J, Henry LK, Clifford J, Koltin Y, Bulawa CE, Becker JM Altered expression of selectable marker URA3 in gene-dismpted Candida albicans strains complicates interpretation of vimlence studies. Infect Iimnim 1998 66 5301-5306. [Pg.124]

Ray TL, Payne CD, Soil DR Variable expression of Candida acid proteinase by switch phenotypes of individual Candida albicans strains. Clin Res 1988 36 687. [Pg.125]

Calderone RA, Wadsworth E Characterization of mannoproteins from a virulent Candida albicans strain and its derived, avimlent strain. Rev Infect Dis 1988 10 S423-S427. [Pg.125]

Borg-von Zepelin M, Meyer I, Thomssen R, Wurzner R, Sanglard D, Telenti A, Monod M HIV-protease inhibitors reduce cell adherence of Candida albicans strains by inhibition of yeast secreted aspartic proteases. J Invest Dermatol 1999 13 747-751. [Pg.127]

Kohler Fink "Candida albicans strains heterozygous and homozygous for mutations in mitogen-activated protein kinase signalling components have defects in hyphal development", Proc. Natl. Acad. Sci. U.S.A., 1996,21, 13223. [Pg.89]

Franz, R., M. Ruhnke, D.C. Lamb, S.L. Kelly, and J. Morschhauser (1998). Multiple molecular mechanisms contribute to a stepwise development of fluconazole resistance in clinical Candida albicans strains. Antimicrob. Agents Chemother. 42, 3065-3072. [Pg.618]

C. LassFlorl, and E. Einsele (1997). Molecular analysis of CYP51 from fluconazole-resistant Candida albicans strains. FEMS Lett. 151, 263-268. [Pg.618]

The mannans isolated from Candida albicans strains, NIH A-207 (serotype A), NIH B-792 (serotype B), and J-1012 (serotype C), were fractionated on a column of DEAE-Sephadex into five subfractions containing different amounts of phosphate. [Pg.110]

A structural investigation, by H-n.m.r. spectroscopy, of matmo-oligosaccharides released by mild acid hydrolysis from the phosphopeptidomaiman of a Candida albicans strain and ranging from ttiose to heptaose, showed them to be of the (l- 2)-linked type. Some general rules pertaining to... [Pg.271]

Capric acid (C10 0) and lauric acid (C12 0) have antimicrobial activity against Candida albicans strains (Bergsson et al., 2001). [Pg.168]

Yucesoy, M. Esen, N. Yulug, N. Use of chromogenic tube and methyl blue-sabouraud agar for the identification of Candida albicans strains. Kobe J. Med. Sci. 2001, 47, 161-167. [Pg.25]

Shibata, N., Kobayashi, H., Tojo, M. and Suzuki, S. (1986) Characterization of phosphomannan-protein complexes isolated from viable cells of yeast and mycelial forms of Candida albicans NIH B-792 strain by the action of Zymolyase-IOOT. Archives of Biochemistry and Biophysics, 251 (2), 697-708. [Pg.55]

Mullick, A. et al., Dysregulated inflammatory response to Candida albicans in a C5-deficient mouse strain, Infect. Immun., 72, 5868, 2004. [Pg.94]

Standard and the isolated strains of the following bacteria, namely Escherichia coli (ATCC 35218), Pseudomonas aeruginosa (ATCC 10145), Proteus mirabilis (ATCC 7002), Klebsiella pneumoniae (RSKK 574), Acinetobacter baumannii (RSKK 02026), Staphylococcus aureus (ATCC 25923), and Enterococcus faecalis (ATCC 29212) for determination of antibacterial activity, along with standard strains of Candida albicans (ATCC 10231) and C. parapsilosis (ATCC 22019) were used for determination of antifungal activity. [Pg.99]

The combined synergistic effects of cyclo(Leu-Pro) and cyclo(Phe-Pro) were effective against five vancomycin-resistant enterococci (VRE) strains Enterococcus faecium (K-99-38), E. faecalis (K-99-17), E. faecalis (K-99-258), E. faecium (K-01-312), and E. faecalis (K-01-511) with MIC values of 0.25—1 mgl . It also showed activity against E. coli, Staphylococcus aureus. Micrococcus luteus, Candida albicans, and Cryptococcus neoformans with MIC values of 0.25—0.5 mg 1. This combination also showed mutagenic activity against Salmonella typhimurium TA98 and TAIOO strains in a Salmonella mutation assay. ... [Pg.683]

Spirocyclic C-aryl glycosides are central structural skeletons of papulacandins that constitute a family of novel antifungal antibiotics isolated from a strain of Papularia sphaerosperna. They have shown potent in vitro antifungal activity against Candida albicans, Candida tropicalis, Pneumocystis carinii, among other microorganisms [110],... [Pg.40]

Candida albicans (gene PGKl [50] strain 3153A [50]) [50]... [Pg.285]

Y. Kondoh, K. Shimizu, and K. Tanaka, Proteinase production and pathogenecity of Candida albicans II. Virulence for mice of C. albicans strains of different proteinase activity, Microbial. Immuno., 31, 1061, 1987. [Pg.116]

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