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Adenosine monophosphate mouse

A/J A Jackson inbred mouse strain ALP Anti-leukoprotease ALS Amyotrophic lateral sclerosis cAMP Cyclic adenosine monophosphate also known as adenosine 3, 5 -phosphate AM Alveolar macrophage AML Acute myelogenous leukaemia AMP Adenosine monophosphate AMVN 2,2 -azobis (2,4-dimethylvaleronitrile)... [Pg.279]

Traub O, Look J, Paul D, Willecke K Cyclic adenosine monophosphate stimulates biosynthesis and phosphorylation of the 26 kDa gap junction protein in cultured mouse hepatocytes. Eur J Cell Biol 1987 43 48-54. [Pg.137]

The measurement of TSH was originally based on bioassays such as the stimulation of colloid droplet formation in the guinea pig thyroid gland and the release of labeled thyroidal iodide into mouse blood. These early in vivo bioassays, however, were of limited sensitivity and precision and were not applicable to the measurement of TSH in unfractionated serum. Most TSH bioassays have involved the in vitro stimulation of thyroid cyclic adenosine monophosphate (cAMP) or adenylate cyclase activity. The rat FRTL-5 thyroid cell line is an example of a particularly convenient and precise assay system. Unfortunately, such methods require purification and concentration of TSH from serum before assay. Sensitive detection of TSH in unfractionated serum is possible using a cytochemical bioassay, but this procedure is technically difficult and time-consuming. At present, immunoassay is the procedure of choice for the measurement of serum TSH in the clinical laboratory. [Pg.2066]

Culpepper, R.M. and Andreoli, T.E. (1983). Interactions among PGE2, antidiuretic hormone and cyclic adenosine monophosphate in modulating Cl absorption in the single mouse medullary thick ascending limbs of Henle. ]. Clin. Invest., 71, 1588—1600... [Pg.55]

Chlorpromazine arrests cultured cells in mitosis and disorganises the organised microtubule structure produced by cyclic adenosine monophosphate (Poffenbarger and Fuller 1977). It causes a reduction in the number of microtubules in spinal ganglion cells (Edstrom et al. 1973, Thyberg et al. 1977) and neuroblastoma cells (EdstrOm et al. 1975) in vitro. The micellar form of chlorpromazine interacts preferentially with one site on brain tubulin (Gann et al. 1981). Ghlorpromazine has been shown to bind reversibly to tubulin prepared from mouse brain via two well-resolved processes (Hin-... [Pg.249]

Jeppesen PB, Gregersen S, Poulsen CR, Hermansen K (2000) Stevioside acts directly on pancreatic (3 cells to secrete insulin actions independent of cyclic adenosine monophosphate and adenosine triphosphate-sensitive K-l—channel activity. Metabolism 49 208-214 Abudula R, Jeppesen PB, Rolfsen SED, Xiao J, Hermansen K (2004) Rebaudioside A potentially stimulates insulin secretion from isolated mouse islets studies on the dose-, glucose-, and calcium-dependency. Metabolism 53 1378-1381... [Pg.2688]

Martinez-Morales JR, Morales A, Marin R, Hern4ndez-Jim nez JG, Acevedo A, Guerra B, Hernandez G, Ldpez-Coviella I, Prieto L, Alonso R (2001) Estrogen modulates norepinephrine-induced accumulation of adenosine cyclic monophosphate in a subpopulation of immortalized luteinizing hormone-releasing hormone secreting neurons from the mouse hypothalamus. Neurosci Lett 298 61-64... [Pg.145]

Tissue electrodes [2, 3, 4, 5, 45,57], In these biosensors, a thin layer of tissue is attached to the internal sensor. The enzymic reactions taking place in the tissue liberate products sensed by the internal sensor. In the glutamine electrode [5, 45], a thick layer (about 0.05 mm) of porcine liver is used and in the adenosine-5 -monophosphate electrode [4], a layer of rabbit muscle tissue. In both cases, the ammonia gas probe is the indicator electrode. Various types of enzyme, bacterial and tissue electrodes were compared [2]. In an adenosine electrode a mixture of cells obtained from the outer (mucosal) side of a mouse small intestine was used [3j. The stability of all these electrodes increases in the presence of sodium azide in the solution that prevents bacterial decomposition of the tissue. In an electrode specific for the antidiuretic hormone [57], toad bladder is placed over the membrane of a sodium-sensitive glass electrode. In the presence of the antidiuretic hormone, sodium ions are transported through the bladder and the sodium electrode response depends on the hormone concentration. [Pg.205]

Herrmann-Erlee, M. P. M. A parathyroid hormone-like action of dibutyryl cyclic adenosine-3 ,5 monophosphate on the explanted embryonic mouse radius. Calc. Tiss. Res. 4, (Suppl.) 70 (1970)... [Pg.124]

Foscarnet competitively inhibits Na+-Pj cotransport in rat, mouse, dog, rabbit, and human renal proximal tubule brush border membrane vesicles, reversibly inhibiting sodium-dependent phosphate transport [37, 38]. Renal cortical Na-K-ATPase and alkaline phosphatase activity are not inhibited by foscarnet, nor is proUne, glucose, succinate, or Na+ transport [37, 38]. Foscarnet induces isolated phosphatuiia without hypophosphatemia in thyroparathyroidectomized rats maintained on a low phosphorus diet, without affecting glomerular filtration rate, urinary adenosine 3 5 -cychc monophosphate (cAMP) activity, or urinary calcium, sodium or potassium excretion [37,39]. Sodium-Pj cotransport in brush border membrane vesicles from human renal cortex was reported to be even more sensitive to inhibition by foscarnet than in rat renal brush border membrane vesicles [38]. [Pg.252]

Clem, B. F., Hudson, E. A., and Clark, B. J. (2005). Cyclic adenosine 3 ,5 -monophosphate (cAMP) enhances cAMP-responsive element binding (CREB) protein phosphorylation and phospho-CREB interaction with the mouse steroidogenic acute regulatory protein gene promoter. Endocrinology 3, 1348-1356. [Pg.405]

Salas VM, Corcoran GB (1997) Calcium-dependent DNA damage and adenosine 3 5 -cyclic monophosphate- independent glycogen phosphorylase activation in an in vitro model of acetaminophen-induced liver injury. Hepatology 25 1432-1438 Salminen WF Jr, Voellmy R, Roberts SM (1998) Effect of N-acetylcysteine on heat shock protein induction by acetaminophen in mouse liver. J Pharmacol Exp Ther 286 519-524 Schiodt FV, Ott P, Christensen E, Bondesen S (2002) The value of plasma acetaminophen half-life in antidote-treated acetaminophen overdosage. Clin Pharmacol Ther 71 221-225 Schnellmann JG, Pumford NR, Kusewitt DF, Bucci TJ, Hinson JA (1999) Deferoxamine delays the development of the hepatotoxicity of acetaminophen in mice. Toxicol Lett 106 79-88 Shen HM, Pervaiz S (2006) TNF receptor superfamily-induced cell death redox-dependent execution. FASEB J 20 1589-1598... [Pg.404]


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See also in sourсe #XX -- [ Pg.367 ]




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