Secondary magnesium phosphate

Synonyms magnesium hydrogen phosphate secondary magnesium phosphate... 

Magnesium Phosphate, Dihasic. Magnesium hydrogen phosphate secondary magnesium phosphate. HMg-0,P mol wt 120.29. H 0,84%, Mg 20.21%, O 53.20%, P 25.74%. MgHPO,. Trihydrate occurs in nature as the min -erals newberyite, phosphor-roesslerite. 

Secondary magnesium phosphate. See Magnesium phosphate dibasic Secondary propyl alcohol. See Isopropyl alcohol... 

Tocopherols and tocotrienols can be separated on silica gel but also, sometimes with greater efficacy, on special phases such as secondary magnesium phosphate or mixtures of silica gel/alumina with zinc carbonate. The tocopherols migrate in the order a-T > p-T > y-T > 5-T (descending Rf values) and run ahead of the more polar tocotrienols. Reversed-phase systems based on silica gel/kieselguhr... 

Magnesium phosphate secondary. See Magnesium phosphate dibasic Magnesium phosphate tertiary. See Magnesium phosphate tribasic Magnesium phosphate tribasic CAS 7757-87-1 (anhyd ) 10233-87-1 (pentahydrate) EINECS/ELINCS 231-824-0 INS342(iii)... 

Advanced stages Increased potassium, phosphorus, and magnesium decreased bicarbonate (metabolic acidosis) calcium levels are generally low in earlier stages of CKD and may be elevated in stage 5 CKD, secondary to the use of calcium-containing phosphate binders. 

Many of the adverse effects of lithium can be ascribed to the action of lithium on adenylate cyclase, the key enz)nne that links many hormones and neurotransmitters with their intracellular actions. Thus antidiuretic hormone and thyroid-stimulating-hormone-sensitive adenylate cyclases are inhibited by therapeutic concentrations of the drug, which frequently leads to enhanced diuresis, h)rpoth)n oidism and even goitre. Aldosterone synthesis is increased following chronic lithium treatment and is probably a secondary consequence of the enhanced diuresis caused by the inhibition of antidiuretic-hormone-sensitive adenylate cyclase in the kidney. There is also evidence that chronic lithium treatment causes an increase in serum parathyroid hormone levels and, with this, a rise in calcium and magnesium concentrations. A decrease in plasma phosphate and in bone mineralization can also be attributed to the effects of the drug on parathyroid activity. Whether these changes are of any clinical consequence is unclear. 

A variety of adverse effects have been reported following the use of antacids. If sodium bicarbonate is absorbed, it can cause systemic alkalization and sodium overload. Calcium carbonate may induce hypercalcemia and a rebound increase in gastric secretion secondary to the elevation in circulating calcium levels. Magnesium hydroxide may produce osmotic diarrhea, and the excessive absorption of Mg++ in patients with renal failure may result in central nervous system toxicity. Aluminum hydroxide is associated with constipation serum phosphate levels also may become depressed because of phosphate binding within the gut. The use of antacids in general may interfere with the absorption of a number of antibiotics and other medications. 

Hypophosphatemia and phosphate depletion may result from inadequate intestinal phosphate absorption. Patients taking aluminum- or magnesium-containing antacids may develop hypophosphatemia, because these antacids bind phosphate in the intestine, rendering it nonabsorbable. The hypophosphatemia observed in patients with malabsorption maybe more closely related to their secondary hyperparathyroidism than to malabsorption of phosphate. Because phosphate is abundant in most foods, dietary deprivation is not usually a cause of phosphate depletion in patients with normal intestinal function and an adequate diet. 

For a specific waste to be treated, the appropriate ratio of the treatment additive (phosphate) and pH control agent (magnesium oxide) that gives the best performance in terms of maximum contaminant level (MCL) and the secondary maximum contaminant levels (SMCL) for drinking water of the EPA s National Primary and Secondary Drinking Water Regulations is determined experimentally. One hundred grams of the waste is blended... 

Phosphates 51% primary (H2PO4), 39% secondary (HPO ") and 10% bound to calcium and magnesium. 

Alkylbenzene sulfonate, paraffin sulfonate, secondary alkanesulfonate, soap, ethoxylated alcohol sulfate, alcohol-sulfate, dialkyldiphenylether disulfonate Ethoxylated phenol, ethoxylated alcohol, amine oxide, alkanolamide, alkyl polyglucoside, alkyl polyalkylene glycol ether Phosphate, carbonate, bicarbonate, silicate, citrate, gluconate, polyacrylate, EDTA-salt, phosphonate Alcohol, alcohol glycol ether, pine oil, rt-limonene Sodium/potassium hydroxide, alkanol-amine, ammonia, magnesium hydroxide Sodium xylenesulfonate, sodium cumene sulfonate, dialkyldiphenylether disulfonate... 

A large variety of secondary uranium minerals are known, many are brilliantly colored and fluorescent. The commonest are gummite (a general term like limonite for mixtures of various secondary hydrated uranium oxides with impurities) hydrated uranium phosphates of the phosphuranylite type, including autunite (with calcium), saleeite (magnesium), and torbernite (with copper) and hydrated uranium silicates such as cof-finite, uranophane (with calcium), and sklodowskite (magnesium). 

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