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Salmonella adherence

The rate of Salmonella recovery on meat surfaces can be seen in Fig. 4. The results show that dry heat treatment at 90 °C had only a limited cidal effect on Salmonella adhering to various food surfaces. In contrast, steam treatment prevented bacterial recovery for at least 24 h on all food surfaces tested. [Pg.371]

Human microbial pathogens that possess the ability to adhere to host tissues have a distinct advantage over those that do not, in that they are better equipped to evade and resist the defense systems of their host. There are numerous adherence mechanisms that have been described to date. However, those that are most commonly studied originate from pafhogens that colonize the GIT and genital-urinary tract, including Salmonella, H. pylori, and pathogenic serotypes of E. coli. [Pg.117]

The adherence mechanisms involved in Salmonella infection have been studied in great deal. Disease associated with S. enterica serovars is initiated by attachment to and invasion of hosf cells, followed by subse-quenf inflammation of the lamina propria and lymph nodes (Darwin and Miller, 1999). Several genetically defined fimbrial or piliar adhesins con-tribufe fo fhe initial attachment and the overall infection process of Salmonella. Some of fhese include t)q)e 1 fimbriae (Fim), plasmid-encoded (PE) fimbriae, long polar (LP) fimbriae, and thin aggregative fimbriae (curli). However, many ofher putative fimbrial operons have been identified within various S. enterica serovar genomes, but the expression of fhese proteins is currently undefined. [Pg.117]

The PE fimbriae, encoded by the pefBACD operon contained on the virulence plasmid pSLT, has been found in only four Salmonella serotypes, S. typhimurium, S. choleraesuis, S. paratyphi, and S. enteritidis (Baumler etal, 1997). The PE fimbria has been demonstrated to mediate adherence to the... [Pg.118]

Recently, a non-fimbrial adhesin, SiiE, has been identified in S. enterica serovar T)q)himurim. Although little is known about SiiE, it has been found to mediate contact-dependent adhesion to HeLa cell surfaces (Gerlach et al., 2007). SiiE is a type 1 secretion system (TISS) secreted protein encoded m the Salmonella pathogenicity island 4 and might functionally resemble the type 1 fimbrial adhesins. More work is needed to elucidate the true role of SiiE in adherence in vivo. [Pg.119]

Giannasca, K. T., Giannasca, P. J., and Neutra, M. R. (1996). Adherence of Salmonella Typhimurium to Caco-2 cells Identification of a glycoconjugate receptor. Infect. Immun. 64,135-145. [Pg.146]

Bessler, H. C., de Oliveira, I. R., and Giugliano, L. G. (2006). Human milk glycoproteins inhibit the adherence of Salmonella typhimurium to HeLa cells. Microbiol. Immunol. 50, 877-882. Bhutta, Z. A., and Yusuf, K. (1997). Early-onset neonatal sepsis in Pakistan A case control study of risk factors in a birth cohort. Am. ]. Perinatol. 14, 577-581. [Pg.70]

Stavric, S., Gleeson, T.M., Blanchfield, B., and Pivnick, P. 1987. Role of adhering microflora in competitive exclusion of Salmonella from young chicks. J. Food Prot. 50, 928-932. [Pg.30]

Hood, S.K. and Zottolla, E.A. 1997. Growth media and surface conditioning influence the adherence of Pseudomonas fragi, Salmonella typhimurium and Listeria monocytogenes cells to stainless steel. Journal of Food Protection 60 1034-1037. [Pg.161]

Campylobacter spp. are labile in acidic environments, much like Salmonella. Therefore, an inoculum of approximately 800 organisms is required to initiate infection. Conditions in the upper small intestine are favorable for multiplication. Flagella-mediated adherence and tissue invasion by bacteria have been demonstrated in the jejunum, ileum, and colon. Infection results in an acute inflammatory enteritis. C. jejuni can produce an enterotoxin or cytotoxin. Both cytotox-ins and enterotoxins may be produced in many strains. Symptom manifestation depends on immunity. Patients infected with Campylobacter develop specific IgG, IgM, and IgA antibodies in serum and IgA antibodies in intestinal secretions. Volunteer studies indicate that immunity does protect against illness. [Pg.2046]

Following the entry of Salmonella bacteria in the gastrointestinal tract, they adhere by fibriae to the epithelial cells of intestinal mucosa. They are transported to the lamina propria by endocytosis where they are released inducing an influx of... [Pg.128]

DiPetrillo, M. D., Tibbetts, T., Kleanthous, H., Killeen, K. P. and Hohmann, E. L. 1999, Safety and immunogenicity of phoP/phoQ-deleted Salmonella typhi expressing Helicobacter pylori urease in adult volunteers. Vaccine, 18 449-459 Donahue, J. P., Israel, D. A., Torres, V. J., Necheva, A. S. and Miller, G. G. 2002, Inactivation of a Helicobacter pylori DNA methyltransferase alters dnaK operon expression following host-cell adherence. FEMS Microbiol.Lett., 208 295-301 Donati, M., Moreno, S., Stomi, E., Tucci, A., Poll, L., Mazzoni, C., Varoli, O., Sambri,... [Pg.334]

To determine whether the mutagenic activity was due to conversion of cycasin to MAM by intestinal flora, as had been reported for carcinogenic action, an antibiotic was used to partially sterilize the large intestine. Mice were treated with (a) cycasin, 10 mg orally, 2 hr before Salmonella (b) ampicillin, 2 mg orally, 20 and 40 hr before Salmonella (c) neither compound or (d) both compounds. Intestinal bacteria were enumerated by using the caecum contents as an indicator of its population. After the peritoneal fluid was withdrawn from four mice, the caeca were removed, rinsed in sterile saline to remove adhering Salmonella and minced in 10.0 ml saline. Samples were then treated, using standard bacteriological procedures, and intestinal contents, prototrophic and auxotrophic, were plated in triplicate. [Pg.288]

Fimbriae were identified as the filaments responsible for developing bacterial adhesion to other cells or surfaces. Various types were distinguished, produced by E. coli, salmonella and many other types of bacteria. Typically these projections were 2 pm long and 7nm in diameter, with up to 400 per cell. When the bacterial adhesion was measured by a blood cell aggregation test, or by a microscope counting of adhering cells, the fimbriae were inhibited by D-mannose addition. [Pg.283]

Solano C., Sesma B., Alvarez M., Humphrey T.J., Thoms C.J., and Gamazo C. 1998. Discrimination of strains of Salmonella enteritidis with differing levels of virulence by an in vitro glass adherence test. J Clin Microbiol 36 674-678. [Pg.121]

Ln vitro studies on HT-29 epithelial cells were made in order to test the capacity of L. johnsonii strains to decrease S. enterica serovar typhimurium UKl-lux invasion (Muller et al., 2011). It was shown that the L. johnsonii proved its exclusion properties just when the probiotic bacteria are growing in minimal medium supplemented with fatty acids. In these conditions, L. johnsonii reduced significantly Salmonella spp. adhesion and invasion. If the L. johnsonii was grown in a medium supplemented with linoleic and linolenic acids, its adhesion was also possible even when the HT-29 cells were infected with Salmonella spp. (Muller et al., 2011). The mechanism proposed in order to explain how probiotics prevent pathogens from infecting cells is that they sterically hinder the adherence of pathogens (Foumiat et al., 1992). [Pg.173]


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See also in sourсe #XX -- [ Pg.117 ]




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