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Acute myeloid

Enzymes Degrading Macromolecules. Enzymes that degrade macromolecules such as membrane polysaccharides, stmctural and functional proteins, or nucleic acids, have all shown oncolytic activity. Treatment strategies include the treatment of inoperable tumors with pepsin (1) antitumor activity of carboxypeptidase (44) cytotoxicity of ribonudease (45—47) oncolytic activity of neuraminidase (48—52) therapy with neuraminidase of patients with acute myeloid leukemia (53) antitumor activity of proteases (54) and hyaluronidase treatment in the management of human soHd tumors (55). [Pg.308]

Moore J, Seiter K, Kolitz J et al (2006) A Phase II study of Bcl-2 antisense (oblimersen sodium) combined with gemtuzumab ozogamicin in older patients with acute myeloid leukemia in first relapse. Leuk Res 30(7) 777-783... [Pg.188]

Seggewiss R, Pittaluga S, Adler RL, Guenaga FJ, Ferguson C, Pilz I, Ryu B, Sorrentino BP, Young WS, Donahue RE, von Kalle C, Nienhuis AW, Dunbar CE (2006) Acute myeloid leukemia associated with retroviral gene transfer to hematopoietic progenitor cells of a rhesus macaque. Blood 107 3865-3867... [Pg.296]

Raponi M, Belly RT, Karp JE, Lancet JE, Atkins D, Wang Y. Microarray analysis reveals genetic pathways modulated by tipifarnib in acute myeloid leukemia. BMC Cancer 2004 4 56. [Pg.161]

Acute myeloid leukemia CD13, CD15, CD33, CD14, CD64, and C-KIT... [Pg.1401]

Gregory J, Arced R. Acute myeloid leukemia in children A review of risk factors and recent trials. Cancer Invest 2002 20 1027-1037. [Pg.1413]

Ishii E, Kawasaki H, Isoyama K, et al. Recent advances in the treatment of infant acute myeloid leukemia. Leukemia Lymphoma 2003 44 741-748. [Pg.1413]

Cancers Multiple myeloma Non-Hodgkin s lymphoma Hodgkin s disease Acute myeloid leukemia Neuroblastoma Germ cell tumors Acute myeloid leukemia Acute lymphoblastic leukemia Chronic myeloid leukemia Myelodysplastic syndrome Myeloproliferative disorders Non-Hodgkin s lymphoma Hodgkin s disease Chronic lymphocytic leukemia Multiple myeloma... [Pg.1448]

GCSF, granulocyte colony stimulating factor GMCSF, granulocyte-macrophage colony stimulating factor SQ, subcutaneous IV, intravenous AML, acute myeloid leukemia. [Pg.1471]

HT is a 55-year-old man with newly diagnosed acute myeloid leukemia. The patient is asymptomatic at this time. Laboratory results reveal the following significant findings ... [Pg.1486]

Acute myeloid leukemia (especially white blood cell count greater than 50/mm3)... [Pg.1486]

American Conference of Governmental Industrial Hygienists Absorption, Distribution, Metabolism, and Excretion acute myeloid leukemia atmosphere... [Pg.173]

Sterkers Y, Preudhomme C, Lai JL, Demory JL, Caulier MT, Wattel E, Bordessoule D, Bauters L, Lenaux P. Acute myeloid leukemia and myelodys-plastic syndromes following essential thrombocythemia treated with hydroxyurea high proportion of cases with 17p deletion. Blood 1998 91 616-622. [Pg.249]

Davies SM, Robison LL, Buckley JD et al. Glutathione S-transferase polymorphisms and outcome of chemotherapy in childhood acute myeloid leukemia. J Clin Oncol 2001 19 1279-1287. [Pg.308]

Liu P, Hajra A et al. Fusion between transcription factor CBF beta/PEBP2 beta and a myosin heavy chain in acute myeloid leukemia. Science 1993 261 1041-1044. [Pg.414]

Bross, P.F., Beitz, J., Chen, G., Chen, X.H., Duffy, E., Kieffer, L., Roy, S., Sridhara, R., Rahman, A., Williams, G., and Pazdur, R. (2001) Approval summary Gemtuzumab ozogamicin in relapsed acute myeloid leukemia. Clin. Cancer Res. 7, 1490-1496. [Pg.1051]

Ajoene has antitumor activity, inhibits cholesterol biosynthesis, modulates membrane-dependent functions of immune cells, inhibits protein prenylation83 and is an anti-leukaemia agent for acute myeloid leukaemia.85 In antithrombotic assays, the Z isomer is more active than the E isomer.84... [Pg.692]

Further studies have shown additional cancer types, most notably ovarian and bladder cancer, non-Hodgkin s lymphoma and acute myeloid leukaemia, to be at least partially responsive to IL-2 treatment. However, a persistent feature of clinical investigations assessing IL-2 effects on various cancer types is variability of response. Several trials have yielded conflicting results, and no reliable predictor of clinical response is available. [Pg.248]

Han, S. S. et al., Arsenic trioxide represses constitutive activation of NF-kappaB and COX-2 expression in human acute myeloid leukemia, HL-60, J. Cell. Biochem., 94, 695, 2005. [Pg.289]

See other pages where Acute myeloid is mentioned: [Pg.438]    [Pg.151]    [Pg.268]    [Pg.1076]    [Pg.590]    [Pg.260]    [Pg.347]    [Pg.149]    [Pg.257]    [Pg.315]    [Pg.1292]    [Pg.1294]    [Pg.1299]    [Pg.1382]    [Pg.1413]    [Pg.1448]    [Pg.296]    [Pg.607]    [Pg.337]    [Pg.301]    [Pg.212]    [Pg.250]    [Pg.272]    [Pg.381]    [Pg.385]   
See also in sourсe #XX -- [ Pg.608 ]



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Acute myelogenous/myeloid leukemia

Acute myeloid leukaemia

Acute myeloid leukemia

Acute myeloid leukemia cell

Acute myeloid leukemia cell proliferation

Acute myeloid leukemia chemotherapy

Acute myeloid leukemia classification

Acute myeloid leukemia clinical presentation

Acute myeloid leukemia evaluation

Acute myeloid leukemia patients

Acute myeloid leukemia prognosis

Acute myeloid leukemia remission induction

Acute myeloid leukemia treatment

Cancer acute myeloid leukemia

Detection of Acute Myeloid Leukemia

Genetic acute myeloid leukemia

Myeloid

Tumor acute myeloid leukemia

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