Strokes description

We begin with a broad-stroke description of the different strategies for using quantum mechanical interference to control product selectivity in a chemical reaction. 

Hypertension is a common and progressive disorder which, if not effectively treated, results in an increased risk of atherosclerosis (see above), haemorrhagic stroke and damage to the kidney. For most cases of hypertension, there is no obvious cause, hence it is known as essential hypertension, so called because it was originally thonght to be essential to maintain tissue perfusion. In order to better nnderstand the regulation of blood pressure, a brief description of the regulation of contraction of smooth mnscle is provided. 

The main differences between necrosis and apoptosis are in the triggers (accidental v. physiological), the process (energy-independent vs. dependent), and the outcomes (with vs. without inflammation). However, apoptosis and/or necrosis can be induced by the same causes in some cases (K16). Alteration of mitochondrial permeability is involved in both apoptosis and necrosis (K16). Both apoptosis and necrosis are found in conditions such as stroke and myocardial infraction (F5), and necrosis can occur secondary to apoptosis (T4). To preserve the usefulness of the two terms for denoting different modes of cell death while still recognizing possible overlap of the two processes (H7), some more descriptive terms have been proposed primary necrosis (oncosis, ischemic cell death) (Ml) and secondary necrosis (apoptotic necrosis, necrosis secondary to apoptosis) (K15). 

With this bold stroke, Boltzmann escaped the futile attempt to describe microscopic molecular phenomena in terms of then-known Newtonian mechanical laws. Instead, he injected an essential probabilistic element that reduces the description of the microscopic domain to a statistical distribution of microstates, i.e., alternative microscopic ways of partitioning the total macroscopic energy U and volume V among the unknown degrees of freedom of the molecular domain, all such partitionings having equal a priori probability in the absence of definite information to the contrary. 

Although many of the proposed applications for these gels requires that they operate under an applied pressure or generate some kind of mechanical force, a detailed understanding of these relationships does not currently exist. There is data available on the effect of the load on the rate of work and stroke, or generated force vs time, for example, but this is often presented on an empirical basis. Furthermore, much of the work has been carried out under conditions where the stimulus is rate-limiting, rather than the polymer network [66, 67], The development of a mathematical description of these phenomena using independently obtainable polymer parameters is needed. 

For the exact description of hemodynamic stroke and identification of individual hemodynamic risk zones, as mentioned earlier, the assessment of the hemodynamic situation is essential. PI offers a means to obtain semiquantitative hemodynamic information in cerebral ischemia with relatively high resolution and short acquisition times covering all cerebral vascular territories. In acute cerebral ischemia dynamic susceptibility contrast... 

I found out much later that after Katerina had arrived home that evening she became extremely ill for several days. From her description of her symptoms, it sounded as if she had suffered from severe heat stroke that was probably brought on or exacerbated by dehydration, although she did not consider this a possible explanation for her illness. 

Apoptosis has been implicated in delayed neuronal death associated with many neurodegenerative disorders such as Parkinson s disease, stroke, Huntington s disease, traumatic head injury, Alzheimer s disease, motor neuron degeneration, spinal cord injury, and multiple sclerosis. Since an extensive description of the role of apoptosis in each of these disorders is beyond the scope of the present text, we shall focus on anti-apoptotic strategies for stroke, Parkinson s disease, and multiple sclerosis. 

Rubin et at, J. Clin. Psychiatry 46, 418 (1985). Clinical trial in post-stroke depression J. R. Lipsey et ai, Lancet 1 297 (1984). Anticholinergic effect and clinical efficacy in urinary dysfunction I. Nissenkom et al., Eur. UroL 12, 109 (1986). Comprehensive description J. L. Hale in Analytical Profiles of Drug Substances vol. 1, K.. Florey, Ed. (Academic Press, New York, 1972) pp 233-247. 

Conclusions Power Stroke Model is Just Plain Wrong as a Description of Chemically... 

The hyperbolic expression can be derived from a picture in which the mechanical motion involves a deterministic power stroke. Although this power stroke model can be generalized to include thermodynamically consistent rate constants (see (3)), the description of the mechanical transitions as deterministic processes is not consistent with microscopic reversibility and is incorrect. 

Descriptions of some of the main secondary diseases following from arteriosclerosis have already been handed down to us from ancient times. Thus, one can find in the Hippocratic writings (Corpus Hippocraticum) indications of gangrene, stroke (apoplexia) and cardiac failure. But the explanation ofthe cause of such diseases first began in early modem times, when autopsies were carried out in increasing numbers to clarify the cause of death. 

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