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Sodium dietary restrictions

Dietary restriction of sodium causes lithium retention and an increased risk of toxicity. The same would be true of a diet that markedly restricted fluid intake. In brief, dietary extremes should be avoided. [Pg.163]

Even more disturbing was a study linking low sodium consumption with an increase in heart attack risk. Working with hypertensive men at the Albert Einstein College of Medicine in New York City, Dr. Michael Alderman detected an unexpectedly high incidence of heart attacks in men who had low amounts of salt in their urine, reflecting their dietary restrictions. The study followed nearly two thousand men for almost four years. More than four times as many heart attacks occurred in men with the lowest amounts of sodium in their urine compared with men who had the highest amounts of urinary sodium. [Pg.128]

Ascites is a defined compartment of the extracellular fluid volume, which is difficult to mobilize. If the reduction in weight is inadequate after appropriate basic therapy (< 1.2 kg after 4 days), stage II should be initiated with the cautious administration of diuretics. The steps already detailed for stage I are to be continued, whereby the intake of dietary sodium is restricted even further (= 3g/day). (s. tab. 16.12)... [Pg.306]

A man with chronic renal insufficiency who followed dietary restriction of potassium developed a raised serum potassium concentration (5.8 mmoPl). He insisted that he had followed his dietary regimen as usual, except for taking noni juice, purchased from a health food store. He was treated with sodium polystyrene sulfonate and told to stop taking noni juice. At the next check-up his potassium was still raised he said that he would never stop taking noni juice and... [Pg.3086]

Regular, frequent, and small balanced meals with complete proteins, essential fatty acids, and complex carbohydrates Low dietary intake of fat and sodium Caffeine restriction Regular exercise Smoking cessation Alcohol restriction Regular sleep... [Pg.1473]

The Food and Nutrition Board has removed the three electrolytes from its table of estimated safe and adequate daily dietary intake because sufficient information is not available to establish a recommended amount. The major dietary source of sodium and chloride is table salt (40% sodium and 60% chloride). Physicians still recommend that the intake of sodium be restricted to 1-2 g daily. The recommended intake of chloride is approximately 1.7-5.1 g daily. However, getting enough sodium and chloride is not a problem. In fact, sodium intake in the United States is about 5-7 g/day, far in excess of the 1-2 g/day required by a normal adult. [Pg.789]

Resodec. A polycarboxylic cation exchange resin. Almost inert, nonabsorbable powder, intended to aid the dietary restriction of salt by removing sodium from the contents of the intestinal tract. [Pg.1296]

Gastrointestinal system Nausea and vomiting Diarrhea Constipation Anorexia Stomatitis (waste buildup) Bleeding (waste buildup, impaired clotting) Parenteral nutrition (if indicated) Enteral nutrition (if indicated) Dietary restriction of potassium (40 mEq or as ordered), sodium, phosphate based on values of labwork Protein intake based on need (0.6-2 g/kg/day)... [Pg.194]

Dietary restriction of sodium—avoid irulk, cheese, canned soup or vegetables, bread, cereal... [Pg.211]

Callstrom et al. (2001) performed laxative-free CT colonography without dietary restrictions in 58 patients. Faecal tagging was obtained with a combination of iodine and barium administered over one or two days. The best results of tagging were obtained by combining 6x225 ml of a 2.1% w/v barium suspension and 1x225 ml dilute diatrizoate meglumine and diatrizoate sodium. All residue with a density >150 H.U. was electronically labelled. In that way a 100% sensitivity for lesions >1 cm was obtained (5/5 lesions). [Pg.48]

Dietary modifications—Rigid, long-term sodium (salt) restriction can be successfully used to control, at least in part, high blood pressure. [Pg.558]

Potassium is widely distributed in foods. Meat, poultry, fish, many fruits, whole grains, and vegetables are good sources. (Although meat, poultry, and fish are good sources of potassium, these items may be restricted when dietary sodium is restricted.)... [Pg.679]

Sodium and Hypertension. Salt-free or low salt diets often are prescribed for hypertensive patients (57). However, sodium chloride increases the blood pressure in some individuals but not in others. Conversely, restriction of dietary NaCl lowers the blood pressure of some hypertensives, but not of others. Genetic factors and other nutrients, eg, Ca " and K", may be involved. The optimal intakes of Na" and K" remain to be estabUshed... [Pg.380]

Dietary sodium restriction Reduce dietary sodium intake to no more than 100 mmol per day (2.4 g sodium or 6 g sodium chloride) 2-8 mm Hg... [Pg.16]

Nonpharmacologic treatment involves dietary modifications such as sodium and fluid restriction, risk factor reduction including smoking cessation, timely immunizations, and supervised regular physical activity. [Pg.33]

All patients with ascites require counseling on dietary sodium restriction. Salt intake should be limited to less than 800 mg sodium (2 g sodium chloride) per day. More stringent restriction may cause faster mobilization of ascitic fluid, but adherence to such strict limits is very difficult. Patients usually respond well to sodium restriction accompanied by diuretic therapy.14,22,31,32 The goal of therapy is to achieve urinary sodium excretion of at least 78 mEq (78 mmol) per day.22 While a 24-hour urine collection provides this information, a spot urine sodium/ potassium ratio greater than 1.0 provides the same information and is much less cumbersome to perform. [Pg.330]

Measure spot urine sodium/potassium ratio to assess adherence to dietary sodium restrictions. [Pg.335]

Provide education regarding dietary sodium restrictions at each visit consider a referral to a dietician if appropriate. [Pg.335]

Treatment for nonketotic hyperglycinemia is less effective than that available for other aminoacidurias. There is no specific therapy. Exchange transfusion and dialysis usually do not alter the progressive neurological deterioration. Sodium benzoate has been administered in the hope that glycine would react with it to form hippuric acid, but this approach is not helpful. It may be that a combination of benzoate and carnitine therapy is more effective [28]. Similarly, the restriction of dietary protein... [Pg.674]

Nonpharmacologic interventions include cardiac rehabilitation and restriction of fluid intake (maximum 2 L/day from all sources) and dietary sodium (approximately 2 to 3 g of sodium per day). [Pg.97]

All patients with prehypertension and hypertension should be prescribed lifestyle modifications, including (1) weight reduction if overweight, (2) adoption ofthe Dietary Approaches to Stop Hypertension eating plan, (3) dietary sodium restriction ideally to 1.5 g/day (3.8 g/day sodium chloride), (4) regular aerobic physical activity, (5) moderate alcohol consumption (two or fewer drinks per day), and (6) smoking cessation. [Pg.126]

Treatment of asymptomatic hypervolemic hypotonic hyponatremia involves correction of the underlying cause and restriction of water intake to less than 1,000 to 1,200 mL/day. Dietary intake of sodium chloride should be restricted to 1,000 to 2,000 mg/day. [Pg.895]

Patients with nephrogenic DI should decrease their ECF volume with a thiazide diuretic and dietary sodium restriction (2,000 mg/day), which often decreases urine volume by as much as 50%. Other treatment options include drugs with antidiuretic properties (Table 78-2). [Pg.897]

Dietary salt restriction was one of the first successful therapeutic maneuvers for the reduction of blood pressure. During the past two decades, a variety of pharmacologic agents have been developed which promote diuresis by interfering with the tubular reabsorption of sodium. Although diuretic agents differ significantly in chemical structure and in their mechanism of action on the renal tubule, they all have in common the ability to decrease blood pressure. [Pg.82]

Dietary sodium restriction has been known for many years to decrease blood pressure in hypertensive patients. With the advent of diuretics, sodium restriction was thought to be less important. However, there is now general agreement that dietary control of blood pressure is a relatively nontoxic therapeutic measure and may even be preventive. Even modest dietary sodium restriction lowers blood pressure (though to varying extents) in many hypertensive persons. [Pg.226]

Sodium removal—by dietary salt restriction and a diuretic—is the mainstay in management of symptomatic heart failure, especially if edema... [Pg.311]

DOC, which also serves as a precursor of aldosterone (Figure 39-1), is normally secreted in amounts of about 200 mcg/d. Its half-life when injected into the human circulation is about 70 minutes. Preliminary estimates of its concentration in plasma are approximately 0.03 mcg/dL. The control of its secretion differs from that of aldosterone in that the secretion of DOC is primarily under the control of ACTH. Although the response to ACTH is enhanced by dietary sodium restriction, a low-salt diet does not increase DOC secretion. The secretion of DOC may be markedly increased in abnormal conditions such as adrenocortical carcinoma and congenital adrenal hyperplasia with reduced P450cll or P450cl7 activity. [Pg.887]


See other pages where Sodium dietary restrictions is mentioned: [Pg.178]    [Pg.116]    [Pg.116]    [Pg.227]    [Pg.232]    [Pg.528]    [Pg.16]    [Pg.16]    [Pg.42]    [Pg.43]    [Pg.1448]    [Pg.1562]    [Pg.1448]    [Pg.1608]    [Pg.244]    [Pg.226]    [Pg.241]    [Pg.341]    [Pg.377]   
See also in sourсe #XX -- [ Pg.31 , Pg.124 ]




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