Prostate DU145 cancer cells

Scarlatti F, Sala G, Ricci C, Maioli C, Milani F, Minella M, Botturi M, Ghidoni R. 2007. Resveratrol sensitization of DU145 prostate cancer cells to ionizing radiation is associated to ceramide increase. Cancer Lett 253 124-130. 

Kampa, M., Bakogeorgou, E., Hatzoglou, A., Damianaki, A., Martin, P.-M., and Castanas, E. 1997. Opioid alkaloids and casomorphin peptides decrease the proliferation of prostatic cancer cell lines (LNCaP, PC3 and DU145) through a partial interaction with opioid receptors. Fur. J. Pharmacol 335, 255-265. 

Peng D, Fan Z, Lu Y, DeBlasio T, Scher H, Mendelsohn J. 1996. Anti-epidermal growth factor receptor monoclonal antibody 225 up-regulates p27KIPl and induces G1 arrest in prostatic cancer cell line DU145. Cancer Res. 56 3666-69... 

Preparation of 1. Prostate cancer cell lines LNCaP, PC3, and DU145 were cul-cellsforPLA tured and grown in T-25 flasks (VWR, West Chester, PA) in... 

We have previously described that 3,4-dihydroxy-phenylacetic acid (PAA) directly decreased NOS activity by 40%, while caffeic acid did not. Furthermore, PAA increased iNOS mRNA while it concurrently reduced eNOS mRNA expression (Kampa et al. 2004). We have also reported that catechin, epicatechin, quercetin, and resveratrol decrease NO secretion by prostate cancer cell lines (LNCaP, PC3, and DU145) and inhibit NO production by T47D breast cancer cells. Although a concurrent decrease in total NOS (eNOS and iNOS) activity was observed after treatment for 24 h or longer, these agents induced a transient early increase in NOS activity. This bimodal effect indicated a possible dual action of polyphe-... 

A. Panov, and Z. Orynb eva, Bioenergetic and antiapoptotic properties of mitochondria fix)m cultured human prostate cancer cell line s PC-3, DU145 and LNCaP. PLoS One, 8 (2013) e72078. 

McCarthy et al. (2007) Gene transfer hOCiNOS plasmid in liposome Human prostate cancer (PC-3 and DU145) Clonogenic cell survival reduced by 80-90% ... 

MEL-28 (melanoma) it was patented in 2001. Jorunna-mydn C is slightly less active than jorumydn against the cancer cell lines HCT116 (colon), QG56 (lung) and DU145 (prostate) jorunnamycins A and B are inactive (Charupant et al, 2007). 

Zi, X.L. et al., A flavonoid antioxidant, silymarin, inhibits activation of erbBl signaling and induces cyclin-dependent kinase inhibitors, G1 arrest and anticarcinogenic effects in human prostate carcinoma DU145 cells, Cancer Res., 58, 1920, 1998. 

It has been shown that EGCG-induced apoptosis, cell-growth inhibition, and cell-cycle dysregulation in human prostate cancer DU145 and LNCaP cells without producing similar effects in normal cells [Ahmad et al., 1997 ... 

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