Projected fiber systems

This is possible because the projection lens system, which for clarity was not shown in Figure 4.7, is normally included behind the objective lens and below the source image plane. This lens system allows the projection of both the diffraction pattern and the specimen image on the observation screen. In Figure 4.8, [50] the electron diffraction pattern of a Fe thin film is shown. In Figure 4.9, the transmission electron micrograph of the mordenite included in the sample CMT-C (see Table 4.1), where fiber-like crystals of mordenite are seen, is shown [51],... 

In my first research project, I demonstrated that indeed argyrophil fibers are present between the cell membranes of plant cells and the extracellular cellulose fibers. I also demonstrated that this argyrophil fiber system could be artificially produced by applying the same histological techniques to... 

Collateral projections of mossy fiber systems to the cerebellar nuclei. 

Oscarsson O (1969) The sagittal organization of the cerebellar anterior lobe as revealed by the projection patterns of the climbing fiber system. In Llinas R (Ed.), Neurobiology of Cerebellar Evolution and Development. AMA/ERF, Chicago, 525-537. 

Hirschfeld, T. Deaton, T. Milanovich, F. Klainer, S. Fitzsimmons, C. Project Summary—Feasibility of Using Fiber Optics for Monitoring Groundwater Contaminants U.S. Environmental Protection Agency, Environmental Monitoring Systems Lab. Las Vegas, NV, January 1984. 

Consistent with these observations, there is some evidence that caffeine may act, in part, on dopaminergic fibers that project into the medial forebrain bundle. Other psychostimulants also appear to act on this system, which may be at least one of the neural mechanisms involved in the development of dependence.271 Further work has suggested a parallel between caffeine and another highly addictive drug, alcohol. Many of the same factors that enter into the development of alcoholism may also influence the development of dependence on caffeine.272... 

If the structural entities are lamellae, Eq. (8.80) describes an ensemble of perfectly oriented but uncorrelated layers. Inversion of the Lorentz correction yields the scattering curve of the isotropic material I (5) = I (s) / (2ns2). On the other hand, a scattering pattern of highly oriented lamellae or cylinders is readily converted into the ID scattering intensity /, (53) by ID projection onto the fiber direction (p. 136, Eq. (8.56)). The model for the ID intensity, Eq. (8.80), has three parameters Ap, dc, and <7C. For the nonlinear regression it is important to transform to a parameter set with little parameter-parameter correlation Ap, dc, and oc/dc. When applied to raw scattering data, additionally the deviation of the real from the ideal two-phase system must be considered in an extended model function (cf. p. 124). 

The nervous system contains an unusually diverse set of intermediate filaments (Table 8-2) with distinctive cellular distributions and developmental expression [21, 22]. Despite their molecular heterogeneity, all intermediate filaments appear as solid, rope-like fibers 8-12 nm in diameter. Neuronal intermediate filaments (NFs) can be hundreds of micrometers long and have characteristic sidearm projections, while filaments in glia or other nonneuronal cells are shorter and lack sidearms (Fig. 8-2). The existence of NFs was established long before much was known about their biochemistry or properties. As stable cytoskeletal structures, NFs were noted in early electron micrographs, and many traditional histological procedures that visualize neurons are based on a specific interaction of metal stains with NFs. 

Seyler coal classification system, 6 709-710, 719 S-glass fibers, 26 758 SG sol-gel abrasives, 7 7 Shadow projection X-ray microscopy, 76 504... 

The EOPs have been directly implicated in the pathophysiology of TS syndrome. Haber and co-workers (1986) reported decreased levels of dynorphin A(l-17) immunoreactivity in striatal fibers projecting to the GP in postmortem material from a small number of Tour-ette s patients. This observation, coupled with the neu-roanatomic distribution of dynorphin, its broad range of motor and behavioral effects, and its modulatory interactions with striatal dopaminergic systems, suggest that dynorphin might have a key role in the patho-biology of TS. However, subsequent studies have failed to confirm these initial observations (van Wattum et al., 1999). 

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