Hemorrhagic pulmonary edema

Because clove cigarettes also contain tobacco, they carry all of the hazards of regular tobacco smoking (Council on Scientific Affairs 1988). Several cases of serious medical illness have been associated with clove cigarettes in the United States (Guidotti et al. 1989). These include cases of hemorrhagic pulmonary edema, pneumonia, bronchitis, and hemoptysis. Because eugenol anesthetizes the respiratory tract, it inhibits the normal gag reflex and has led to aspiration pneumonia in at least one case. Accidental overdoses in children have led to CNS depression, urinary abnormalities, and anion-gap acidosis. These cases were treated successfully with supportive measures (Lane et al. 1991). 

Of three chemical workers who were observed after accidental exposures to perchloromethyl mercaptan, two survived episodes of pulmonary edema, and the third died after 36 hours. The fatality resulted from a spill of the liquid on the clothing and floor with exposure to the vapor. At autopsy, there was necrotizing tracheitis, massive hemorrhagic pulmonary edema, marked toxic nephrosis, and vacuolization of centrilobular hepatic cells. 

Respiratory Effects. Hemorrhagic pulmonary edema was found at necropsy in a l-year-old baby who died after having 20 ml of a cresol- containing product spilled on his head (Green 1975). 

Hydrogen fluoride Respiratory irritation, hemorrhagic pulmonary edema... 

A 28-year-old died after an overdose of diphenhydramine. He developed hyperpyrexia and tachycardia and died from a cardiac arrest (13). Hemorrhagic pulmonary edema and renal shock were the most prominent findings. At the time of death, the plasma concentration of diphenhydramine was 5 pg/ml and there were particularly high concentrations in the lungs (55 mg/kg) and kidneys (50mg/kg). 

Acute, heavy exposure to SM causes loss of the columnar cells of the upper respiratory tract, peribronchial edema, hyperemia of the blood vessels, cellular infiltrations in the submucosa, and intense vacuolization and disorganization of the cytoplasmic and nuclear structures (Emad and Rezaian, 1997, 1999). Pulmonary hemorrhage, pulmonary edema, and respiratory failure similar to ARDS may also occur. These cytotoxic effects are associated with acute thermal injury sustained by the airway mucosa and lead to scarring and development of stenosis of the tracheobronchial tree as was observed in 9.64% of the SM-exposed patients. 

ACUTE HEALTH RISKS convulsions labored breathing vomiting apprehension auditory hallucinations tingling sensations of the nose and face facial numbness twitching pulsus altemans ectopic heartbeats tachycardia ventricular fibrillation respiratory failure hemorrhagic pulmonary edema degeneration of renal tubules unconsciousness may result in death. 

Hemorrhagic pulmonary edema Microcytic hypochromic anemia Radial nerve palsy... 

Hydrogen chloride hemorrhagic pulmonary edema Severe respiratory tract irritant. ... 

A. Severe hypertension, which may result in intracranial hemorrhage, pulmonary edema, or myocardial necrosis. 

Untoward effects of both E and NE (usually to a lesser degree) are anxiety, headache, cerebral hemorrhage (from vasopressor effects), cardiac arrhythmias, especially in presence of digitaUs and certain anesthetic agents, and pulmonary edema as a result of pulmonary hypertension. The minimum subcutaneous lethal dose of E is about 4 mg, but recoveries have occurred after accidental overdosage with 16 mg subcutaneously and 30 mg intravenously, followed by immediate supportive treatment. 

NOC1 is intensely irritating to the eyes, skin and mucous membranes. Inhalation can cause pulmonary edema and hemorrhage (Ref 3) Refs 1) J.R. Morton H.W. Wilcox, Inorg-Synth 4 (1953), 48 2) Anon, C EN 35 (43),... 

Serious adverse effects of epinephrine potentially occur when it is given in an excessive dose, or too rapidly, for example, as an intravenous bolus or a rapid intravenous infusion. These include ventricular dysrhythmias, angina, myocardial infarction, pulmonary edema, sudden sharp increase in blood pressure, and cerebral hemorrhage. The risk of epinephrine adverse effects is also potentially increased in patients with hypertension or ischemic heart disease, and in those using (3-blockers (due to unopposed epinephrine action on vascular Ui-adrenergic receptors), monoamine oxidase inhibitors, tricyclic antidepressants, or cocaine. Even in these patients, there is no absolute contraindication for the use of epinephrine in the treatment of anaphylaxis [1,5,6]. 

Postmortem lesions include excessive fluid in the sac surrounding the heart (hydropericardium) in the chest cavity (hydrothorax) pulmonary edema hemorrhage in the abomasum and intestine hemorrhagic gastroenteritis enlarged liver, spleen, and lymph nodes and edema and hemorrhage of the brain. 

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