Depression emotional

Psychiatric changes such as depression, emotional lability, psychosis, euphoria, anxiety, and decreased cognition (85%)... 

Dizziness, headache, fatigue, fever, insomnia, irritability, depression, emotional lability, impaired concentration, alopecia, rash, pruritus, nausea, anorexia, dyspepsia, vomiting, decreased hemoglobin, hemolysis, arthralgia, musculoskeletal pain, dyspnea, sinusitis, flu-like symptoms Occasional (1 %-10%)... 

The affective and other behavioral disturbances category of adverse drug reactions included Depression, Psychotic Depression, Emotional lability, Euphoria, Hostility, Personality disorder, and Decreased libido. ... 

Topical p-blockers have been associated with adverse central nervous system (CNS) effects, including depression, emotional lability, and sexual dysfunction. Complaints of lethargy, lightheadedness, weakness, fetigue, mental depression, dissociative behavior, and memory loss are most common. The onset of symptoms varies from a few days to months after initiation of therapy. In most cases these symptoms are mild and transient. In certain patients, however, timolol must be discontinued. 

Barriers to compliance must be identified during the history. Emotions, cognitive function, and physical ability can affect patient adherence to therapy. If a patient suffers from depression (emotional barrier), schizophrenia or dementia (cognitive barrier), or severe arthritis of the hands (physical barrier), compliance can diminish. Special attention should be given to these three areas, and barriers should be indicated on the history record. This process directs the implementation of specific aids to improve compliance. 

Atypical antipsychotics such as aripiprazole, olanzapine, que-tiapine, risperidone, and ziprasidone are effective as monotherapy or adjunctive therapy with lithium and valproate in the treatment of acute mania. Some antipsychotics have the potential to cause adverse effects such as extrapyramidal reactions, sedation, depression, emotional blunting, sexual dysfunction, weight gain, and orthostatic hypotension. Prophylactic use of antipsychotics may be needed for some patients with recurrent mania or mixed states, but the risks versus benefits must be weighed because of long-term adverse effects (e.g., obesity, type 2 diabetes, hyperlipidemia, hyperprolactinemia, cardiac disease, and tardive dyskinesia). ... 

Barbiturates are dangerous for those who already have low blood pressure, heart defects, or suffer from anxiety or depression. Emotionally unstable individuals will often attempt a near-overdose to gain sympathy and attention. Needle freaks who shoot downers are taking a much greater risk than they would be with most other drugs, and risk increased amounts of abscesses and other complications. 

Fear is a strong and depressed emotional state which individuals or groups felt deeply in danger of real or imagined. It often manifests as nervous, fear, inability to concentrate, mind going blank, and inability to correctly judge or control behavior. Fear is an innate emotional response of all animal including human. Fear is emotional reaction to an expected to be hurt or unpleasant when face stressors, which usually cause avoidance behavior. 

Headache Depression Emotional distress Irritability Memory loss... 

Acute benzene poisoning results in CNS depression and is characterized by an initial euphoria followed by staggered gait, stupor, coma, and convulsions. Exposure to approximately 4000 ppm benzene results in complete loss of consciousness. Insomnia, agitation, headache, nausea, and drowsiness may persist for weeks after exposure (126). Continued inhalation of benzene to the point of euphoria has caused irreversible encephalopathy with tremulousness, emotional lability, and diffuse cerebral atrophy (125). In deaths arising from acute exposure, respiratory tract infection, hypo- and hyperplasia of sternal bone marrow, congested kidneys, and cerebral edema have been found at autopsy. 

Air-poUutant effects on neural and sensory functions in humans vary widely. Odorous pollutants cause only minor annoyance yet, if persistent, they can lead to irritation, emotional upset, anorexia, and mental depression. Carbon monoxide can cause death secondary to the depression of the respiratory centers of the central nervous system. Short of death, repeated and prolonged exposure to carbon monoxide can alter sensory protection, temporal perception, and higher mental functions. Lipid-soluble aerosols can enter the body and be absorbed in the lipids of the central nervous system. Once there, their effects may persist long after the initial contact has been removed. Examples of agents of long-term chronic effects are organic phosphate pesticides and aerosols carrying the metals lead, mercury, and cadmium. 

Lethargy, dizziness, insomnia, anorexia, nausea, sexual dysfunction, headache, emotional lability, depression, sweating, acne, breast atrophy, peripheral edema, lower urinary trad symptoms, hot flashes, pain, edema, upper respiratory tract infedion, rash... 

Emotional instability - displaying depressive, aggressive or simply moody behaviour is a taboo for mentors. They must learn how to manage their emotions so as to be capable of providing mentees with the calmness, patience and reflectivity that is required of them. 

F43.2 Adjustment disorders. 20 Brief depressive reaction. 21 Prolonged depressive reaction. 22 Mixed anxiety and depressive reaction. 23 With predominant disturbance of other emotions. 24 With predominant disturbance of conduct. 25 With mixed disturbance of emotions and conduct. 28 With other specified predominant symptoms... 

This behavioural syndrome, rather emotively called learned helplessness", is widely believed to share many features of depression, not least because both culminate in psychomotor retardation and both are linked with experience of uncontrollable, unpredictable stress. Whether or not learned helplessness really is an analogue of depression remains controversial (Maier 1993). Nevertheless, escape deficits in rats are prevented by pretreatment with antidepressants from different generic groups. Other psychotropic agents, such as CNS stimulants and neuroleptics, are generally ineffective. 

Lorazepam (Ativan) 0.5-2 mg prior to chemotherapy Most common Sedation, amnesia Rare Respiratory depression, ataxia, blurred vision, hallucinations, paradoxical reactions (weeping, emotional reactions)... 

It is widely accepted that it takes approximately 2 to 4 weeks of treatment before improvement is seen in emotional symptoms of depression, such as sadness and anhedonia. Furthermore, it may take as long as 6 to 8 weeks of treatment to see the full effects of antidepressant therapy. 

Unfortunately, antidepressants do not produce a clinical response immediately. Improvement in physical symptoms, such as sleep, appetite, and energy, can occur within the first week or so of treatment. Although a recent meta-analysis suggests earlier effects of antidepressant treatment,36 it is widely accepted that it takes approximately 2 to 4 weeks of treatment before improvement is seen in emotional symptoms of depression, such as sadness and anhedonia. Furthermore, it may take as long as 6 to 8 weeks of treatment to see the full effects of antidepressant therapy.7 22 23... 

Reduced lean body mass Reduced muscle strength Reduced exercise performance Thin, dry skin cool peripheries poor venous access Depressed affect, labile emotions Impaired cardiac function... 

In 1976, Aaron Beck, a psychiatrist at the University of Pennsylvania, proposed a cognitive theory of emotions and emotional disorders - a theory that was to become the foundation for cognitive behavioural therapy for depression. According to Beck, fear is produced by the anticipation of harm, joy by the expectancy of positive events, and sadness by the sense that something important has for ever been lost. As a consequence, overcoming fear and depression requires changing the beliefs that have produced them. 

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