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Behavior imipramine

Ciraulo DA, Jaffe JH Tricyclic antidepressants in the treatment of depression associated with alcoholism. Clin Psychopharmacol 1 146—150, 1981 Ciraulo DA, Nace E Benzodiazepine treatment of anxiety or insomnia in substance abuse patients. Am J Addict 9 276—284, 2000 Ciraulo DA, Barnhill JG, Jaffe JH, et al Intravenous pharmacokinetics of 2-hydroxy-imipramine in alcoholics and normal controls. J StudAlcohol 51 366-372, 1990 Ciraulo DA, Knapp CM, LoCastro J, et al A benzodiazepine mood effect scale reliability and validity determined for alcohol-dependent subjects and adults with a parental history of alcoholism. Am J Drug Alcohol Abuse 27 339—347, 2001 Collins MA Tetrahydropapaveroline in Parkinson s disease and alcoholism a look back in honor of Merton Sandler. Neurotoxicology 25 117-120, 2004 COMBINE Study Research Group Testing combined pharmacotherapies and behavioral interventions in alcohol dependence rationale and methods. Alcohol Clin Exp Res 27 1107-1122, 2003a... [Pg.43]

Antidepressant Some animal models show antidepressant effects of lobelia extract (Subarnas et al. 1992). Similar to imipramine and mianserin, beta-amyrin palmitate shows antidepressant-like effects in the forced-swimming test (Subarnas et al. 1993a). Whereas mianserin and beta-amyrin palmitate reduce locomotor activity induced by methamphetamine, imipramine increases it. It potentiates sodium pentobarbital-induced sleep more potently than imipramine, but less than mianserin. Collectively, the effects of beta-amyrin palmitate in behavioral and physiological assays suggests it may work in a manner more similar to mianserin than imipramine. However, the mechanism of antidepressant-like effects of lobelia is uncertain. It may be through the beta-amyrin palmitate s ability to release norepinephrine (Subarnas et al. 1993b). An antidepressant effect of lobelia has not been established in humans. [Pg.127]

Imipramine (Tofranil) [Antidepressant/TCA] WARNING Close observation for suicidal thinking or unusual changes in behavior Uses Depres-sion, enuresis, panic attack, chronic pain Action TCA t CNS synaptic serotonin or norepinephrine Dose Adults. Hospitalized Initial 100 mg/24 h PO in doses T over several wk 300 mg/d max Output Maint 50-150 mg PO hs, 300 mg/24 h max Peds. Antidepressant 1.5-5 mg/kg/24 h daUy-qid Enuresis >6 y 10-25 mg PO qhs T by 10-25 mg at 1-2-wk int vals (max 50 mg for 6-12 y, 75 mg for >12 y) Rx for 2-3 mo, then tap Caution [D, /-] Contra Use w/ MAOIs, NAG, acute recovery from MI, PRG, CHF, angina, CVD, arrhythmias Disp Tabs, caps SE CV Sxs, dizziness, xerostomia, discolored urine Interactions t Effects W/ amiodarone, anticholinergics, BBs, cimetidine, diltiazem, Li, OCPs, quinidine, phenothiazines, ritonavir, verapamil, EtOH, evening primrose oil t effects OF CNS depressants, hypoglycemics, warfarin T risk of serotonin synd W/MAOIs 4-... [Pg.190]

Flint J, Corley R, De Fries JC, Fulker DW, Gray JA, MUler S, Collins AC (1995) A simple genetic basis for a complex psychological trait in laboratory mice. Science 269 1432-1435 Fontana DJ, Commissaris RL (1988) Effects of acute and chronic imipramine administration on conflict behavior in the rat a potential animal model for the study of panic disorder Psychopharmacology (Berl) 95 147-150... [Pg.63]

Barlow DH, Gorman JM, Shear MK, Woods SW (2000) Cognitive-behavioral therapy, imipramine, or their combination for panic disorder a randomized controlled trial. JAMA 283 2529-2536... [Pg.495]

Bernstein, G.A., Borchardt, C.M., Perwien, A.R., Crosby, R.D., Kushner, M.G., Thuras, P.D., and Last, C.G. (2000) Imipramine plus cognitive-behavioral therapy in the treatment of school refusal. J Am Acad Child Adolesc Psychiatry 39 276—283... [Pg.293]

Jason, K.M., Cooper, T.B., and Friedman, E. (1981) Prenatal exposure to imipramine alters early behavioral development and beta-adrenergic receptors in rats. / Pharmacol Exp Ther 217 461 66. [Pg.651]

Tricyclic Antidepressants. A study of imipramine treatment led to the cessation of CD behaviors in 85% of 13 boys with CD and MDD (Fuig-Antich, 1982). [Pg.678]

Kahn NH, Shelton SJ Defensive behaviors in infant rhesus monkeys environmental cues and neurochemical regulation. Science 243 1718-1721, 1989 Kahnowsky LB, Kennedy F Observations in electric shock therapy apphed to problems of epilepsy. J Nerv Ment Dis 98 56-67, 1943 Kampen D, Sherwin B Estrogen use and verbal memory in healthy postmenopausal women. Obstet Gynecol 83 979-983, 1994 Kane JM, Quitkin FM, Rifkin A, et al Lithium carbonate and imipramine in the prophylaxis of unipolar and bipolar 11 illness a prospective placebo-controlled comparison. Arch Gen Psychiatry 39 1065-1069, 1982 Kaneno S, Komatsu H, Fukamauchi F, et al Biochemical basis of antidepressant-effect of low dose of sulpiride. Japanese Journal of Psychiatry and Neurology 45 131-132, 1991... [Pg.669]

Kim SW, Dysken MW, Pandey GN, et al Platelet 3H-imipramine binding sites in OC behavior. Biol Psychiatry 30 467-474, 1991 Kindler S, Shapira B, Hadjez J, et al Factors influencing response to bilateral electroconvulsive therapy in major depression. Convulsive Therapy 7 245-254, 1991... [Pg.673]

Martin P, Laurent S, Massol J, et al Effects of dihydropyridine drugs on reversal by imipramine of helpless behavior in rats. Eur J Pharmacol 162 185-188, 1989 Masana MI, Bitran JA, Hsiao JK, et al Lithium effects on noradrenergic linked adenylate cyclase activity in intact rat brain an in vivo microdialysis study. Brain Res 538 333-336, 1991... [Pg.691]

Prange AJ Jr, Wilson 1C, Breese GR, et al Hormonal alteration of imipramine response a review, in Hormones Behavior and Psychopathology. Edited by Sachar EJ. New York, Raven, 1976, p 41... [Pg.724]

The behavioral syndrome induced in mice and rats by reserpine sedation, catalepsy and ptosis can be reversed by imipramine. [Pg.118]

Most efficacy trials with reboxetine have so far only been published in review articles ( 178). Most of these articles did not have peer review and do not contain the full details concerning methodology or results. This fact limits the ability to accurately determine its relative efficacy and tolerability. In short-term (4 to 8 weeks), placebo-controlled clinical trials, reboxetine produced a response (defined as at least a 50% reduction in severity scores) in 56% to 74% of patients. These results were statistically superior to placebo in most studies. Reboxetine was also found to be as effective as imipramine and desipramine in four double-blind, randomized, active-controlled (but not placebo-controlled) studies involving more than 800 outpatients or inpatients with major depression. Reboxetine produced equivalent antidepressant response rates compared with fluoxetine in two clinical trials, one of which was also placebo-controlled. However, reboxetine was reported to have improved social motivation and behavior more than fluoxetine as assessed by the newly developed Social Adaptation Self-Evaluation Scale. In all of the studies, reboxetine had a similar time (i.e., 2 to 3 weeks) to onset of the antidepressant efficacy as do other antidepressants. [Pg.124]

Although several studies have found that the combination of imipramine and behavioral therapy may be superior to either treatment alone, no studies have specifically examined the effects of combining the behavioral techniques with BZDs (132, 133,134, 135,136, 137 and 138). In a review of the literature, however, Wardle found that diazepam was superior to placebo in three of four studies in which patients also received exposure therapy ( 139). Combined treatment with BZDs and CBT may be advantageous for some patients, but it must be carefully designed to avoid potential problems ( 140). [Pg.260]

In preclinical studies, a number of TCAs (imipramine, amitriptyline, nortriptyline, desipramine) were shown to inhibit pain behavior in the formalin test after systemic as well as after i.t. administration, and this effect did not seem to be related to an antiinflammatory effect of these drugs (Sawynok and Reid, 2001). The effects of TCAs in preclinical acute pain models (involving acute thermal or mechanical stimuli) have been reviewed by Eschalier et al. (1999). TCAs were also active in models of chronic inflammation (Butler et al., 1985) and in models of neuropathic pain involving nerve injury (e.g. Ardid and Guilbaud, 1992 Abdi et al., 1998). [Pg.269]

Iwashita, T., Shimizu, T. Imipramine inhibits intrathecal substance P induced behavior and blocks spinal cord substance P receptors in mice, Brain Res. 1992, 581, 59-66. [Pg.282]

I.R. Coyle, Changes in developing behavior following prenatal administration of imipramine, Pharmacol. Biochem. Behav., 3(5) (1975) 799-807. [Pg.311]

Precautions The tricyclic antidepressants should be used with caution in manic-depressive patients, since they may unmask manic behavior. The tricyclic antidepressants have a narrow therapeutic index for example, 5 to 6 times the maximal daily dose of imipramine can be lethal. Depressed patients who are suicidal should be given only limited quantities of these drugs and should be monitored closely. Drug interactions with the tricyclic antidepressants are shown in Figure 12.5. [Pg.132]

Nishida KJ, Dougherty GG, Ellinwood EH, Rockwell WJK (1990) Effects of chronic chlorimipramine and imipramine administration on food hoarding behavior in male rats. Res Commun Psychol Psychiatry Behav 15 115-128 Rosofsky M, Geary N (1989) Phenylpropanolamine and amphetamine disrupt postprandial satiety in rats. Pharmacol Biochem Behav 34 797-803... [Pg.194]

Aggressiveness during treatment in patients taking imipramine and amitriptyline in relatively low doses has been described (68). Violent behavior has also been attributed to amitriptyline (SEDA-17,18). [Pg.11]


See other pages where Behavior imipramine is mentioned: [Pg.99]    [Pg.817]    [Pg.197]    [Pg.93]    [Pg.890]    [Pg.353]    [Pg.151]    [Pg.231]    [Pg.432]    [Pg.655]    [Pg.45]    [Pg.225]    [Pg.408]    [Pg.430]    [Pg.746]    [Pg.299]    [Pg.303]    [Pg.135]    [Pg.592]    [Pg.63]    [Pg.181]    [Pg.110]   
See also in sourсe #XX -- [ Pg.294 ]




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