Trauma stress

Clark, R. D., Canive, J. M., Calais, L. A. et al. Divalproex in posttraumatic stress disorder an open-label clinical trial. /. Trauma Stress 12 395-401,1999. 

Glover D, Poland R (2002) Urinary cortisol and catecholamines in mothers of child cancer survivors with and without PTSD. Psychoneiu oendocrinology 27 805-819 Goeinjian AK, Pynoos RS, Steinberg Am, Endres D, Abraham K, Geffner ME, Fairbanks LA (2003) Hypothalamic-pituitary-adrenal activity amongArmenian adolescents with PTSD symptoms. J Trauma Stress 16 319-323... 

Goldberg et al. Co-twin control study of relationships among combat expo- CS335 sure, combat-related PTSD, and other mental disorders. J Trauma Stress 2003 16(5) 433-438. 

Zaidi, L.Y. and Foy, D.W. (1994) Childhood abuse experiences and combat-related PTSD. Trauma Stress 7 33 2. 

Forster PL, Schoenfeld FB, Marmar CR, et al. Lithium for irritability in post-traumatic stress disorder. J Trauma Stress 1995 8 143-149. 

Ford N. The use of anticonvulsants in posttraumatic stress disorder case study and overview. J Trauma Stress 1996 9 857-863. 

Harvey AG, Bryant RA. Dissociative symptoms in acute stress disorder. J Trauma Stress 1999 12 673-680. 

Zayfert C, DeViva JC (2004) Residual insomnia following cognitive behavioral therapy for PTSD. J Trauma Stress 17 69-73... 

Both types of latent HSV are thought to be reactivated by many factors, including UV exposure, trauma, stress, extreme temperatures, immunosuppression, and menstruation. When activated, the virus travels along the sensory nerve to peripheral tissue to cause recurrent HSV infection. An estimated 300,000 cases of primary and recurrent HSV infections develop each year. 

Check with provider when insulin demands are altered (e.g., fever, infection, trauma, stress, heavy physical activity). 

Penk WE, Allen IM Clinical assessment of post-traumatic stress disorder (PTSD) among American minorities who served in Vietnam. J Trauma Stress 4 41-66,1991... 

Endorphin 31 7 Suppresses avoidance/alarm behavior (/r,) Suppresses behavioral reactiveness to pain, trauma, stresses ( u.,) Stimulatesappetite pi) ... 

North, C.S., L. Tivis, J.C. McMillen, et al. 2002a. Coping, functioning, and adjustment of rescue workers after the Oklahoma City homhing. J Trauma Stress 15(3) 171-5. 

Lees-Haley PR Malingering traumatic mental disorder on the Beck Depression Inventory cancerphobia and toxic exposure. Psychol Rep 65 623-626,1989c Lees-Haley PR Malingering mental disorder on the Impact of Event Scale (IES) toxic exposure and cancerphobia. J Trauma Stress 3 315-321, 1990 Lees-Haley PR, Brown RS Biases in perception and reporting following a perceived toxic exposure. Percept Mot Skills 75 531-544, 1992 LeQuesne PM, Axford AT, McKerrow CB, et al Neurological complications after a single severe exposure to toluene di-isocyanate. British Journal of Industrial Medicine 33 72-78, 1976... 

Baker DB, Greenland S, Mendlein J, et al A health study of two communities near the Stringfellow waste disposal site. Arch Environ Health 43 325-334, 1988 Bowler RM, Mergler D, Huel G, et al Psychological, psychosocial, and psychophys-iological sequelae in a community affected by a railroad chemical disaster. I Trauma Stress 7 601-624, 1994... 

Havenaar JM, van den Brink W, van den Bout J, et al Mental health problems in the Gomel region (Belarus) an analysis of risk factors in an area affected by the Chernobyl disaster. Psychol Med 26 845-855,1996 Kolominsky Y, Igumnov S, Drozdovitch V The psychological development of children from Belarus exposed in the prenatal period to radiation from the Chernobyl atomic power plant. J Child Psychol Psychiatry 40 299-305, 1999 Koscheyev VS, Martens VK, Kosenkov AA, et al Psychological status of Chernobyl nuclear power plant operators after the nuclear disaster. J Trauma Stress 6 561-568, 1993... 

Psoriasis is a chronic, inflammatory and hyperprolifera-tive disease of the skin, scalp, nails, and joints, affecting 1 to 2% of the U.S. population. It is found worldwide its frequency varies from 0 to 3% among different ethnic groups. Most of its variable clinical presentations eventuate into erythematous, scaly plaques with or without nail disease and arthritis. Susceptibility to psoriasis is umnistakably heritable, but the phenotype is controlled by multiple genes as well as enviromnental factors. Trauma, stress, and infections are important determinants of disease onset and severity. At the cellular level, psoriasis is characterized by markedly increased epidermal proliferation and incomplete differentiation elongation, dilatation, and leakiness of the superficial plexus of dermal capillaries and a mixed inflammatory and immune cell infiltrate of the epidermis and papillary dermis. A multitude of plausible pathomechanisms... 

North CS, Pollio DE, Pfeflferbaum B a aL Capitol hill staff workers experiences of biotetrorism Qualitative finding fiom focus groups. J Trauma Stress 2005 18(l) 79-88. 

The lirst rib is probably the rib most commonly involved in somatic dysfunction of all the ribs. It is affected by trauma, stress, and posture as well as by dysfunction ofthe C7-T1 complex. The patient may report "shoulder" pain, stiff neck, upper back or neck pain, and an inability to turn the head while driving. The first rib can impinge the neurovascular bundle as it passes between it and the clavicle through the costoclavicular space. The anterior and middle scalene muscles, which raise the first rib, may likewise compress the brachial plexus when they are in spasm and result in thoracic outlet syndrome symptoms. The patient s symptoms are then described as pain, numbness, or paresthesias ofthe arm or hand on the involved side. The physician needs to be aware that this may cause confusion should the patient demonstrate a herniated cervical disc on magnetic resonance imaging [MRO. The symptoms may be caused by the rib dysfunction rather than the herniated disc, so evaluation ofthe rib for normal motion and treatment of any dysfunction should be performed in these cases. Osteopathic manipulation may save the patient unnecessary surgery. 

Gill, J., Vythilingam, M., Page, G.G., 2008. Low cortisol, high DHEA, and high levels of stimulated TNF-alpha, and IL-6 in women with PTSD. J. Trauma. Stress 21, 530-539. 

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