Nightmares weakness

Giddiness, tension, anxiety, jitteriness, restlessness, emotional lability, excessive dreaming, insomnia, nightmares, headaches, tremor, withdrawal and depression, bursts of slow waves of elevated voltage in EEC, especially on over-ventilation, drowsiness, difficult concentration, slowness on recall, confusion, slurred speech, ataxia, generalized weakness, coma, with absence of reflexes, Cheyne-Stokes respirations, convulsions, depression of respiratory and circulatory centers, with dyspnea, cyanosis, and fall in blood pressure. 

Adverse reactions include the following anorexia, apprehension, body/joint pain, chest pains, confusion, confusional states/memory impairment, congestion, constipation, coordination disorders, cramps/pain, depression, diarrhea, dreaming/nightmares, dry mouth, dysesthesia, euphoria, Gl pain, GU complaints, headache, heartburn, insomnia, irritability, lack of concentration, nausea, nervousness, palpitations, paresthesia, relaxed feeling, restlessness, tachycardia, taste alterations, tinnitus, tiredness, tremor, vomiting, weakness. 

Tremors of the hands and sleep disturbances in the form of vivid dreams, nightmares, and insomnia have been reported in association with the use of amiodarone. Ataxia, staggering, and impaired walking have been noted. Peripheral sensory and motor neuropathy or severe proximal muscle weakness develops infrequently. Both neuropathic and myopathic changes are observed on biopsy. Neurological symptoms resolve or improve within several weeks of dosage reduction. 

The most common early manifestations of digoxin toxicity are G1 disturbances (anorexia, nausea, vomiting) and neurologic abnormalities (fatigue, headache, depression, weakness, drowsiness, confusion, nightmares). 

The most common side effects are Raynaud s phenomenon with cold or even cyanotic distal extremities and digits, tiredness or weakness, bradycardia, and sexual impotence. Less common side effects are depression and dysphoria, bronchoconstriction, congestive heart failure, hallucinations, hypotension, vomiting or nausea, diarrhea, insomnia and nightmares, dizziness, and hypoglycemia. When due attention is paid to contraindications and the treatment is carefully monitored, the side effects of beta-blocker treatment are generally mild. 

Occupational workers repeatedly exposed to coumaphos have shown impaired memory and concentration, disorientation, severe depression, irritability, confusion, headache, speech difficulties, delayed reaction times, nightmares, sleepwalking, and drowsiness or insomnia. An influenza-like condition with headache, nausea, weakness, loss of appetite, and malaise also has been reported. Data on reproductive effect and genotoxicity are inadequate.37-39... 

Chnical features like skin lesions and neuropathy are crude and imprecise indicators of the severity of poisoning. The early clinical symptoms of arsenic toxicity are headache, dizziness, insomnia, weakness, nightmare, numbness in the extremities, anemia, palpitations, and fatigue. White striae in the fingernails are also a useful clue in the diagnosis of... 

Greater risk of future psychoses, personality disorders, and neuroses decrease in cognitive and intellectual performance mental retardation fatigue somnolence (postirradiation syndrome) Radiation necrosis decreased appetite, weakness, depression, nightmares, paranoia, psychosis, labile mood, personality changes, cognitive decline, dementia... 

Tetraethyl lead and tetramethyl lead are lipid-soluble compounds that are absorbed readily from the skin, GI tract, and lungs. The toxicity of tetraethyl lead is believed to be due to its metabolic conversion to triethyl lead and inorganic lead. The major symptoms of intoxication with tetraethyl lead are referable to the CNS insomnia, nightmares, anorexia, nausea and vomiting, diarrhea, headache, muscular weakness, and emotional instability. Subjective CNS symptoms such as... 

Methyidopa (Aldomet) As for clonidine. Also, synthesized to methyinorepinephrine which acts as a weak sympathomimetic "false neurotransmitter" which decreases sympathetic outflow from the CNS. As for clonidine. Used to treat hypertension in pregnant women. Dry mouth, sedation, slight orthostatic hypotension. Some patients experience impotence, psychic disturbances, nightmares, involuntary movements, or hepatotoxicity. 

Systemic effects which may occur from cholinergic eye-drops include nausea, diarrhoea, abdominal pain, muscle spasm and weakness, sweating, lacrimation, salivation, hypotension, bradycardia, bronchial constriction, respiratory failure and nightmares. Ocular effects, contributing to the decrease in lOP, are pupillary constriction, ciliary muscle contraction, dilatation of the conjunctival and iris blood vessels and increased aqeous outflow. The visual complications include accommodative spasm, myopia and reduced visual acuity changes in vision sensitivity, dark adaptation and visual fields may occur (20 ). 

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