Dreams, excessive

The adverse reactions most often associated with the administration of the COMT inhibitors include disorientation, confusion, light-headedness, dizziness, dyskinesias, hyperkinesias, nausea, vomiting, hallucinations, and fever. Other adverse reactions are orthostatic hypotension, sleep disorders, excessive dreaming, somnolence, and muscle cramps. A serious and possibly fatal adverse reaction that can occur with the administration of tolcapone is liver failure... 

Giddiness, tension, anxiety, jitteriness, restlessness, emotional lability, excessive dreaming, insomnia, nightmares, headaches, tremor, withdrawal and depression, bursts of slow waves of elevated voltage in EEC, especially on over-ventilation, drowsiness, difficult concentration, slowness on recall, confusion, slurred speech, ataxia, generalized weakness, coma, with absence of reflexes, Cheyne-Stokes respirations, convulsions, depression of respiratory and circulatory centers, with dyspnea, cyanosis, and fall in blood pressure. 

Among the effects on the central nervous system the following symptoms were pronounced excessive dreaming, insomnia, nightmares and headaches. 

One peculiar effect of GHB is that it seems to reduce the symptoms of narcolepsy, a relatively rare sleep disorder. People with narcolepsy are excessively sleepy all the time and often have a condition called cataplexy, in which the person can suddenly and unexpectedly lose all muscle tone and fall immediately into rapid eye movement (REM) sleep (the stage of sleep during which dreaming occurs). 

Nausea, insomnia, somnolence, anorexia, diarrhea, muscle cramps, orthostatic hypotension, excessive dreaming Occasional (11 %-4%)... 

Bai He is sweet, bland and slightly cold, and enters the Lung and Heart meridians. It can nourish the Yin and slightly tonify the Qi of the Lung and Heart. It is selected in the formula to reduce excess and deficiency of heat in the Lung when the pathogenic heat has injured the Yin and Qi in a febrile disease. Patients main complaints are of a warm, blocked sensation in the chest, restlessness and depression, a dry cough, insomnia and dream-disturbed sleep. 

When Sean was 19 and a freshman in collie, he started to have anxiety problems. He would feel scared a lot of the time for no particular reason. His mind was preoccupied by worrying about little or absurd things, he had difficulty concentrating, and he started to have bad dreams. He realized his anxiety was excessive but thought the problem would just go away on its own. When it didn t go away after a few years, he decided to see a psychiatrist to determine if anything could be done to alleviate the anxiety. 

The most obvious hypothesis is that in mania, as in dream psychosis with elation and grandiosity, it is possible to raise to very high levels the general activation of the brain and the specific activation of the positive emotion generator in the medial septum and other limbic regions. Because this effect can be artificially accomplished by taking amphetamines, it is reasonable to propose that excessive endogenous dopamine release (or increased receptor sensitivity) may be involved. 

Case (ii) A 25-year-old female patient with idiopathic spontaneous hypoglycemia. Symptoms recurrent hypoglycemic shock, blood sugar 18-20 mg/lOOml during the episode, hot patient, excessive craving for sweets, short necked, fanciful dreams. Earlier she had recurrent tonsilitis and skin disease treated by antibiotics. 

Sleep loss is therefore common in and contributes powerfully to the development of psychosis. In the case of schizophrenia (now thought to be a disorder of excessive dopamine release and/or heightened effectiveness of dopamine), we can hypothesize an indirect but positive interaction with the other modulators of the awake state, noradrenaline and serotonin, and a direct negative interaction with acetylcholine. Significantly for my dreaming-as-delirium thesis, no distinctive changes in sleep are seen in chronic schizophrenia. 

Maxmen and Ward (1995) provided an extensive list of tricyclic antidepressant withdrawal symptoms. One group of withdrawal symptoms includes a flulike syndrome without fever anorexia, nausea, vomiting, diarrhea, queasy stomach, and cramps. A second group involves sleep disturbances insomnia, hypersomnia, excessive dreaming, and nightmares. A third group includes mania and hypomania. Maxmen and Ward pointed out that these symptoms can also be experienced between doses as the blood level drops. 

Repeated low-dose administration of OP compounds can produce symptoms and signs that are not seen after single exposures to the same doses. For example, subjects given dally injections of DFF reported the additional symptoms of insomnia, excessive dreaming, emotional lability. Increased libido, paresthesias, visual hallucinations, and tremor (90) and prolonged administration in animals induces sensorimotor neuropathy. 

In seven patients treated with doxapram after anesthesia, the adverse effects were not serious and comprised excessive coughing, weeping, muscle tremor, nausea, and hysterical reactions to dreams. However, sweating, excessive salivation, and vomiting were noted in the control patients, and the authors concluded that recovery was smoother with doxapram. 

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