Disease neurological

Neurological diseases myasthenia gravis, multiple sklerosis, Guillain-Barre-syndrom... [Pg.241]

Neurological diseases Huntington s disease (A), amylotrophic lateral sclerosis (A,I), Parkinson s disease (A), traumatic brain injury (A,I), glaucoma (A)... [Pg.332]

Dyskinesias are abnormal movements, usually caused by neurological diseases or by diugs used to treat neurological (e.g., levodopa) or psychiatric diseases (e.g., neuroleptics). [Pg.454]

Caceda R, Kinkead B, Nemeroff CB (2006) Neurotensin role in psychiatric and neurological diseases. Peptides 27 2385-2404... [Pg.836]

The Complexity of Aluminum-DNA Interactions Relevance to Alzheimer s and Other Neurological Diseases... [Pg.181]

HCMV is widespread in the human population. In immunocompetent individuals, the infection is inapparent or associated with mild symptoms. However, HCMV is frequently transmitted perinatally and is the leading cause of neurological disease and hearing loss in congenitally infected newborns, affecting some 8,000 newborns per year in USA alone (Arvin and Alford 1990). Furthermore, following the first 100 days after transplantation, HCMV-induced pneumonia develops in about 50%... [Pg.164]

The limited toxicity data available for endosulfan suggest that several subgroups of the population may be more susceptible to endosulfan exposure than the general population. These subgroups include the unborn and neonates the elderly and people with liver, kidney, or neurological diseases, - effects that have been better characterized in animal studies. [Pg.183]

SEVERAL NEUROLOGIC DISEASES RESULT FROM ALTERED PROTEIN CONFORMATION... [Pg.37]

Adapted from Harding AE Neurological disease and mitochondrial genes.Trends Neurol Sci 1991 14 132. [Pg.323]

Sacktor N (2002) The epidemiology of human immunodeficiency virus-associated neurological disease in the era of highly active antiretroviral therapy. J Neurovirol 8(Suppl 2) 115-121... [Pg.30]

Sacktor N, Lyles RH, Skolasky R, Kleeberger C, Seines OA, Miller EN, Becker JT, Cohen B, McArthur JC (2001) HIV-associated neurologic disease incidence changes Multicenter AIDS Cohort Study, 1990-1998. Neurology 56(2) 257-260... [Pg.30]

Bacellar H, Munoz A et al (1994) Temporal trends in the incidence of HIV-l-related neurologic diseases Multicenter AIDS Cohort Study, 1985-1992. Neurology 44(10) 1892-1900 Banki K, Hutter E et al (1998) Molecular ordering in HIV-induced apoptosis. Oxidative stress, activation of caspases, and cell survival are regulated by transaldolase. . J Biol Chem 273(19) 11944-11953... [Pg.77]

Wesselingh SL, Glass J et al (1994) Cytokine dysregulation in HIV-associated neurological disease. Adv Neuroimmunol 4(3) 199-206... [Pg.86]

Neurological disease associated with HlV-1 infection results from primary replication within the CNS, which generally occurs during advanced stages of the disease when viral isolates reportedly expand their coreceptor usage from CCR5 to... [Pg.123]

Imayoshi I, Sakamoto M, Ohtsuka T, Takao K, Miyakawa T, Yamaguchi M, Moii K, Ikeda T, Itohara S, Kageyama R (2008) Roles of continuous neurogenesis in the structural and functional integrity of the adult forebrain. Nat Neurosci 11 1153-1161 Imitola J (2007) Prospects for neural stem ceU-based therapies for neurological diseases. Neurotherapeutics 4 701-714... [Pg.215]

Marcario JK, Riazi M, Adany I, Kenjale H, Fleming K, Marquis J, Nemon O, Mayo MS, Yankee T, Narayan O, Cheney PD (2008) Effect of morphine on the neuropathogenesis of SlVmac infection in Indian Rhesus Macaques. J Neuroimmune Pharmacol 3 12-25 Mattson MP, Haughey NJ, Nath A (2005) CeU death in HIV dementia. Cell Death Differ 12 893-904 Mayne M, Bratanich AC, Chen P, Rana F, Nath A, Power C (1998) HlV-1 tat molecular diversity and induction of TNF-alpha implications for HIV-induced neurological disease. Neuroimmunomodulation 5 184-192... [Pg.372]

The genes of the inducible and the constitutively expressed forms of NOS have been cloned and expressed. The expression of inducible NOS in the brain tissue of animals with experimentally induced neurological disorders (boma disease virus and rabies virus in rats), herpes simplex virus (mice) and experimental allergic encephalitis (in rats) suggests that NO produced by induced NOS may be a toxic fector in the pathogenesis of neurological diseases (Koprowski et /., 1993). [Pg.267]

A number of azetidine-based compounds have been disclosed in patent applications from Aventis Pharma for CBi-modulated treatment of diseases such as obesity, Parkinson s disease, schizophrenia, respiratory and neurological diseases [330-334]. Compound (556) was specifically claimed for use in two formulation patent applications [330, 331] for a stable semi-solid composition and oral emulsion composition, respectively. The optional coadministration of an agent that activates norepinephrinergic and se-rotoninergic neurotransmission (for example, sibutramine) or dopaminergic neurotransmission was also claimed for the treatment of obesity. The optional use of a dopamine agonist (for example, levodopa) was claimed... [Pg.301]

Wilson s disease is another autosomal recessive disease leading to cirrhosis. Protein abnormalities result in excessive copper deposition in body tissues. The faulty protein is responsible for facilitating copper excretion in the bile, so copper accumulates in hepatic tissue. High copper levels within hepatocytes are toxic, and fibrosis and cirrhosis may develop in untreated patients. Those with Wilson s disease usually present with symptoms of liver or neurologic disease while still in their teens. [Pg.329]

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