Nausea neck, stiff

In meningitis there may be a severe generalised headache associated with fever, nausea, neck stiffness, pain behind knees when extended (Kernig s sign) and a purpuric rash in later stages. Refer urgently any child with headache, high temperature and who is unable to bend head forward easily. 

Adults fever injection site complaints of soreness, erythema, swelling, induration and numbness, varicellalike rash upper respiratory illness headache fatigue cough myalgia disturbed sleep nausea diarrhea stiff neck irritability nervousness constipation... 

In the non-AIDS patient, the symptoms of cryptococcal meningitis are nonspecific. Headache, fever, nausea, vomiting, mental status changes, and neck stiffness are generally observed. In AIDS patients, fever and headache are common, but meningismus and photophobia are much less common than in non-AIDS patients. 

In the headache evaluation, diagnostic alarms should be identified. These include acute onset of the first or worst headache ever, accelerating pattern of headache following subacute onset, onset of headache after age 50 years, headache associated with systemic illness (e.g., fever, nausea, vomiting, stiff neck, and rash), headache with focal neurologic symptoms or papilledema, and new-onset headache in a patient with cancer or human immunodeficiency virus infection. 

Hypertensive crisis, marked by severe hypertension, occipital headache radiating frontally, neck stiffness or soreness, nausea, vomiting, sweating, fever or chilliness, clammy skin, dilated pupils, palpitations, tachycardia or bradycardia, and constricting chest pain. 

Most patients treated for neurocysticercosis with praziquantel develop an early cerebrospinal fluid reaction a similar late reaction, some 2 weeks after treatment has finished, has also been described (16). In both cases clinical signs and sjmptoms can include papilledema, headache, nausea, vomiting, neck stiffness, and even focal seizures. Glucocorticoids can usually prevent or relieve both the early and late reactions, but they can also reduce efficacy by lowering plasma concentrations of the drug by some 50% (17). 

Adverse reactions following therapeutic use include sedation, dizziness, insomnia, agitation, tardive dyskinesia, dysphoria, dystonic reactions, tachycardia, syncope, anorexia, nausea, vomiting, constipation, diarrhea, and dyspepsia. The most frequently reported dystonic reactions include akathisia, stiff neck, stiff or protruding tongue, and tremor. 

The manufacturer recommended that fenfluramine should not be used in patients with a history of depression, or during treatment with antidepressants (especially the MAOIs), and there should be an interval of 3 weeks between stopping the MAOIs and starting fenfluramine. A woman taking phenelzine developed severe headache, neck stiffness and nausea within an hour of taking fenfluramine 20 mg, and then collapsed and re-... 

One of the earliest symptoms of hypertensive crisis is headache (usually occipital), followed by a stiff or sore neck, nausea, vomiting, sweating, fever, chest pain, dilated pupils, and bradycardia or tachycardia. If a hypertensive crisis occurs, immediate medical intervention is necessary to reduce the blood pressure Strokes (cerebrovascular accidents) and death have been reported. 

Patient 1. This 24-year-old male university student was brought to the emergency department at 1600 h by his roommate. He was delirious and had a depressed level of consciousness. Although he had been well the previous day, that morning he had complained of a fever, severe headache, severe neck and back stiffness, nausea, and vomiting. He had become progressively unwell over 7-8 hours. On physical examination he was acutely ill with a temperature of 40°C. He was delirious and had neck rigidity with severe resistance to any attempt to passively flex his neck. A CT scan of his brain was normal. A spinal tap was performed and cerebrospinal fluid (CSF) was removed it was cloudy. 

Neurology recognizes that relatively minor head trauma—even without the delirium, loss of consciousness, and seizures associated with ECT— frequently produces chronic mental dysfunction and personality deterioration (Bernat et al., 1987). If a woman came to an emergency room in a confusional state from an accidental electrical shock to the head, perhaps from a short circuit in her kitchen, she would be treated as an acute medical emergency. If the electrical trauma had caused a convulsion, she might be placed on anticonvulsants to prevent a recurrence of seizures. If she developed a headache, stiff neck, and nausea—a triad of symptoms typical of post-ECT patients—she would probably be admitted for observation to the intensive care unit. Yet ECT delivers the same electrical closed-head injury, repeated several times a week, as an alleged means of improving mental function. ECT is electrically induced closed-head injury. 

Signs occipital headache, stiff neck, nausea and vomiting, chest pain, dilated pupils, nosebleed, elevated blood pressure... 

The incubation period of TBE is usually between 7 and 14 days and is asymptomatic. Shorter incubation times have been reported after milk-borne exposure. A characteristic biphasic febrile illness follows, with an initial phase that lasts 2 to 4 days and corresponds to the viremic phase. It is non-specific with symptoms that may include fever, malaise, anorexia, muscle aches, headache, nausea, and/or vomiting. After about 8 days of remission, the second phase of the disease occurs in 20 to 30% of patients and involves the central nervous system with symptoms of meningitis (c.g., fever, headache, and a stiff neck) or encephalitis (e.g., drowsiness, confusion, sensory disturbances, and/or motor abnormalities such as paralysis) or meningoencephalitis. In contrast to RSSE, TBE is more severe in adults than in children. 

In high doses (aqueous extract of 30 g as a single dose), abdominal pain, nausea, vomiting, vertigo, impaired consciousness, and stiff neck have been reported (Bensky et al. 2004). 

At the time of her first visit, she reported a slight headache and a feeling of nausea, as well as the neck pain and stiffness. She had been well until the time of the accident and was using no regular medications. She seldom experienced headaches and had no past history of musculoskeletal problems or injuries. 

---