Mercury exposure

Burgess, N.M. and Meyer, M.W. (2008). Methyl mercury exposure associated with reduced productivity in common loons. Ecotoxicology 17, 83-92. [Pg.341]

Wildlife indicators of mercury exposure and trends are important elements of a comprehensive approach to assess mercury in the environment and the monitoring of trends that may assist regulators and the regulated community in long-term evalnation of the need and usefulness of mercury somce controls. It is important to understand, however, that bioindicator data alone are insufficient to answer snch critical qnestions as identification of mercniy sonrces, or the relative importance of local, regional, and global inputs of mercury somces to atmospheric deposition and errvirorrmerrtal loading in specific areas. [Pg.127]

The use of feathers as an indicator of mercury exposure requires imderstanding the life cycle and ecology of each species. Because some species migrate, the location of exposme can be problematic. However, this disadvantage can be ehminated by... [Pg.148]

Wildlife toxicologists should be attuned to developments in human health mercury, as assays that have been used successfully on humans may be suitable or adaptable for other vertebrate species. Echeverria and co-workers (Echeverria et al. 2005, 2006 Heyer et al. 2006) have characterized a gene encoding coproporphyrinogen oxidase, a gene in the heme biosynthetic pathway. Polymorphism in this gene predicts differential response to elemental mercury exposure in human subjects. Plans to modify this assay for other mercury species in matrices from wildlife are under way. [Pg.152]

Identify the role of loadings versus other factors (e.g., food web changes) in mercury exposure changes ... [Pg.158]

Champoux L, Masse D, Evers DC, Lane O. (In press). Assessment of mercury exposure and potential effects in common loons in Quebec. Hydrobiologia. [Pg.171]

Counard CJ. 2000. Mercury exposure and effects on Common Loon Gavia immer) behavior in the Upper Midwestern United States. University of Minnesota. MS thesis. [Pg.172]

Dawson MA. 1982. Effects of long-term mercury exposure on hematology of striped bass, Morone-Saxatilis. Fish Bull 80 389-392. [Pg.172]

Echeverria D, Woods JS, Heyer NJ, Rohhnan D, Farin FM, Li T, Garabedian CE. 2006. The association between a genetic polymorphism of coproporphyrinogen oxidase, dental mercury exposure and neurobehavioral response in humans. Neurotoxicol Teratol 28 39-48. [Pg.173]

Evers DC, Burgess N, Major A, Champoux L, Goodale W, Taylor R. 2005. Patterns of mercury exposure in the avian community of northeastern North America. Ecotoxicology 14 193-221. [Pg.174]

Evers DC, Reaman P. 1997. A comparison of mercury exposure between artificial impoundments and natural lakes measured in Common Loons and their prey, sediments, and game fish. BioDiversity Research Institute, Fahnouth, ME. [Pg.174]

Fevold BM, Meyer MW, Rasmussen PW, Temple SA. 2003. Bioaccumulation patterns and temporal trends of mercury exposure in Wisconsin common loons. Ecotoxicology 12 83-93. [Pg.174]

Ilback NG, Sundberg J, Oskarsson A. 1991. Methyl mercury exposure via placenta and milk impahs natural killer (NK) cell function in newborn rats. Toxicol Left 58 149-158. [Pg.178]

Kenow KP, Gutreuter S, Elines RK, Meyer MW, Fournier F, Karasov WEI. 2003. Effects of methyl mercury exposure on the growth of juvenile common loons. Ecotoxicology 12 171-182. [Pg.179]

Meyer MW, Evers DC, Hartigan JJ, Rasmussen PS. 1998. Patterns of Common Loon Gavia immer) mercury exposure, reproduction, and survival in Wisconsin, USA. Environ Toxicol Chem 17 184-191. [Pg.181]

Nisbet I, Montoya J, Burger J, Hatch J. 2002. Use of stable isotopes to investigate individual differences in diets and mercury exposures among common terns Sterna hirundo in breeding and wintering grounds. Mar Ecol Progr Ser 242 267-274. [Pg.182]

Ponce RA, BarteU SM, Kavanagh TJ, Woods JS, Griffith WC, Lee RC, Takaro TK, Faustman EM. 1998. Uncertainty analysis methods for comparing predictive models and biomarkers a case study of dietary methyl mercury exposure. Regulatory Toxicol Pharmacol 28 96-105. [Pg.183]

Scheuhammer AM, Atchison CM, Wong AHK, Evers DC. 1998a. Mercury exposure in breeding common loons (Gavia immer) in central Ontario, Canada. Environ Toxicol Chem 17 191-196. [Pg.185]

Holmes P, James KAF, Levy LS (2009) Is low-level environmental mercury exposure of concern to human health Sci Total Environ 408 171-182... [Pg.439]

Angelow, R.V. and D.M. Nicholls. 1991. The effect of mercury exposure on liver mRNA translatability and metallothionein in rainbow trout. Comp. Biochem. Physiol. 100C 439-444. [Pg.425]

Diamond, S.A., M.C. Newman, M. Mulvey, and S.I. Guttman. 1991. Allozyme genotype and time-to-death of mosquitofish, Gambusia holbrooki, during acute inorganic mercury exposure a comparison of populations. Aquat. Toxicol. 21 119-134. [Pg.428]

Fleming, L.E., S. Watkins, R. Kaderman, B. Levin, D.R. Ayyar, M. Bizzio, D. Stephens, and J.A. Bean. 1995. Mercury exposure to humans through food consumption from the Everglades of Florida. Water Air Soil Pollut. 80 41-48. [Pg.429]

Gentile, J.H., S.M. Gentile, G. Hoffman, J.F. Heltshe, and N. Hairston, Jr. 1983. The effects of a chronic mercury exposure on survival, reproduction and population dynamics of Mysidopsis bahia. Environ. Toxicol. Chem. 2 61-68. [Pg.430]

Hill, E.F. and J.H. Soares, Jr. 1984. Subchronic mercury exposure in Cotumix and a method of hazard evaluation. Environ. Toxicol. Chem. 3 489-502. [Pg.431]

Scheuhammer, A.M. and PJ. Blancher. 1994. Potential risk to common loons (Gavia immer) from methyl-mercury exposure in acidified lakes. Hydrobiologia 279/280 445-455. [Pg.439]

Selenium lessens the toxicity of divalent mercury in animals, the protection being less at continuous mercury exposure. Selenium has been found to affect the distribution of mercuric mercury in mice [134], rats [135], rabbits [136, 137] and pigs [ 138]. Mercury forms a mercury-selenium protein complex with selenium with little biological activity [139]. Mercury is thus retained longer in the blood, liver and spleen and as a consequence lessens accumulation in the kidney. In fish, selenium pretreatment probably retarded mercury uptake rather than promoting mercury excretion [140]. [Pg.198]

In several epidemiological studies, no increased risk for congenital abnormalities, stillbirths, or spontaneous abortions was observed with occupational exposure to mercury. Exposure of pregnant rats on gestational days 10-15 at 0.5mg/m resulted in an increased incidence of resorptions gross cranial defects occurred at this dose when it was administered throughout the entire gestational period. "... [Pg.437]

Blood and urine mercury concentrations are commonly used as biomarkers of mercury exposure. ... [Pg.438]

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