Disease Gastroesophageal reflux

The key factor in the development of GERD is the abnormal reflux of gastric contents from the stomach into the esophagus. [Pg.263]

I Foods and Medkalions That May Worsen I Gastroesophageal Reflux Disease Symptoms [Pg.264]

Omeprazole given empirically in standard or double doses as a therapeutic trial for diagnosing GERD may be as beneficial as ambulatory pH monitoring while also being less expensive, more convenient, and more readily available. However, there is no standard dosing regimen standard-dose or double-dose omeprazole has been used. [Pg.265]

Esophageal manometry to evaluate motiUty should be performed in any patient who is a candidate for antireflux surgery. It is useful in determining which surgical procedure is best for the patient. [Pg.265]

The goals of treatment are to reduce or eliminate symptoms, decrease the frequency and duration of gastroesophageal reflux, promote healing of the injured mucosa, and prevent the development of complications. [Pg.265]

Upon completion of the chapter, the reader will be able to [Pg.257]

Explain the underlying causes of gastroesophageal reflux disease (GERD). [Pg.257]

Differentiate between typical, atypical, and complicated symptoms of GERD. [Pg.257]

Determine which diagnostic test should be recommended based on the patient s clinical presentation. [Pg.257]

Identify the desired therapeutic outcomes for patients with GERD. [Pg.257]

On the pathophysiological side, hyperactive nNOS has been implicated in A/-methyl-D-aspartate (NMDA)-receptor-mediated neuronal death in cerebrovascular-stroke. Some disturbances of smooth muscle tone within the gastrointestinal tract (e.g., gastroesophageal reflux disease) may also be related to an overproduction of NO by nNOS in peripheral nitrergic nerves. [Pg.863]

Gastrin Releasing Peptide Gastroesophageal Reflux Disease (GERD)... [Pg.1492]

Adapted from Williams DB, Schade RR. Gastroesophageal reflux disease. In DiPiro JT, Talbert RL, Yee GC, et al, eds. Pharmacotherapy A Pathophysiologic Approach. 6th ed. McGraw-Hill, New York McGraw-Hill 2005 615, with permission. [Pg.259]

GERD gastroesophageal reflux disease H2RA histamine2-receptor antagonist LES lower esophageal sphincter... [Pg.267]

DeVault KR, Castell DO. Updated guidelines for the diagnosis and treatment of gastroesophageal reflux disease. Am J Gastroenterol 2005 100 190-200. [Pg.267]

Wong WM, Wong BCY. Definition and diagnosis of gastroesophageal reflux disease. J Gastroenterol Hepatol 2004 19 S26-S32. [Pg.267]

FIGURE 15-2. Approach to the patient presenting with ulcer-like symptoms. GERD, gastroesophageal reflux disease HP Helicobacter pylori ... [Pg.275]

Refer to Chapter 14 on gastroesophageal reflux disease for more information on the PPIs. The PPI omeprazole is superior to both ranitidine and misoprostol for preventing recurrence of NSAID-associated PUD. In one study, omeprazole 20 mg daily was compared to misoprostol 200 meg twice daily for NSAID-associated PUD prevention. At 6 months, the omeprazole-treated group had significantly fewer ulcers than those taking misoprostol. Furthermore, more patients discontinued ulcer prophylaxis in the misoprostol group due to adverse events.26... [Pg.278]

ETEC enterotoxigenic Escherichia coli GERD gastroesophageal reflux disease GI gastrointestinal... [Pg.321]

Father died at age 45 from coronary disease, mother alive at age 62 with type 2 diabetes mellitus, hypertension, hyperlipidemia, and gastroesophageal reflux disease... [Pg.331]

Weight loss, initially intentional through diet and exercise, but now unintentionally excessive Type 2 diabetes, requiring insulin Gastroesophageal reflux disease... [Pg.343]

Gastrointestinal Gastroesophageal reflux disease, gastrointestinal (Gl) bleeding, and abdominal distention. [Pg.378]

GERD Gastroesophageal reflux disease IBW Ideal body weight... [Pg.1555]

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