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Dysplasia

Dysplasia is a special type of cell adaptation that literally means disordered growth, and it is usually associated with neoplasia or preneoplastic changes. Dysplasia implies a loss in the uniformity of the individual cells and a loss in the normal tissue growth pattern. For example, a dysplastic area involving the epidermal layers of the skin might contain jumbled, atypical epithelial cells with loss of their normal polarity and orderly differentiation. [Pg.289]


Development of MMP null mice carrying specific MMP deletions has provided an opportunity to explore the role of MMPs in normal development as well as in such diverse conditions and diseases as skeletal dysplasias, coronary artery and heart disease, arthritis, cancer, and brain disorders. [Pg.748]

Arrhythmogenic Right Ventricular Dysplasia/ Cardiomyopathy Aiteriogenesis Arteriosclerosis Arylhydrocarbon Receptor L-Ascorbic Acid ASF Family of Transporters Asn-linked Glycosylation ASON... [Pg.1487]

Achondroplasia (MIM 100800) and thanatophoric dysplasia (MIM 187600) Mutations In the gene encoding FGFR3. [Pg.551]

Thanatophoric (Gk thanatos "death" + pharos "bearing") dysplasia is the most common neonatal lethal skeletal dysplasia, displaying features similar to those of homozygous achondroplasia. [Pg.551]

After our formulation of the hypothesis in 1975, many pieces of evidence have come to support the basic theme. A positive association of dysplasia, representing advanced stages of the process, with nitrite levels in the gastric juice has been reported in England and Colombia (, . Higher than expected... [Pg.327]

Autoimmune nephrotic syndromes Bronchopulmonary dysplasia Mineral dust pneumoconiosis... [Pg.200]

Functional bowel problems (e.g., Hirschsprung s disease, neuronal intestinal dysplasia, pseudo-obstruction, proteinlosing enteropathy, microvillous inclusion disease)... [Pg.831]

Precancerous cells have cellular changes that are abnormal but not yet malignant and may be described as hyperplastic or dysplastic. Hyperplasia occurs when a stimulus is introduced and reverses when the stimulus is removed. Dysplasia is an abnormal change in the size, shape, or organization of cells or tissues. [Pg.1280]

Dysplasia Abnormal change in size, shape, or organization of cells in a tissue. [Pg.1565]

NakayamaK, IshidaN, Shirane Metal 1996 Mice lacking p27(Kipl) display increased body size, multiple organ hyperplasia, retinal dysplasia, and pituitary tumors. Cell 85 707-720... [Pg.106]

Enteric Neuropathies. Different kinds of familial visceral neuropathies have been described the dominant type 1 [134], the recessive type 2 [135] and a recessive form with calcified basal ganglia [134], Furthermore, aganglionosis of the small bowel (Hirschsprung s disease) [136], hypergan-glionosis (neurofibromatosis) [137], neuronal intestinal dysplasia [138] and Parkinson s disease [139] are neuropathies to consider. The recognition of the pacemaker cells of the small bowel, the interstitial cells of Cajal, has prompted studies to detect abnormalities of these cells, another possible cause of pseudoobstruction [140],... [Pg.13]

Milla PJ, Smith VV Intestinal neuronal dysplasia. J Pediatr Gastroenterol Nutr 1993 17 ... [Pg.21]

The application of flavonoids for the treatment of various diseases associated with free radical overproduction is considered in Chapter 29. However, it seems useful to discuss here some studies describing the activity of flavonoids under certain pathophysiological conditions. Oral pretreatment with rutin of rats, in which gastric lesions were induced by the administration of 100% ethanol, resulted in the reduction of the area of gastric lesions [157]. Rutin was found to be an effective inhibitor of TBAR products in the gastric mucosa induced by 50%i ethanol [158]. Rutin and quercetin were active in the reduction of azoxymethanol-induced colonic neoplasma and focal area of dysplasia in the mice [159], Chemopreventive effects of quercetin and rutin were also shown in normal and azoxymethane-treated mouse colon [160]. Flavonoids exhibited radioprotective effect on 7-ray irradiated mice [161], which was correlated with their antioxidative activity. Dietary flavones and flavonols protected against the toxicity of the environmental contaminant dioxin [162], Rutin inhibited ovariectomy-induced osteopenia in rats [163],... [Pg.867]

Tetracycline Discolored teeth, enamel dysplasia, and bone growth disturbances in children... [Pg.16]

Daily energy requirements for children are approximately 150% of basal metabolic rate with additional calories to support activity and growth (Table 57-5). Requirements increase with fever, sepsis, major surgery, trauma, burns, long-term growth failure, and chronic conditions (e.g., bronchopulmonary dysplasia, congenital heart disease, and cystic fibrosis). [Pg.664]

Yamamoto T, Ozono K, Kasayama S, Yoh K, Hiroshima K, Takagi M, Matsumoto S, Michigami T, Yamaoka K, Kishimoto T, Okada S (1996) Increased IL-6-production by cells isolated from the fibrous bone dysplasia tissues in patients with McCune-Albright syndrome. J Clin Invest 98 30-35... [Pg.192]

Mutations in COL9A1, COL9A2, and COL9A3 cause multiple epiphyseal dysplasia (MED), an autosomal dominant chondrodysplasia. Also, mutations in COMP, which interacts with type IX collagen, cause MED. [Pg.490]

G, alpha Insertions/dele- syndrome fibrous dysplasia of bone cafe-au-lait skin lesions sexual precocity pituitary, thyroid, or adrenal tumors Albright s hereditary with constitutive cAMP production Inactivating G a (86-89)... [Pg.87]

Shenker, A., Weinstein, L. S., Sweet, D. E., and Spiegel, A. M. (1994) An activating Gs alpha mutation is present in fibrous dysplasia of bone in the McCune-Albright syndrome. J. Clin. Endocrinol. Metab. 79, 750-755. [Pg.102]


See other pages where Dysplasia is mentioned: [Pg.222]    [Pg.222]    [Pg.419]    [Pg.707]    [Pg.1098]    [Pg.136]    [Pg.238]    [Pg.554]    [Pg.98]    [Pg.322]    [Pg.881]    [Pg.1513]    [Pg.136]    [Pg.153]    [Pg.58]    [Pg.92]    [Pg.85]    [Pg.107]    [Pg.144]    [Pg.181]    [Pg.34]    [Pg.41]    [Pg.481]    [Pg.483]    [Pg.489]    [Pg.84]   
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See also in sourсe #XX -- [ Pg.24 ]

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See also in sourсe #XX -- [ Pg.139 , Pg.213 ]




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Angiogenic squamous dysplasia

Arrhythmogenic Right Ventricular Dysplasia

Arrhythmogenic Right Ventricular Dysplasia Cardiomyopathy

Arteriohepatic dysplasia

Bone dysplasia

Bronchopulmonary dysplasia

Cervical dysplasia

Cleidocranial dysplasia

Cocaine fibromuscular dysplasia

Cortical dysplasia

Deferoxamine bone dysplasia

Dysplasia markers

Dysplasia urinary

Epithelial cell dysplasia

Fibromuscular dysplasia

Fibrous dysplasia

Hip Dysplasia

Intraepithelial cervical dysplasia

Intraepithelial dysplasia

Kidney cystic dysplasia

Malignant dysplasia

Multicystic dysplasia

Neonates bronchopulmonary dysplasia

Neuronal intestinal dysplasia, intestine

Osteofibrous dysplasia

Polyp Dysplasia

Renal dysplasia

Skeletal dysplasias

Thanatophoric dysplasia

Tooth dysplasia

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