Chloride poisoning

Figure 2. Study of chloride poisoning hot spot location vs. time on stream...

He F Lu B, Zhang S, et ah Chronic allyl chloride poisoning. An epidemiology clinical, toxicological and neuropathological study. G Ital Med Lav 7 5-15, 1985... [Pg.34]

Spevak L, Nadj V, Felle D Methyl chloride poisoning in four members of a family. Br J IndMed 33 272-274, 1976... [Pg.463]

Gudmundsson G Letter. Methyl chloride poisoning 13 years later. Arch Environ Health MlZCi-TM, 1977... [Pg.463]

Egfjord, M., J.A. Jansen, H. Flachs, and J.S. Schou. 1988. Combined boric acid and cinchocaine chloride poisoning in a 12-month-old infant evaluation of haemodialysis. Human Toxicol. 7 175-178. [Pg.1583]

PHOSGENE Carbonyl chloride, Chloroformyl chloride Poisonous Gas or Liquid, Class A 4 0 0 ... [Pg.107]

Ku elova, M., Gemy, J., Hlavova, S., Hub, M., Kunor. V. Popler, A. (1975) Lethal methylene chloride poisoning with severe chilblains. Pracov. Lek., 27, 317-319 (in Czech)... [Pg.307]

Manno. M., Chirillo. R.. Daniotti, G, Cocheo, V. Albrizio, F. (1989) Carboxyhaemoglobin and fatal methylene chloride poisoning. Lancet, ii, 274... [Pg.307]

In some cases, the reaction of silicon and methanol has been optimized for formation of (MeO)4Si. As discussed above, thiophene addition favored formation of (MeO SiH. Both thiophene and propyl chloride poison copper copper poisoning seems to favor formation of the trialkoxysilane. High-temperature pretreatment disfavors trialkoxysilane formation copper is formed on the surface of the silicon during pretreatment at 450 °C98. Metallic Cu catalyzes dehydrogenation of alcohols and favors formation of (RO)4Si. Workers from Tonen Corporation reported 50% conversion of silicon to make (MeO Si with 92% selectivity if silicon, methanol and Cu(OMe)2 were pretreated (lower conversion and selectivity without pretreatment) and then reacted at 180 °C and 1 atmosphere99. [Pg.1591]

The mercury(n) iodide reagent is prepared by dissolving 6.6 g of mercury(n) chloride (POISONOUS) in a solution of 16.3 g of potassium iodide in 16.3 ml of water and adding 12.5 ml of 10 per cent sodium hydroxide solution. [Pg.1237]

Cherroet G, Bumel D, Lehr PR. 1996. [Effect of intraperitoneal aluminum chloride poisoning in the young and adult rat Aluminemia, urinary elimination and cerebral choline acetyltransferase activity]. Bull Acad Soc lorraines sci 35 123-136. (French). [Pg.299]

Graham CF. 1934. Barium chloride poisoning [abstract], J Am Med Assoc 102 1471. [Pg.115]

Talwar KK, Sharma BK. 1979. Myocardial damage due to barium chloride poisoning. Indian Heart J 31 244-245. [Pg.127]

Wetherill SF, Guarino MJ, Cox RW. 1981. Acute renal failure associated with barium chloride poisoning. Ann Intern Med 95 187-188. [Pg.129]

Chlorinated hydrocarbons (CHCs) are widely used in industry but bring both environmental and health risks 5,120 catalytic oxidation is a low cost method for their destruction. The most active catalysts are the platinum group metals supported on alumina, but high temperature is needed to obtain a satisfactory rate and to overcome chloride poisoning,121 but hydrogen chloride attacks the alumina support, so the use of other supports that... [Pg.301]

Figure 5.8.B Cyanogen chloride poison gas delivered by mortar shells, Panama, 1944. Photographs by U.S. Army. Pictures obtained courtesy Archives CBS 60 Minutes.

Singer AJ, Mofenson HC, Caraccio TR, and Ilasi J (1994) Mercuric chloride poisoning due to ingestion of a stool fixative. Journal of Toxicology Clinical Toxicology 32(5) 577-582. [Pg.1620]

Chugh KS, Singhal PC, Uberoi HS. 1978. Rhabdomyolysis and renal failure in acute mercuric chloride poisoning. Med J Aust 2 125-126. [Pg.592]

Usually, these catalysts are characterized in terms of their efficiency in some test reaction, usually CO oxidation, under arbitrary conditions (O2 and CO concentrations, humidity, flow rates, and catalyst time on stream) that would be difficult to reproduce in another laboratory. Characterization on any length scale, from molecular to morphological, is extremely difficult. In many cases, it is uncertain whether all the reactants and by-products have been removed from the catalyst surface. It is reported that chlorides poison the active sites. This raises the question of a role for gold ions, and these, rather than Au(0), are reported to be the active sites in the water-gas shift reaction. ... [Pg.1807]

We have attempted to support this conclusion by Inoculating seawater with surface material gently scraped off the surfaces of nodosum and F. vesiculosis plants and monitoring for polybromomethane formation. In both cases bromoform concentrations In the water were observed to Increase (Figure 3). Mercuric chloride poisoning, 0.2 pm filtering, and autoclaving the Inoculated seawater prevented this bromoform production. Thus, some component of the epiphytic community may be Involved. [Pg.320]

Rioux, J.P. and Myers, R.A., Hyperbaric oxygen for methylene chloride poisoning report of two cases, Ann. Emerg. Med., 18, 691-695, 1989. [Pg.386]

Rioux JP and Myers RA (1988). Methylene chloride poisoning a pragmatic review. J Emerg Med, 6, 227-238. [Pg.610]

AMA Archives of Industrial Hygiene and Occupational Medicine 8 328-334,1953 Jones AM Methyl chloride poisoning. Q J Med 35 29-43, 1942... [Pg.213]

Kegel AH, McNally WD, Pope AS Methyl chloride poisoning from domestic refrigerators. JAMA 93 353-358, 1929... [Pg.213]

TABLE 17-3. Physical signs and symptoms of vinyl chloride poisoning... [Pg.265]

Suciu I, Prodan L, Ilea E, et al Clinical manifestations in vinyl chloride poisoning. Ann N Y Acad Sci 246 53-69, 1975... [Pg.269]

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