C6 glioma cells

Zhong, H., Guerrero, S. W., Esbenshade, T. A., and Minneman, K. P. (1996). Inducible expression of 3 - and P2-adrenergic receptors in rat C6 glioma cells Functional interactions between closely related subtypes. Mol. Pharmacol. 50 175-184. 

Bohn LM, Belcheva MM, Coscia CJ (2000) p-opioid agonist inhibition of kappa-opioid receptor-stimulated extracellular signal-regulated kinase phosphorylation is dynamin-dependent in C6 glioma cells. J Neurochem 74 574-581... 

Tonner LE, Katz DI, Heiman AS. 1997. The acute effect of lead acetate on glucocorticoid receptor binding in C6 glioma cells. Toxicology 116 109-122. 

Bouzier, A. K., Voisin, R, Goodwin, R. etal. Glucose and lactate metabolism in C6 glioma cells evidence for the preferential utilization of lactate for cell oxidative metabolism, Dev. Neurosci. 20 331-338,1998. 

Brasuel M, Kopelman R, Miller TJ, Tjalkens R, Philbert MA (2001) Fluorescent nanosensors for intracellular chemical analysis decyl methacrylate liquid polymer matrix and ion-exchange-based potassium PEBBLE sensors with real-time application to viable rat C6 glioma cells. Anal Chem 73 2221-2228... 

CYPl lAl mRNA in adult rat brain, primary glial cultures, C6 glioma cells (Mellon and Deschepper, 1993 Zhang et al., 1995). 

Zhang W, Yamada H, Sakai N, Nozawa Y (1993) Sensitization of C6 glioma cells to radiation by staurosporine, a potent protein kinase C inhibitor. J Neurooncol 15 1-7... 

Ayasolla K., Singh A. K., and Singh I. (2002). Vitamin E restores amyloid peptide (25-35) induced changes in cellular redox in C6 glioma cells Implications to Alzheimers disease. In Pasquier C. (ed.), XI Biennial Meeting of the Society for Free Radical Research International. Monduzzi Editore, 40128 Bologna, pp. 503-508. 

Zimmer DB, Van Eldik LJ. 1989. Analysis of the calcium-modulated proteins, S100 and calmodulin, and their target proteins during C6 glioma cell differentiation. J Cell Biol 108(1) 141—151. 

In order to further elucidate the fate of LCM in tumor cells, their subcellular localization was determined using chemical markers for various organelles. Permeabilized C6 glioma cells were treated with propidium iodide to label nuclei, WGA for the Golgi apparatus and the plasma membrane, and Rhodamine 123 for mitochondria. None of these subcellular compartments were found to contain LCM. In order to identify intracellular acidic compart-... 

C6 glioma cells were grown in F-10 medium supplemented with 10% horse serum and 2.5% fetal calf serum. Cells were plated on glass coverslips (22 mm2) and used when they reached 50% confluence. (LCM, lipid-coated microbubbles S.D., standard deviation.)... 

C6 glioma cells with the Eu3+ complex (10-500 pM, 45 min and 1 h at 37 °C) provided clear images consistent with the localization of the complex at mitochondria. Incubations of the cells at 4 °C, on the other hand, resulted in no probe uptake by the cells, which indicated that the lanthanide probe entered the cells through some biological activities. Finally, the authors performed luminescence and MR imaging experiment by using cocktail mixtures of Eu3+ and Gd3+ complexes (for example, Eu3+ Gd3+ = 40 60), and successfully obtained nice luminescence and MR images from the same population of C6 cells. 

Le Grand B, Panissie A, Pauwels PJ, John GW. Activation of recombinant h5-HT1B and h5-HT1D receptors stably expressed in C6 glioma cells produces increases in Ca2+-dependent K+ current. Naunyn Schmiedebergs Arch Pharmacol 1998 358 608-615. 

Bartrup JT, Newberry NR. 5-IIT2A receptor-mediated outward current in C6 glioma cells is mimicked by intracellular IP3 release. Neuroreport 1994 5 1245-1248. 

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