Asthma incidence

King, M.E., Mannino, D.M., and Holguin, F., Risk factors for asthma incidence. A review of recent prospective evidence, Panminerva Med., 46, 97, 2004. 

Gilliland FD, Islam T, Berhane K, et al. Regular smoking and asthma incidence in adolescents. Am J Respir Crit Care Med 2006 174(10) 1094 100. 

Yeatts KB, Sly P, Shore S, Weiss S, Martinez F, Geller A, Bromberg P, Enright P, Keren H, Weissman D, Selgrade M. A brief targeted review of susceptibility factors, environmental exposures, asthma incidence, and recommendations for future asthma incidence research. Environ Health Perspect 2006 114 634-640. 

Yunginger JW et al. (1992) A conununity-based study of the epidemiology of asthma. Incidence rates, 1964-1983. Am Rev Respir Dis 146(4) 888-894... 

The association between flavonoid intake and chronic diseases has been studied in Finland in 10054 men and women. The incidence of cerebrovascular disease was lower at higher kaempferol, nar-inginin, and hesperetin intakes. Asthma incidence was lower at higher quercetin, naringinin, and hesperetin intakes. Men with high quercetin intakes had a lower... 

Epidemiological studies of nickel-producing and nickel-using workers seldom indicate excess mortaUty from nonmalignant respiratory disease. Evidence for such effects exists mainly as a few reports of isolated incidents of asthma, pulmonary fibrosis, chronic bronchitis, and emphysema in nickel workers. Nickel may or may not play a causal role in these incidents (131). 

Increase in incidence and severity of asthma attacks moderate < ye irritation... 

For children, Uie new standard will reduce respiratory problems, such as asthma tUtacks. It will result in one million fewer incidences of decreased lung function in cliildren each year... 

Respiratory allergies and infections are the most common form of illness in the United States and Europe and account for more missed school and work days than any other disease [1], A substantial body of experimental work has clearly shown that airborne toxicants such as tobacco smoke, ozone, and other air pollutants can alter many aspects of the host defense network to either decrease resistance to infection, or exacerbate respiratory allergies and asthma [2], Exposure to air toxicants can suppress a number of key host defenses including mucociliary clearance in the airways, pulmonary macrophage function, and development of specific immune responses such as IgG antibody production and cell mediated immunity. In contrast, immune stimulation in the form of increased T cell activity and IgE antibody formation has also has been shown to occur under some circumstances, resulting in increased incidence or severity of allergic lung disease. 

Other personal risk factors such as increased body mass index are being considered as to whether they are important to asthma [45,46], Whether emerging risk factors, along with the general increase in atopic diseases from the mid-1960s to the present [47] will have an impact on development and incidence of OA remain to be determined. 

Gautrin, D., Ghezzo, H., and Malo, J.L., Rhinoconjunctivitis, bronchial responsiveness, and atopy as determinants for incident non-work-related asthma symptoms in apprentices exposed to high-molecular-weight allergens, Allergy, 58, 608, 2003. 

Archambault, S. et al., Incidence of sensitization, symptoms, and probable occupational rhinoconjunctivitis and asthma in apprentices starting exposure to latex, J. Allergy Clin Immunol., 107, 921, 2001. 

The most important health effects, in terms of economic damages that can be assigned monetary values, are premature mortality and increased incidence of chronic heart and lung disease. The air pollutants that have shown the strongest association with premature mortality and heart and lung disease are PM and airborne lead. PM has also been associated with hospital admissions, respiratory infections, and asthma attacks. Ozone has also been associated with mortality, hospital admissions, asthma attacks and respiratory restricted activity days (RADs), days on which a person cuts back on his or her normal activities, but does not necessarily miss work or stay in bed. S02 and NOx do not have such significant direct effects, though they do have... 

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