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US Environmental Protection Agency EPA

CHEMblST " The CHEMLlS l Pile contains chemical substances on national inventories, registered by the US Environmental Protection Agency (EPA), TTie dala in GPdHMLIS l arc from 1979 to the present, there arc more than 228 380 records (September, 2002). CHHMLlS l is updated weekly with more than 50 addilions to eUsling records or new subslances. [Pg.243]

The US Environmental Protection Agency (EPA) Risk Assessment Foriim An endocrine disrupter is an exogenous agent that interferes with the synthesis, secretion, transport, binding, action, or elimination of natural... [Pg.4]

Ethene and propene are produced as bulk feedstocks for the chemical (polymer) industry and therefore their purities are important parameters. In particular, H2S and COS are compounds which may not only cause corrosion problems in processing equipment, but also may have detrimental effects on the catalysts in use. Eurthermore, air pollution regulations issued by, among others, the US Environmental Protection Agency (EPA) require that most of the sulfur gases should be removed in order to minimize Sulfur emissions into the atmosphere. Therefore, these compounds have to be determined to the ppb level. [Pg.381]

The definitions of method detection and quantification limits should be reliable and applicable to a variety of extraction procedures and analytical methods. The issue is of particular importance to the US Environmental Protection Agency (EPA) and also pesticide regulatory and health agencies around the world in risk assessment. The critical question central to risk assessment is assessing the risk posed to a human being from the consumption of foods treated with pesticides, when the amount of the residue present in the food product is reported nondetect (ND) or no detectable residues . [Pg.60]

Finally, to avoid the parallel use of similar but not identical method validation studies to fulfil the registration requirements, e.g., of the EU, US Environmental Protection Agency (EPA) or Japanese authorities, an adaptation of different data requirements for residue analytical methods for post-registration control and monitoring purposes would help to save resources. [Pg.111]

The LODs for imidacloprid and metabolites, when determined by US Environmental Protection Agency (EPA) guidelines, are 0.021-0.024 ngL The LOQ is defined as 0.1 ng for each metabolite. Recoveries from control water samples fortified at the 0.1 and 1 ngL levels ranged between 84 and 107% for all metabolites. No relative standard deviation between the respective recoveries for any of the analytes was greater than 10%. [Pg.1325]

Alexander J. Krynitsky US Environmental Protection Agency, EPA Environmental Science Center, Fort Meade, MD, USA... [Pg.1454]

The Comprehensive Environmental Response, Compensation, and Liability Act (CERCLA) [42 U.S.C. 9601 et seq ], as amended by the Superfimd Amendments and Reauthorization Act (SARA) [Pub. L. 99-499], requires that the Agency for Toxic Substances and Disease Registry (ATSDR) develop jointly with the US. Environmental Protection Agency (EPA), in order of priority, a list of hazardous substances most commonly found at facilities on the CERCLA National Priorities List (NPL) prepare toxicological profiles for each substance included on the priority list of hazardous substances and assure the initiation of a research program to fill identified data needs associated with the substances. [Pg.597]

Despite the difficulties of evaluating the true extent of contamination, the latest estimations in Europe in 2006 set a number of 3.5 million potentially contaminated sites [23]. Of these, approximately 0.5 million were expected to require urgent treatment. The National Priorities List of the 2012 US Environmental Protection Agency (EPA) identified 1,305 superfund sites, which are defined as abandoned hazardous waste sites [24]. [Pg.5]

Toxicity and exposure studies indicate PFOA is immunosuppressive and can cause developmental problems and other adverse effects in laboratory animals, such as rodents [Lau et al (2004), Lau et al (2006)]. In 2005 the US Environmental Protection Agency (EPA) released a draft risk assessment of its potential human health effects [U S. EPA (2005)]. A subsequent review by the EPA science advisory board concluded that there is sufficient evidence to classify PFOA as likely human carcinogenic. [Pg.64]

USEPA (1985), Odor and corrosion control in sanitary sewerage systems and treatment plants, US Environmental Protection Agency, EPA 625/1-85/018, Washington DC. [Pg.127]

Among all chlorophenols, 2,4,6-trichlorophenol (TCP) and pentachlorophenol (PCP) are listed as priority pollutants by the US Environmental Protection Agency (EPA) (IRIS electronic database) and the EU [256]. In particular, PCP has been classified as a B2 probable carcinogen for humans from animal toxicity studies and human clinical data. [Pg.161]

In practice there will often be old studies or published papers that can provide adequate information to assess the properties of phase in existing substances. The US Environmental Protection Agency (EPA) has issued useful guidance on searching for literature data (a. 15). [Pg.12]

A code of two letters followed by seven digits is a reference to Registry of Toxic Effects of Chemical Substances (RTECS) of National Institute for Occupational Safety and Health/Occupational Safety and Health Administration (NIOSH/OSHA). Standard samples are commercially available for most compounds with reference to protocols of die US Environmental Protection Agency (EPA) and the US Pharmacopea (USP)74. [Pg.1053]

Jaber HM, Mabey WR, Liu AT, et al. 1984. Data acquisition for environmental transport and fate screening for compounds of interest to the Office of Emergency and Remedial Response. Washington, D.C. US Environmental Protection Agency. EPA-600/6-84-011. NTIS no. PB84-245281, 147. [Pg.122]

Short RD, Minor JL, Ferguson B, et al. 1976. The developmental toxicity of ethylene dibromide inhaled by rats and mice during organogenesis. US Environmental Protection Agency. Washington, D.C. US Environmental Protection Agency. EPA-560/6-76-018 NTIS no. PB-256659, 11. [Pg.131]

Zweidinger R, Erickson M, Cooper S, et al. 1982. Direct measurement of volatile organic compounds in breathing-zone air, drinking water, breath, blood, and urine. Washington, DC US Environmental Protection Agency. EPA 600/4-82-015. [Pg.136]

EPA 1995. Toxic chemical release inventory reporting form R and instructions- Revised 1994 version. Office of Pollution Prevention, US Environmental Protection Agency, EPA 745-K-95-051 Washington, DC. [Pg.155]

US-EPA. 1995. The use of the benchmark dose approach in health risk assessment. US Environmental Protection Agency (EPA), Office of Research and Development, Doc. EPA/630/R-94/007, Washington, DC. [Pg.208]

US Environmental Protection Agency (EPA) Summary Review of the Health Effects Associated with Phenol Health Issue Assessment, 37pp. Washington, DC, US Government Printing Office, January 1986... [Pg.570]

US Environmental Protection Agency EPA and Surgeon General Call for Radon Home Testing. Environmental News, September 12, 1988. Office of Public Affairs (A-107). [USEPA 1988]... [Pg.616]

US Environmental Protection Agency (EPA) - Integrated Risk Information System (IRIS). Online. Available HTTP (accessed 1 April 2003). [Pg.12]


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EPA

EPA (Environmental Protection

EPA (US Environmental Protection

EPA, Environmental

Environmental Protection Agency

Environmental protection

Environmental protection agency (EPA

US EPA

US Environmental Protection Agency

US,ENVIRONMENTAL

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