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Naphthalene dietary

Collier TK, LC Thomas, DC Malins (1978) Influence of environmental temprature on disposition of dietary naphthalene in coho salmon (Oncorhynchus kisutch) isolation and identification of individual metabolites. Comp Biochem Physiol 61C 23-28. [Pg.100]

Sediments and biota collected from the Hersey River, Michigan, in 1978, were heavily contaminated with phenanthrene, benz[a]anthracene, and benzo[a]pyrene when compared to a control site. Elevated PAH concentrations were recorded in sediments, whole insect larvae, crayfish muscle, and flesh of lampreys (family Petromyzontidae), brown trout (Salmo trutta), and white suckers (Catostomus commersoni), in that general order (Black et al. 1981). The polluted collection locale was the former site of a creosote wood preservation facility between 1902 and 1949, and, at the time of the study, received Reed City wastewater treatment plant effluent, described as an oily material with a naphthalene-like odor (Black et al. 1981). In San Francisco Bay, elevated PAH concentrations in fish livers reflected elevated sediment PAH concentrations (Stehr et al. 1997). In Chesapeake Bay, spot (Leiostomus xanthurus) collected from a PAH-contaminated tributary (up to 96 mg PAHs/kg DW sediment) had elevated cytochrome P-450 and EROD activity in liver and intestine microsomes (Van Veld et al. 1990). Intestinal P-450 activity was 80 to 100 times higher in fish from highly contaminated sites than in conspecifics from reference sites intestinal EROD activity had a similar trend. Liver P-450 and EROD activity was about 8 times higher in spot from the contaminated sites when compared to the reference sites. Liver P-450 activity correlated positively with sediment PAH, but intestinal P-450 activity seemed to reflect dietary exposure (Van Veld et al. 1990). The poor correlation between hepatic concentrations of PAHs and P-4501A is attributed to the rapid metabolism of these compounds (van der Weiden et al. 1994). [Pg.1361]

Environmental temperature influences the degree of hydrocarbon accumulation in marine fish. Collier et al. (10) found that depressed temperature (4° vs. 10°C) resulted in significantly (P<0.05) increased retention of dietary C-naphthalene in brain, liver, kidney, and blood of coho salmon (0. kisutch). Thus, environmental conditions can have a pronounced effect on the disposition of hydrocarbons in exposed marine species. [Pg.60]

Burreau S, Axelman J, Brogman D, et al. 1997. Dietary uptake in pike Esox incius) of some polychlorinated biphenyls, polychlorinated naphthalenes and polybrominated diphenyl ethers administered in natural diets. Environ Toxicol Chem 16 2508-2513. [Pg.415]

Another important pathway leading to PAHs is the thermal elimination reaction of benzene derivatives (Figure 12.22). For example, the elimination of water from phenolic compounds (X = OH) yields, via 1,2-didehydrobenzene (often incorrectly called ben-zyn), another reactive intermediate benzobicyclo[2,2,2]triene and naphthalene as a final product. The contribution of endogenously generated compounds to the total dietary intake of PAHs is not too significant as the contamination of food is largely exogenous. [Pg.939]

Calanus helgolandicus) accumulated more naphthalene after 24 hr when uptake was by diet as opposed to exposure in water without prey. Assessment of the importance of dietary accumulation is difficult in aquatic systems because prey can release compounds to the water, thus confounding the experimental design of assessing only dietary input. For most aquatic organisms, we would expect dietary uptake of LPAHs, such as via sediment ingestion, to have a minor impact on tissue concentrations for these compounds when prey and water are at steady state with each other. [Pg.98]

It is unclear why a different elimination rate should occur in animals depending on whether they take up PAHs through the gills or the gut. When adult female copepods Calamus helgolandicus) accumulated naphthalene through the diet, their elimination rate was approximately 20% of that for water exposure (Corner et al. 1976). After 10 d in clean water, about 30% of the naphthalene from dietary uptake remained in the tissues while only 5% from water uptake remained. These results suggest that with dietary uptake the PAH is distributed to compartments from which elimination is less rapid. [Pg.110]


See other pages where Naphthalene dietary is mentioned: [Pg.94]    [Pg.1558]    [Pg.500]    [Pg.429]    [Pg.658]    [Pg.122]    [Pg.124]    [Pg.66]    [Pg.404]    [Pg.413]    [Pg.97]    [Pg.249]    [Pg.265]   
See also in sourсe #XX -- [ Pg.60 ]




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