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Diseases Minamata

Minamata Disease is defined as neuropathy arising from intake of fish and shellfish containing high concentrations of methylmer-cury. The outbreak is dependent on factors that include mercury concentrations in water, bioconcentration and biomagnification of mercuric compounds by aquatic plants and animals, and continuous daily intake of mercury-contaminated fish in large quantities. Minamata Disease patients have neurological symptoms that include paresthesia, visual field [Pg.467]

The first recorded case of Minamata Disease was that of a child in 1953. In 1954, a total of 12 victims were documented 7 adults and 5 children. In 1954, total mercury concentrations in brain from Minamata Disease victims ranged between 0.35 and 5.3 mg/kg FW. In 1955, the total number dead was 15, and in 1956 it had risen to 50 22 adults, 21 children, and 7 fetal cases. Death rate of victims was 36.9%, being higher in summer and lower in winter, and was correlated with fish landings. [Pg.467]

Concentrations of mercury in blood, urine, and especially hair are generally recognized as the best indicators of methylmercury exposure. Concentrations of total mercury in the hair of persons with known occupational exposure to mercury and with a low consumption of fish are usually less than 5.0mg/kg FW. However, persons in Sweden and Finland with high consumption of mercury-contaminated fish and without symptoms of mercury intoxication often contain hair mercury levels 30.0 mg/kg, and in one case 180.0 mg/kg. [Pg.468]

At the end of 1981, mortality analysis of 439 victims among 1483 patients with Minamata Disease in Kumamoto Prefecture showed that mortality rate for all causes of death was significantly higher in both sexes when compared to [Pg.468]

A 71-year-old male severely afflicted with Minamata Disease in 1956 died in 1982. On autopsy, methylmercury concentration inhrain was within the normal range however, the total mercury remained high in the brain and mercury was clearly demonstrated in macrophages over wide areas of the brain and in neurons of specific brain areas. The half-time persistence of methylmercury in brain was estimated at 240-245 days. [Pg.468]

Because of the occurrence of Minamata disease, the importance of pollution problems came to be widely recognized in Japan, and in the whole world, and measures for pollution conhol were taken. New techniques for trace analysis were energetically developed. [Pg.800]


Harada, M., Minamata disease Methylmercury poisoning in Japan caused by environmental pollution,... [Pg.1330]

Humans, Homo sapiens, dying of Minamata disease, 1957-89, autopsy results Brain 0.1-21.3 FW 9... [Pg.358]

Davies, F.C.W. 1991. Minamata disease a 1989 update on the mercury poisoning epidemic in Japan. Environ. Geochem. Health 13 35-38. [Pg.428]

Fujiki, M. 1963. Studies on the course that the causative agent of Minamata disease was formed, especially on the accumulation of the mercury compound in the fish and shellfish of Minamata Bay. Jour. Kumamoto Med. Soc. 37 494-521. [Pg.429]

Fujiki, N. 1980. The pollution of Minamata Bay by mercury and Minamata disease. Pages 493-500 in R.A. Baker (ed.). Contaminants and Sediments, Vol. 2. Ann Arbor Science Publ., Ann Arbor, MI. [Pg.429]

Irukayama, K. 1967. The pollution of Minamata Bay and Minamata disease. Pages 153-180 in J.P. Maroto and F. Josa (eds.). Advances in Water Pollution Research, Vol. 3. Proc. Third Int. Conf., Munich. [Pg.432]

Most cases of mercury poisoning led to handicap, chronic disease, or death. The most frequent symptoms include numbness of limbs, lips and tongue, speech abnormalities, limb function disorders, visual acuity disorders, deafness, and muscular atrophy. Insomnia, hyperactivity, and coma have also been reported. Methylmercury penetrates the blood-brain barrier and causes central nervous system injuries. Mercury also has a teratogenic effect, leading to congenital abnormalities or congenital Minamata disease. [Pg.242]

An epidemic of intoxication from ingestion of fish contaminated with methyl mercury occurred in the Minamata district in Japan, and, as a result, methyl mercury intoxication is often referred to as Minamata disease." Infants born to mothers with exposure to large amounts of methyl mercury had microen-cephaly, mental retardation, and cerebral palsy with convulsions. In an incidence in Iraq, ingestion of wheat products contaminated with methyl mercury fungicide by pregnant women caused similar symptoms of neurological damage and mental retardation. The fetus is... [Pg.439]

Mercury. In Japan in the 1950s and 1060s, wastes from a chemical and plastics plant containing mercury were discharged into Minamata Bay. The mercury was converted to the readily absorbed methylmercury by bacteria in the aquatic sediments. Consumption of fish and shellfish by the local population resulted in numerous cases of mercury poisoning, or Minamata disease. By 1970, at least... [Pg.41]

A common example of neurobehavioral teratogenic effects caused by polluted food is the Minamata disease. In a number of areas in Japan methylmercury from industrial waste accumulated in fish which was one of the main sources of the food of the local population. Another case of poisoning with methylmercury occurred in Iraq, where people consumed grain seed treated with a methylmercury fungicide. Women with only minimal poisoning symptoms gave birth to children that showed many neurological and behavioral abnormalities, such as mental retardation, coordination deficits and seizures (refs. 12, 13). [Pg.272]

Harada M (1978) Congenital Minamata disease intrauterine methylmercury poisoning. Teratology, 18(2) 285-288. [Pg.267]

Between 1956 and 1960 severe accidents related to Me-Hg, the health consequences of which were afterwards grouped under the name of Minamata disease, occurred in Japan following long-term consumption of contaminated fish and fish products. A large amount of Hg-polluted effluents (from 200 to 600 tons) reached Minamata Bay, Japan, where they accumulated not only in the bottom sediment, but also in fish and shellfish. The consumption of such foods resulted in a mass outbreak of Me-Hg poisoning [9]. [Pg.708]

A case of Minamata disease caused by the methyl mercury discharged by a chemical factory in Japan provides us with bitter lessons. Inappropriate treatments of toxic chemical substances for the duration of 1932 to 1968 resulted in creating and torturing more than 14,000 victims with a neurological syndrome by severe mercury poisoning. It has been fifty years since the disease was official discovered in 1956. The case is not yet fully solved and lawsuits and claims for compensations continue even to this day. [Pg.87]


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Diseases Minamata disease

Diseases Minamata disease

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