Lung cells exposure

Odum J, Foster JR, Green T. 1992. A mechanism for the development of Clara cell lesions in the mouse lung after exposure to trichloroethylene. Chem Biol Interact 83 135-153. 

Reubel, B., C. Atzmuller, F. Steinhausler, and W. Huber, Biophysical Effects on Human Lung Cells Due to Radon Exposure, this volume (1987). 

Biophysical Effects of Radon Exposure on Human Lung Cells... 

Aufderheide et al. [84] performed experiments with human lung cells that were directly exposed to diesel exhaust. In contrast to other exposure concepts for complex mixtures, this experimental setup facilitates a direct and reproducible contact between the cell monolayer and the test atmosphere. This could be achieved by following improvements (i) a strict separation of the medium... 

NT036 Cavarra, E., M. Lucattelli, F. Gambelli, et al. Human SLPI inactivation after cigarette smoke exposure in a new in vivo model of pulmonary oxidative stress. Am J Physiol Lung Cell Mol Physiol 2001 281(2) L412-L417. 

NT141 Gairola, C. G. Free lung cell response of mice and rats to mainstream cigarette smoke exposure. Toxicol Appl Pharmacol 1986 84(3) 567-575. 

Chromosomal aberrations were not induced in rat liver cells, Chinese hamster lung cells or Chinese hamster ovary cells by in-vitro exposure to triethanolamine. It did not induce cell transformation in Syrian hamster embryo cells. 

Rylander R. 1973. Toxicity of cigarette smoke components. Free lung cell response in acute exposures. Am Rev Resp Dis 108 1279-1282. 

THC does not appear to be carcinogenic, but there is plenty of evidence that the tar derived from cannabis smoke is. Bacteria exposed to cannabis tar develop mutations in the standard Ames test for carcinogenicity (53), and hamster lung cells in tissue culture develop accelerated malignant transformations within 3-6 months of exposure to tobacco or cannabis smoke (54). 

Other Cell-Mediated Mechanisms. In addition to the release of active oxygen species, alveolar macrophages and other cells, including pleural mesothelial and lung cells, release a number of cellular factors in response to asbestos exposure. These factors are mediators of a number of cellular reactions including inflammation, macrophage recruitment and cell proliferation (for reviews, see Driscoll et al. 

Brody AR, Liu J-Y, Brass D, et al. 1997. Analyzing the genes and peptide growth factors expressed in lung cells in vivo consequent to asbestos exposure in vitro. Environ Health Perspect Suppl 105 1165-1171. 

Chang L-Y, Overby LH, Brody AR, et al. 1988. Progressive lung cell reactions and extracellular matrix production after a brief exposure to asbestos. Am J Pathol 131 156-170. 

Aghanouri R, Ghanei M, Aslani J et al. (2004). Fi-brogenic cytokine levels in bronchoalveolar lavage aspirates 15 years after exposure to sulfur mustard. Am J Physiol Lung Cell Mol Physiol, 287, LI 160-L1164. 

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