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Iron deficiency manganese toxicity

Cobalt, copper, molybdenum, iodine, iron, manganese, nickel, selenium, and zinc are sometimes provided to mminants. Mineral deficiency or toxicity in sheep, especially copper and selenium, is a common example of dietary mineral imbalance (21). Other elements may be required for optimal mminant performance (22). ExceUent reviews of trace elements are available (5,22). [Pg.156]

Chandra SV, Tandon SK. 1973. Enhanced manganese toxicity in iron-deficient rats. Environ Physiol Biochem 3 230-235. [Pg.443]

One of the important benefits of chelates, other than supplying metals, is realized as a result of their improvement of the micronutrient balance of plants. For example. Holmes and Brown (1955), in studies with calcareous soils, observed that certain effective chelates increased the concentration of iron in soybeans, and at the same time decreased the concentration of manganese and copper. This suggests that the cause of iron-deficiency chlorosis in this case may have been improper microelement balance in these calcareous soils. These workers also observed that when chelate was applied to several soils, it alleviated iron chlorosis completely in some soils, partly in others, and caused toxicity in yet others. From the work of others we know that under other conditions chelates may also eliminate or greatly decrease toxic effects where heavy metals are present in excessive amounts. Numerous soil factors influence these varying responses. [Pg.308]

Chandra, S. V. and Tandon, S. K. (1973) Inhanced manganese toxicity in iron deficient animals. Environ. Physiol. Biochem. 3, 230-235. [Pg.191]

In domestic animals, the major reported lesion associated with chronic manganese toxicity is iron deficiency, resulting from an inhibitory effect of manganese on iron absorption. Additional signs of manganese toxicity in domestic animals include depressed growth, depressed appetite, and altered brain function. [Pg.261]

Soil pH is easily tested for and determines the availability of nutrients and the success of white clover. Very acid soils (below pH 5.0) will cause a deficiency of the trace elements iron, boron, copper and molybdenum and conversely will cause injury to plant growth by increasing the availability of aluminium and manganese to toxic levels. Over-liming, on the other hand, which can raise the pH above 6.5, will reduce the availability of certain essential elements such as phosphorus, manganese and boron. [Pg.21]

The importance of dose is well illustrated by metals that are essential in the diet but are toxic at higher doses. Thus iron, copper, magnesium, cobalt, manganese, and zinc can be present in the diet at too low a level (deficiency), at an appropriate level (maintenance), or at too high a level (toxic). The question of dose-response relationships is fundamental to toxicology (see Section 1.2). [Pg.4]

Transferrin is mainly synthesized in the hepatocytes. There are about 20 known variants. Iron is transported by transferrin (approx. 30% of transferrin is saturated with iron). With the help of a membrane receptor, the iron-transferrin complex is taken up and released in the liver cell, where it is immediately bound (because of its toxicity) to ferritin. The liver cells take up iron predominantly from transferrin, to a lesser degree also from haptoglobin, haemopexin, lactoferrin and circulating ferrin. Transferrin, which is mainly formed in the hepatocytes, may also bind and transport, in decreasing order, chromium, copper, manganese, cobalt, cadmium, zinc and nickel. The half-life of transferrin is 1 - 2 hours, which is very short in view of its total blood concentration of 3-4 mg. Approximately 0.4 g ferritin iron is stored in the liver. In the case of transferrin deficiency, its bacteriostatic and fungistatic effects are also reduced. Transferrin without iron saturation is known as apo-transferrin. (31, 66, 67)... [Pg.50]

The micronutrients of major interest to soil chemistry because of plant deficiencies are boron, manganese, iron, cobalt, copper, zinc, and molybdenum. Other ions— chromium, nickel, cadmium, mercury, and lead—behave similarly in soils but the problems are usually plant toxicity. The availability of most of the micronutrient and toxic ions increases with increasing soil acidity. Those present as anions—-molybdenum, chromium, and boron—differ in that their availability generally decreases with increasing acidity. [Pg.273]

In contrast to its essentiality, every element of the periodic system may be toxic it is only a question of the intake quantities and the element specification. An intoxication can induce interactions with essential elements and induce deficiency symptoms well-known examples are the interactions of nickel with zinc, magnesium, and manganese (Anke etal. 19971), or cadmium with copper, zinc, and iron (Anke et al 1970). [Pg.343]

Moderate excesses of iron, which do not have direct toxic effects, may produce nutritional deficiencies by interfering with the absorption of copper, manganese, and zinc, and by destroying vitamins C and E. [Pg.728]


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See also in sourсe #XX -- [ Pg.261 ]




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