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Carotid artery dissection

Findlay JM, Ashforth R, Dean N. Malignant carotid artery dissection. Can J Neurol Sci 2002 29 378-385. [Pg.135]

Lucas C, Moulin T, Deplanque D, Tatu L, Chavot D. Stroke patterns of internal carotid artery dissection in 40 patients. Stroke 1998 29 2646-2648. [Pg.160]

Engelter S, Lyrer P, Kirsch E, Steck AJ. Long-term follow-up after extracranial internal carotid artery dissection. Eur Neurol 2000 44 199-204. [Pg.160]

Lyrer P, Engelter S. Antithrombotic drugs for carotid artery dissection. Cochrane... [Pg.161]

Malek AM, Higashida RT, Phatouros CC, Lempert TE, Meyers PM, Smith WS, Dowd CP, Halbach VV. Endovascular management of extracranial carotid artery dissection achieved using stent angioplasty. AJNR Am J Neuroradiol 2000 21 ... [Pg.161]

Guillon B, Levy C, Bousser MG (1998) Internal carotid artery dissection an update. J Neurol Sci 153 146-158 Hacke W, Brott T, Caplan L et al. (1999) Thrombolysis in acute ischemic stroke controlled trials and clinical experience. Neurology 53 S3-14... [Pg.100]

Gass A, Gaa J, Schwartz A (1997) Cerebral infarction due to internal carotid artery dissection. J Neurol Neurosurg Psychiatry 63 420... [Pg.236]

The incidence of diagnosed internal carotid artery dissection is approximately 1-4 per 100000 per year. Vertebral dissection is a little less common. The actual incidence of dissections is likely to be considerably higher, but the diagnosis is often missed, particularly in older patients. Usually only one artery is involved but in about 10%, multiple arteries... [Pg.67]

Ultrasound findings in carotid artery dissection analysis of 43 patients. Neurology 45 691-698... [Pg.89]

Headache is not uncommon around the time of stroke onset. It is more often severe in primary intracerebral hemorrhage than ischemic stroke, and more often severe with posterior than anterior circulation strokes. If the headache is localized at all, it tends to be over the site of the lesion. Headache is more common in cortical and posterior circulation than lacunar infarcts (Kumral et al. 1995). Severe unilateral neck, orbital or scalp pain suggests internal carotid artery dissection, particularly if there is an ipsilateral Horner s syndrome. Severe occipital headache can occur with vertebral artery dissection. Headache is also a particular feature of venous infarcts. Unusual headache in the days before stroke would suggest giant cell arteritis or perhaps a mass lesion rather than a stroke. [Pg.121]

Atypical antipsychotic drugs Chronic subdural hematoma (Ch. 7) Vertebral/carotid artery dissection (Ch. 6) Fat embolism (Ch. 6) nternal carotid artery stenosis (distal) Dural arteriovenous fistula (Ch. 7)... [Pg.125]

Fig. 12.2. Digitally subtracted arteria angiogram showing a distal interna carotid artery dissection (tapering lumen lower arrow) and a complicating aneurysm (upper arrow). Fig. 12.2. Digitally subtracted arteria angiogram showing a distal interna carotid artery dissection (tapering lumen lower arrow) and a complicating aneurysm (upper arrow).
German neurologists have reported 10 cases of ischemic stroke due to either vertebral arterial dissection (n = 8) or internal carotid artery dissection (n — 2) after chiropractic spinal manipulation (179). There were no identifiable predisposing factors. In three cases the dissections were bilateral. The onset of sjmptoms was immediate (n = 5) or delayed by up to 2 days. Neurological deficits developed during up to 3 weeks. In five patients the eventual clinical outcome was good while marked deficits persisted in three patients. One patient continued to suffer from a locked-in sjmdrome and another was in a persistent vegetative state. [Pg.894]

Ozdoba, C., M. Sturzenegger, and G. Schroth, Internal carotid artery dissection MR imaging features and clinical-radiologic correlation Radiology, 1996. 199(1) p. 191-8. [Pg.143]

Fig. 8.12 Choice of different arterial input functions. The patient is 39-year-old female with a history of right internal carotid artery dissection and left-sided weakness, who was imaged at 8 h after symptom onset. When the left middle cerebral artery (MCA) stem is chosen as the arterial input function (AIF), there is longer delay between the AIF and the tissue sam-... Fig. 8.12 Choice of different arterial input functions. The patient is 39-year-old female with a history of right internal carotid artery dissection and left-sided weakness, who was imaged at 8 h after symptom onset. When the left middle cerebral artery (MCA) stem is chosen as the arterial input function (AIF), there is longer delay between the AIF and the tissue sam-...
Matsuura JH, Rosenthal D, Jerius H, Clark MD, Owens DS (1997) Traumatic carotid artery dissection and pseudoaneurysm treated with endovascular coils and stent. J Endo-vasc Surg 4 339-343... [Pg.291]

Dissection of the internal carotid and vertebral arteries is a common cause of stroke, particularly in young patients. Although many occur due to trauma, it is estimated that over half occur spontaneously. The mechanism of stroke following arterial dissection is either by artery-to-artery embolism, by thrombosis in situ, or by dissection-induced lumenal stenosis with secondary cerebral hypoperfusion and low-flow watershed infarction. Occasionally, dissection may lead to the formation of a pseudoaneurysm as a source of thrombus formation. Vertebrobasilar dissections that extend intracranially have a higher risk of rupture leading to subarachnoid hemorrhage (SAH). ° ... [Pg.152]

Jacobs A, Lanfermann H, Neveling M, Szelies B, Schroder R, Heiss WD. MRI- and MRA-guided therapy of carotid and vertebral artery dissections. J Neurol Sci 1997 147 27-34. [Pg.160]

Schievink W. The treatment of spontaneous carotid and vertebral artery dissections. Curr Opin Cardiol 2000 15 316-321. [Pg.160]

Mokri B, Silbert PL, Schievink WI et al (1996) Cranial nerve palsy in spontaneous dissection of the extracranial internal carotid artery. Neurology 46 356-359 Molina CA, Montaner J, Abilleira S et al (2001) Timing of Spontaneous Recanalization and Risk of Hemorrhagic Transformation in Acute Cardioembolic Stroke. Stroke 32 1079-1084 Molina CA, Alvarez-Sabin J, Montaner J et al (2002) Thrombolysis-related hemorrhagic infarction a marker of early reperfusion, reduced infarct size, and improved outcome in patients with proximal middle cerebral artery occlusion. Stroke 33 1551-1556... [Pg.16]

Anomalies of the ICA are infrequent, and aplasia of the ICA is a rarity (Van de Perre et al. 2004). In hypoplasia, the vessel tapers off behind a normal proximal segment and can sometimes be followed up to the circle of Willis. Occasionally differential diagnosis is difficult against an arterial dissection or a very high grade stenosis with a collapsing distal lumen (pseudoocclusion). In cases of doubt, the CT shows the hypoplastic osseous carotid canal (Chen et al. 1998). An aberrant lateral course of ICA... [Pg.85]

MCA infarcts are mainly caused by cardioembolism, internal carotid artery (ICA) thrombosis, dissection or embolism and rarely (in Caucasians) by intrinsic MCA disease. MCA atherothrombotic territory infarctions related to intrinsic MCA disease often cause concomitant small cortical (territorial or borderzone) and subcortical infarcts (Min et al. 2000). [Pg.210]

The presence of cranial neuropathy may result in a misdiagnosis of brainstem stroke. Cranial nerve palsies may result from local pressure from the false internal carotid artery lumen, thromboembolism or hemodynamic compromise to the blood supply of the nerve. Cranial nerve III receives its blood supply from the ophthalmic artery, branches of the internal carotid or the posterior cerebral artery and, consequently, may rarely become ischemic after carotid dissection. [Pg.67]

Rubba P, Mercuri M, Faccenda F et al. (1994). Premature carotid atherosclerosis does it occur in both familial hypercholesterolemia and homocystinuria Ultrasound assessment of arterial intima-media thickness and blood flow velocity. Stroke 25 943-950 Rubinstein SM, Peerdeman SM, van Tulder MW et al. (2005). A systematic review of the risk factors for cervical artery dissection. Stroke 36 1575-1580... [Pg.88]

Neurological examination is primarily to localize the brain lesion but there may also be clues as to the cause of the stroke a Horner s syndrome ipsilateral to a carotid distribution infarct suggests dissection of the internal carotid artery or sometimes acute atherothrom-botic carotid occlusion. Lower cranial nerve lesions ipsilateral to a hemispheric cerebral infarct can also occur in carotid dissection. [Pg.127]

Tenderness of the branches of the external carotid artery (occipital, facial, superficial temporal) points towards giant cell arteritis. Tenderness of the common carotid artery in the neck can occur in acute carotid occlusion but is more Ukely to be a sign of dissection, or arteritis. Absence of several neck and arm pulses in a young person occurs in Takayasu s arteritis (Ch. 6). Delayed or absent leg pulses suggest coarctation of the aorta or, much more commonly, peripheral vascular disease. Other causes of widespread disease of the aortic arch are atheroma, giant cell arteritis, syphihs, subintimal fibrosis, arterial dissection and trauma. [Pg.127]


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See also in sourсe #XX -- [ Pg.564 ]




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