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Blood pressure diabetics

Nervilia purpurea (Hayata) Schltr. Yi Dian Hong (whole plant) Cyclonerviol, cyclomonerviol, stigmasterol, dihydrocyclonervilol, ergosterol, epibrassicasterol, nervisterol, cyclonervilol.58 As a protective medicine post partum, treat throat infection, pneumonia, high blood pressure, diabetes. [Pg.116]

Very early worsening after stroke is more likely to be caused by neurological factors than systemic ones. The mechanism of worsening may not be clear in an individual patient and is likely to be the result of complex interactions between hemodynamic and other physiological factors. High or low blood pressure, diabetes, coronary heart disease, early CT signs of infarction and middle cerebral artery occlusion have all been associated with increased risk of early deterioration after stroke. [Pg.210]

Men and women are getting fatter and fatter, and we re paying the price for those supersize meals with an increased risk of developing high blood pressure, diabetes, heart attacks, and strokes. The scariest statistic I ve read is that today s youngsters, who are heavier and more sedentary than ever, will be the first generation with a life expectancy shorter than that of their parents. [Pg.57]

High-blood pressure, diabetes, arteriosclerosis Improves oxygen consumption for strokes, etc. [Pg.420]

Advanced age, heart rate, systolic blood pressure, diabetes mellitus, recurrent or persistent pain. The prognosis is worse in diabetics and elderly patients, especially in presence of renal failure, sinus tachycardia and evident haemodynamic impairment (hypotension, pulmonary oedema, etc.) (grade 3-4 of Killip classification) (Wiviott et al, 2006). [Pg.260]

Although data are limited, the minor to modest possible ineieases in frov-atriptan, naratriptan, sumatriptan and zoimitriptan pharmacokinetics described are not likely to produce clinically relevant adverse effects. Almotriptan, rizatriptan and sumatriptan do not appear to have any clinically important effect on levels of contraceptive steroids. The significance of the single case report of ischaemic colitis associated with concurrent use of naratriptan and a combined oral contraceptive is unclear. Note that ischaemic colitis has, rarely, been reported with naratriptan itself The manufacturers have found no cases of ischaemic colitis in approximately 450 women on oral contraceptives and taking naratriptan for prophylaxis for 5 to 6 days. However, caution may be needed with concurrent use in those patients with risk factors for ischaemic colitis, such as those with a history of abdominal surgery, low blood pressure, diabetes, cardiovascular disease or stroke. [Pg.1005]

Do any heart diseases, high blood pressure, diabetes, high cholesterol, or high triglycerides run in your family Explain. [Pg.1210]

Figure 2 O (WG) and (refined) adjusted HRR for age and total energy intake. (WG) and (refined) adjusted for age, energy intake, marital status, education, high blood pressure, diabetes, heart disease, cancer, BMI, WHR, physical activity, smoking, alcohol intake, use of vitamin supplements, HRT, total fat, saturated fat, intake of fruits and vegetables, intake of meat and intake of fish and seafood. Figure 2 O (WG) and (refined) adjusted HRR for age and total energy intake. (WG) and (refined) adjusted for age, energy intake, marital status, education, high blood pressure, diabetes, heart disease, cancer, BMI, WHR, physical activity, smoking, alcohol intake, use of vitamin supplements, HRT, total fat, saturated fat, intake of fruits and vegetables, intake of meat and intake of fish and seafood.
Engineered commensal bacteria can also be nsed in the treatment of metabolic diseases. Examples of these metabolic diseases are obesity, high blood pressure, diabetes and hyperlipidemia that arises from unhealthy lifestyles and diet, and that needs to be treated nsing pills and injections in specified doses at particular time-points (Heng et al., 2015), thns reducing a patient s qnaUty of life. [Pg.473]

In the treatment of hypertension, ACE inhibitors are as effective as diuretics, (3-adrenoceptor antagonists, or calcium channel blockers in lowering blood pressure. However, increased survival rates have only been demonstrated for diuretics and (3-adrenoceptor antagonists. ACE inhibitors are approved for monotherapy as well as for combinational regimes. ACE inhibitors are the dtugs of choice for the treatment of hypertension with renal diseases, particularly diabetic nephropathy, because they prevent the progression of renal failure and improve proteinuria more efficiently than the other diugs. [Pg.10]

Criteria for initiation of drug treatment now take into consideration total cardiovascular risk rather than blood pressure alone, such that treatment is now recommended for persons whose blood pressure is in the normal range but still bear a heavy burden of cardiovascular risk factors. Thus, the role of simultaneous reduction of multiple cardiovascular risk factors in improving prognosis in hypertensive patients is stressed. In addition, more aggressive blood pressure goals are recommended for hypertensive patients with comorbid conditions such as diabetes mellitus or renal insufficiency. [Pg.142]

Historically the only melanocortin peptide to be used clinically is the parent hormone from which all these peptides are derived from namely ACTH (see above). It has also been used in the treatment infantile spasms for epilepsy, where it is administered as an intramuscular injection only over a 2-12 weeks period. Obvious side effects include weight gain, puffy face, high blood pressure and an increased risk of infection and should never be administered to patients with diabetics, renal or heart failure. ACTH is also used as a stimulation test to measure adrenal cortex activity, i.e. production of cortisol and is used to ascertain whether someone has Addison s disease. [Pg.753]

Goal blood pressure tor patients with diabetes is < 130/85 mm Hg Goal blood pressure tor patients wilh renal disease is < 130/85 mm Hg or < 125/75 mm Hg in palients wi1h proteinuria > 1 gram/24 hours... [Pg.395]

If the patient has recently received a diagnosis of diabetes mellitus and has not received an oral antidiabetic drug, or if the patient is known to have diabetes and has been taking one of these drags, the nurse should include weight, blood pressure, pulse, and... [Pg.504]

Before administering die first dose of vasopressin for die management of diabetes insipidus, die nurse takes die patient s blood pressure, pulse, and respiratory rate. The nurse weighs the patient to obtain a baseline weight for future comparison. Serum electrolyte levels and odier laboratory tests may be ordered by die primary health care provider. [Pg.519]

Essential hypertension, whose prevalence is increased nearly two-fold in the diabetic population, may be another source of free-radical activity. The vascular lesions of hypertension can be produced by free-radical reactions (Selwign, 1983). In the recent Kuopio Ischaemic Heart Risk Factor Study in Finnish men, a marked elevation of blood pressure was associated with low levels of both plasma ascorbate and serum selenium (Salonen etal., 1988). A few studies report a hypotensive effect of supplementary ascorbate in patients with hypertension, but the actual changes in both systolic and diastolic pressure after ascorbate were not statistically significant in comparison with placebo (Trout, 1991). [Pg.193]

Routine antioxidant vitamin supplementation, e.g. with vitamins C and/or E, of the diabetic diet should be considered. Vitamin C depletion is present in all diabetics irrespective of the presence of vascular disease. A recent study demonstrated no significant difference between the dietary intake of vitamin C (the main determinant of plasma ascorbate) in patients with diabetes and age-matched controls, confirming the view that ascorbate depletion is secondary to the diabetic process and su esting that diabetic patients require additional intakes of the vitamin to maintain optimal levels (Sinclair et /., 1994). Antioxidant supplementation may have additive beneficial effects on a wide variety of processes involved in diabetic vascular damage including blood pressure, immune function, inflammatory reactions. [Pg.194]

National High Blood Pressure Education Program noninsulin-dependent diabetes mellitus non-steroidal anti-inflammatory drug peripheral resistance... [Pg.31]

Factors that predispose an individual to IHD are listed in Table 4—2. Hypertension, diabetes, dyslipidemia, and cigarette smoking are associated with endothelial dysfunction and potentiate atherosclerosis of the coronary arteries. The risk for IHD increases two-fold for every 20 mm Hg increment in systolic blood pressure and up to eight-fold in the presence of diabetes.5,6 Physical inactivity and obesity independently increase the risk for IHD, in addition to predisposing individuals to other cardiovascular risk factors (e.g., hypertension, dyslipidemia, and diabetes). [Pg.65]

Like dyslipidemia, hypertension is a major, modifiable risk factor for the development of IHD and related complications. Unfortunately, awareness, treatment, and control of blood pressure are not nearly enough.30 Aggressive identification and control of hypertension is warranted in patients with IHD to minimize the risk of major adverse cardiac events. Goal blood pressure in patients with IHD is less than 140/90 mm Hg or less than 130/80 mm Hg in patients with diabetes. Because of their cardioprotective benefits, 3-blockers and ACE inhibitors (or ARBs in ACE-inhibitor-intolerant patients), either alone or in combination, are appropriate for most patients with both hypertension and IHD. [Pg.75]

Monitor for adverse effects of 3-blockers—heart rate, blood pressure, fatigue, masking of symptoms of hypoglycemia and/or glucose intolerance (in patients with diabetes), wheezing or shortness of breath (in patients with asthma or chronic obstructive pulmonary disease), etc. [Pg.125]

A 73-year-old man with a history of diabetes mellitus, chronic kidney disease, gout, osteoarthritis, and hypertension is hospitalized with possible urosepsis. He recently completed a 10-day course of antibiotics and was ready for discharge when his morning labs showed an increase in BUN and serum creatinine concentration. Upon examination, he was found to have 2+ pitting edema, weight gain, nausea, elevated blood pressure, and rales on chest auscultation. [Pg.363]


See other pages where Blood pressure diabetics is mentioned: [Pg.755]    [Pg.49]    [Pg.122]    [Pg.755]    [Pg.782]    [Pg.257]    [Pg.148]    [Pg.413]    [Pg.904]    [Pg.1328]    [Pg.126]    [Pg.755]    [Pg.49]    [Pg.122]    [Pg.755]    [Pg.782]    [Pg.257]    [Pg.148]    [Pg.413]    [Pg.904]    [Pg.1328]    [Pg.126]    [Pg.538]    [Pg.303]    [Pg.132]    [Pg.213]    [Pg.275]    [Pg.857]    [Pg.579]    [Pg.769]    [Pg.47]    [Pg.349]    [Pg.10]    [Pg.10]    [Pg.17]    [Pg.21]    [Pg.22]    [Pg.24]    [Pg.368]   
See also in sourсe #XX -- [ Pg.47 ]




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