Colic, abdominal

As is true of the other effects, these tend to be rather vague. They include conditions such as loss of appetite, indigestion, constipation, and griping, the latter being a form of abdominal colic which can be extremely severe and even lead to hospitalization. 

A 48-year-old man developed abdominal colic, muscle pain, and fetigue. Following a 3-week hospitalization, acute intermittent porphyria was initially diagnosed based on a high level of urinary 5-aminolevulinic acid. Subsequent analysis of the patient s circulating red blood ceUs revealed that 70% contained elevated levels of zinc protoporphyrin, and the diagnosis was corrected. The correct diagnosis is most likely to be... 

Erythromycin estoiate Hepatic dysfunction with or without jaundice has occurred, chiefly in adults, in association with erythromycin estolate administration. It may be accompanied by malaise, nausea, vomiting, abdominal colic, and fever. In some instances, severe abdominal pain may stimulate an abdominal surgical emergency. 

It is used for abdominal colic and in aerosol form it is used in bronchial asthma. 

Gastrointestinal Effects. In a case report, two female gardeners, dermally exposed to spilled diazinon of unknown purity, developed signs of acute pancreatitis which included abdominal colic, diarrhea, nausea, vomiting, and epigastric pain, as well as elevated serum amylase and urinary diastase levels (Lee 1989). 

Gastrointestinal Abdominal colic, abdominal discomfort, diarrhea, proctitis (with suppository use), atony of colon... 

Triclabendazole is a benzimidazole derivative primarily used in veterinary medicine but has also been used experimentally in man. Chills, fever, leukopenia, and upper abdominal colic have been described (SEDA-14, 263). 

Constipation, abdominal colic, diarrhea, dysentry, hoarseness, hiccough Tonic, stomachic... 

A single case report described a self-administered ammonium solution enema that resulted in anal pain, diffuse abdominal colic, and tenesmus (da Fonseca et al. 1998). Sigmoidoscopy showed diffuse erythematous friable mucosa with large ulcerations covered by yellowish exudate. Six days later, the ulcers had receded, but the colon was still erythematous. Three months postexposure, biopsies showed chronic inflammation and fibrosis of the rectum and sigmoid colon, but no stenosis. 

Mushrooms are a rich source of toxins mushroom poisoning has increased as the result of the popularity of hunting wild mushrooms. High concentrations of muscarine are present in various species o/Inocybe and Clitocybe. The symptoms of muscarine intoxication (salivation, lacrima-tion, nausea, vomiting, headache, visual disturbances, abdominal colic, diarrhea, bronchospasm, bradycardia, hypotension, shock) develop within 30-60 minutes of ingestion. Treatment with atropine (1-2 mg intramuscularly every 30 minutes) effectively blocks these effects. 

Items 1-2 A small child is brought to a hospital emergency room suffering from severe gastrointestinal distress and abdominal colic. 

Overdoses of Chinese rhubarb can cause a toxic reaction, especially if the fresh herb is used. Symptoms include diarrhea, nausea, vomiting, dizziness, abdominal colic, and jaundice. Long-term use may cause severe damage to the large intestine, cirrhosis of the liver, and potassium loss (Bensky et al. 2004). 

Gastrointestinal. Lead may also affect the gastrointestinal system producing abdominal colic or diffuse abdominal pain, constipation, obstipation, diarrhea, anorexia, nausea and vomiting. Lead colic rarely develops at blood lead levels below 80 [ig/dl. 

Greco-Roman era to 600 CE and later Abdominal colic, motor peripheral paralysis with foot and wrist drop, seizures, death Chronic lead poisoning with chronic encephalopathic and peripheral neuropathic features Pb chronic Gl effects Paul of Aegina Seventh century Major (1945) Nriagu (1983a,b)... 

Battery production workers PbB range 55-81 ig/dl, battery workers 7-33 ig/dl, control workers Gl symptoms "abdominal colic," constipation Increased prevalence of Gl symptoms in the battery workers Awad El Karim et al. (1986)... 

This disease, recently studied by Rimington, " appears also to have a familial tendency. The disease is characterized by intermittent abdominal colic and photosensitivity, but no nervous symptoms. During remission there is excreted in the feces some 40-50 mg. of protoporphyrin per day, or about 20% of the total normal production of protoporphyrin. During attack, fecal excretion stops, but there is considerable excretion in the urine of series I and III uro- and coproporphyrins. 

Cardiovascular A 17-year-old man with a 3-year history of ulcer symptoms, diarrhea, and bouts of abdominal colic developed severe hypotension (50/20 mmHg) after receiving intravenous metoclopramide for acute vomiting with diarrhea [9 ]. He then developed pneumonia, rhabdomyolysis, renal tubular necrosis, and disseminated intravascular coagulation. A diagnosis of gastrinoma was made. During hormonal assessment, he received a second dose of... 

Several reasons for the occurrence of this symptom have been discussed. A rapid increase in size of liver and spleen precipitated by fatty meals or otherwise may be one possible cause of the abdominal colics resulting in stretching of the capsule of these organs. In Holt s case, 8 gm per 100 ml appeared to be a critical level for serum lipids with values above this level abdominal crises occurred frequently. An alternative explanation for these acute episodes would be the occurrence of recurrent pancreatitis as has been discussed repeatedly in the literature. This aspect will be considered below. A third possible cause in some cases may be impaired oxygen supply to the intestines as has been postulated by Kuo et al. (1959) and others for the myocardium, resulting in a kind of intermittent claudication of the bowel. 

A similarly interesting case is that of a 68 year old male patient who was first seen in 1958 with symptoms of marked myocardial ischemia and electrocardiographic evidence of an old inferior myocardial infarction. Insulin dependent diabetes melli-tus had been present since 1932. In spite of diabetic control with diet and insulin in this subject, lipemia (approximately 2 gm neutral fat per 100 ml plasma) persisted and abdominal colics recurred. With heparin and later Depot-Thrombocid his neutral lipids ranged between 300—500 mg per 100 ml and clinical symptoms (angina pectoris and episodes of abdominal pain) improved considerably. An additional feature in this patient was gout with uric acid levels above 9 mg per 100 ml and attacks of acute gouty arthritis on several occasions between 1958 and 1959. 

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