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Myocardial infarction inferior

It is important to obtain a baseline EKG and cardiac enzymes to evaluate the possibility of an acute myocardial infarction. The short-term (2-4 weeks) stroke risk after acute myocardial infarction (AMI) is 2.5%. Stroke is usually an early (within 14 days) complication of AMI and is more common in anterior wall (4—12%) than in inferior wall infarction (1%). Approximately 40% of patients with an anterior wall myocardial infarction develop left ventricular thrombus. [Pg.204]

Obtain right precordial leads in any patient with an inferior wall myocardial infarction... [Pg.34]

Effects after myocardial infarction In patients with evidence of LV dysfunction early after myocardial infarction, the Valsartan in Acute Myocardial Infarction Trial (VALIANT) (48) demonstrated that valsartan had a benefit that was not inferior to that of ACE inhibitors without an advantage in terms of tolerability. However, the addition of an angiotensin II receptor blocker to an ACE inhibitor did not improve the outcomes and resulted in more side effects. [Pg.457]

A 34-year-old man who smoked a pack of cigarettes a day took amfetamine for mild obesity. He developed an acute myocardial infarction 1 week later. Echocardiography showed inferior left ventricular hypokinesia and a left ventricular ejection fraction of 50%. Coronary cineangiography showed normal coronary arteries but confirmed the inferior left ventricular hypokinesia. Blood and urine toxicology were positive only for amfetamine. [Pg.454]

A 62-year-old woman developed chest pain and sinus bradycardia (41/minute). She had third-degree heart block and was given atropine 1 mg intravenously. Three minutes later, her chest pain increased and the electrocardiogram now showed an acute inferior myocardial infarction, confirmed by serum markers. Angioplasty recanalized the right coronary artery. [Pg.375]

A 21-year-old woman had an inferior myocardial infarction, in the absence of cardiovascular risks and with normal coronary arteries on angiography (2). She made a good recovery, but with some persistent posterior-wall akinesia. [Pg.559]

Adult respiratory distress syndrome occurred after the intravenous infusion of dextran 40 in a 30-year-old woman, a smoker with a history of insulin-dependent diabetes mellitus, who had sustained an acute inferior myocardial infarction the dextran was given in... [Pg.1082]

Adverse cardiac effects due to morphine are rare. They comprise inappropriate heart rate responses to hypotension, rather than conduction defects. They are not especially associated with inferior myocardial infarction, as was previously thought (SED-11,142) (14). [Pg.2387]

A 41-year-old man employed in a munitions factory was admitted with crushing chest pain on a Sunday morning. There was evidence of an acute inferior myocardial infarction, and cardiac catheterization showed 80% narrowing of the proximal right coronary artery, which was reduced to 10% by intracoronary administration of glyceryl trinitrate. The electrocardiogram became normal. [Pg.2532]

A 46-year-old man developed a Q-wave inferior and a right ventricular myocardial infarct with postinfarction angina after the third cycle of vincristine -I- doxorubicin for multiple myeloma. The patient had no risk factors for ischemic heart disease, except for a positive smoking history, nor for hyperviscosity (24). [Pg.3634]

Figure 3.17 (A) and (B) ECG-VCG correlation of the T wave and the T loop of subepicardial ischaemia in two patients with myocardial infarction (A) of the inferior wall and (B) of the inferior and lateral walls. Observe that a T loop in both cases shows homogeneous inscription and is directed upwards (see FPa) in the first case and upwards and forward in the second case (see HPa). The QRS loop of (A) rotates only clockwise and of (B) rotates first clockwise and later counter-clockwise. In the first case inferior Ml is isolated and in the second, associated to superoanterior... Figure 3.17 (A) and (B) ECG-VCG correlation of the T wave and the T loop of subepicardial ischaemia in two patients with myocardial infarction (A) of the inferior wall and (B) of the inferior and lateral walls. Observe that a T loop in both cases shows homogeneous inscription and is directed upwards (see FPa) in the first case and upwards and forward in the second case (see HPa). The QRS loop of (A) rotates only clockwise and of (B) rotates first clockwise and later counter-clockwise. In the first case inferior Ml is isolated and in the second, associated to superoanterior...
Figure 4.17 Acute myocardial infarction with ST-segment elevation in II, III and VF and ST-segment depression in V1-V3. This pattern corresponds classically to an infarction involving inferior and posterior walls. Nowadays, this is the pattern of STE-ACS of inferolateral zone evolving to inferolateral infarction due to distal occlusion of a dominant RCA (ST-segment depression in I and V1-V3,... Figure 4.17 Acute myocardial infarction with ST-segment elevation in II, III and VF and ST-segment depression in V1-V3. This pattern corresponds classically to an infarction involving inferior and posterior walls. Nowadays, this is the pattern of STE-ACS of inferolateral zone evolving to inferolateral infarction due to distal occlusion of a dominant RCA (ST-segment depression in I and V1-V3,...
Figure 5.30 Typical example of inferior myocardial infarction (Qr in II, III and VF) with leftward AQRS. Nevertheless, the left-deviated AQRS (-35°) is not explained by an added superoanterior hemiblock (SAH), but simply by the inferior necrosis, because although the majority of the QRS loop in the frontal plane is above 0°, as it completely rotates in the clockwise sense, a small terminal r (Qr morphology) in II, III and VF is recorded. If an added SAH exists, the first part of the loop would be the same, but would later rotate in the counter-clockwise direction and would generate QS with notches but without the final r wave in inferior leads. Figure 5.30 Typical example of inferior myocardial infarction (Qr in II, III and VF) with leftward AQRS. Nevertheless, the left-deviated AQRS (-35°) is not explained by an added superoanterior hemiblock (SAH), but simply by the inferior necrosis, because although the majority of the QRS loop in the frontal plane is above 0°, as it completely rotates in the clockwise sense, a small terminal r (Qr morphology) in II, III and VF is recorded. If an added SAH exists, the first part of the loop would be the same, but would later rotate in the counter-clockwise direction and would generate QS with notches but without the final r wave in inferior leads.
Figure 8.15 A 48-year-old patient with an acute inferior myocardial infarction due to RCA occlusion after the RV branches with an evident alteration of ST segment (STT in I, STf in III > II and STf in V1-V2). The fibrinolytic... Figure 8.15 A 48-year-old patient with an acute inferior myocardial infarction due to RCA occlusion after the RV branches with an evident alteration of ST segment (STT in I, STf in III > II and STf in V1-V2). The fibrinolytic...
Figure 9.1 Inferior myocardial infarction. (A) The ECG shows Qr in leads Dill and VF and rS in V1. (B) CE-CMR image in a vertical long-axis (sagittal-like) view confirming inferior myocardial infarction as showed by delayed hyperenhancement (arrows). (C-E) Contrast-enhanced... Figure 9.1 Inferior myocardial infarction. (A) The ECG shows Qr in leads Dill and VF and rS in V1. (B) CE-CMR image in a vertical long-axis (sagittal-like) view confirming inferior myocardial infarction as showed by delayed hyperenhancement (arrows). (C-E) Contrast-enhanced...
Bairey CN, Shah PK, Lew AS, Hulse S. Electrocardiographic differentiation of occlusion of the left circumflex versus the right coronary artery as a cause of inferior acute myocardial infarction. Am J Cardiol 1987 60 456. [Pg.310]

Birnbaum Y, Solodky A, Herz I et al. Implications of inferior ST segment depression in anterior acute myocardial infarction electrocardiographic and angiographic correlation. Am Heart J 1994 127 1467. [Pg.311]

Eskola MJ, Nikus KC, Niemela KO, Sclarovsky S. How to use ECG for decision support in the catheterization laboratory cases with inferior ST elevation myocardial infarction. J Electrocardiol 2004 37 257. [Pg.314]

Fiol M, Cygankiewicz I, Bayes-Genis A et al. The value of ECG algorithm based on ups and downs of ST in assessment of a culprit artery in evolving inferior myocardial infarction. Am J Cardiol 2004b 94 709. [Pg.314]

Haraphongse M, Tanomsup S, Jugdutt BI. Inferior ST segment depression during acute anterior myocardial infarction clinical and angiographic correlations. J Am Coll Cardiol 1984 4 467. [Pg.315]

Herz I, Assali AR, Adler Y, Solodky A, Sclarovsky S. New electrocardiographic criteria for predicting either the right or left circumflex artery as the culprit coronary artery in inferior wall acute myocardial infarction. Am J Cardiol... [Pg.315]

Hurd HR Starling MR, Crawford MH, Dlabal PW, O Rouke RA. Comparative accuracy of electrocardiographic and vectorcardiographic criteria for inferior myocardial infarction. Circulation 1981 63 1025-1029. [Pg.316]

Kabakci G, Yildirir A, Yildiran L et al. The diagnostic value of 12-lead electrocardiogram in predicting infarct-related artery and right ventricular involvement in acute inferior myocardial infarction. Arm Noninvasive Electrocardiol 2001 6(3) 229-35. [Pg.316]

Kurum T, Oztekin E, Ozcelik F, Eker H, Ture M, Ozbay G. Predictive value of admission electrocardiogram for multivessel disease in acute anterior and anterio-inferior myocardial infarction. Ann Noninvasive Electrocardiol 2002 7 369. [Pg.317]

Lew AS, Laramee P, Shah PK, Maddahi J, Peter T, Ganz W. Ratio of ST-segment depression in lead V2 to ST-segment elevation in lead aVF in evolving inferior acute myocardial infarction an aid to the early recognition of right ventricular ischemia. Am J Cardiol 1986 57 1047. [Pg.317]

Mavric L, Laputovic L, Matana A et al. Prognostic significance of complete atrioventricular block in patients with acute inferior myocardial infarction with and without right ventricular involvement. Am Heart J 1990 119 823. [Pg.318]

Sadanandan S, Hochman S, Kolodzjez A et al. Clinical and electrocardiographic characteristics of patients with combined anterior and inferior ST segment elevation in the initial ECG during acute myocardial infarction. Am Heart J 2003 146 653. [Pg.321]

Sapin PM, Musselman DR, Dehmer GJ, Cascio WE. Implications of inferior ST segment elevation accompanying anterior wall myocardial infarction for the angiographic morphology of the left anterior descending coronary artery morphology and site of occlusion. Am J Cardiol 1992 69 860. [Pg.321]

Saw J, Davies C, Fung A, Spinelli JJ, Jue J. Value of ST elevation in lead III greater than lead II in inferior wall acute myocardial infarction for predicting in-hospital mortality and diagnosing right ventricular infarction. Am I Cardiol 2001 87 448. [Pg.321]

Starr J, Wagner G, Draffin R, Reed J, Walston A, Behar V. Vectocardiographic criteria for the diagnosis of inferior myocardial infarction. Circulation 1974 59 829. [Pg.322]

Tamura A, Kataoka H, Mikuriya Y, Nasu M. Inferior ST segment depression as a useful marker for identifying proximal left anterior descending artery occlusion during acute anterior myocardial infarction. Eur Heart J 1995a 16 1795. [Pg.322]

TamuraA, KataokaH, Nagase K, MikuriyaY, Nasu M. Clinical significance of inferior ST elevation during acute anterior myocardial infarction. Br Heart J 1995b 74 611. [Pg.322]


See other pages where Myocardial infarction inferior is mentioned: [Pg.455]    [Pg.473]    [Pg.483]    [Pg.508]    [Pg.175]    [Pg.622]    [Pg.1372]    [Pg.306]    [Pg.218]    [Pg.243]    [Pg.245]    [Pg.254]    [Pg.277]    [Pg.283]    [Pg.312]    [Pg.316]   
See also in sourсe #XX -- [ Pg.103 , Pg.138 , Pg.158 , Pg.294 ]




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