Symptomatic treatment nausea

These symptoms are consistent with the pharmacological action and usually appear in the order of nausea, chest pains, blurred vision. Removal from exposure and symptomatic treatment have thus far been followed by rapid abatement of symptoms. 

Cholinesterase inhibitors are the only class of drugs currently approved by most Health Authorities and recommended by professional associations (e.g. Doody et al.. 2001) for the symptomatic treatment of AD. However, ChE-Is intervene at a late stage of the pathophysiological cascade leading to AD (Fig. 7.2) furthermore, their efficacy is limited and they may cause a number of side effects, most frequently nausea, vomiting, diarrhea, anorexia and dizziness. For these reasons, major efforts are being made to alter the biological processes... 

Compliance with awkward regimens such as five times a day may be problematic as usual reference points such as meals are less useful. Mr DM has a combination of regular and when required medication which would make the use of a monitored dosing system (MDS) a challenge. However, symptomatic treatment of nausea, which is usually self-limiting, and constipation are likely to be less of a problem. 

Mild gastrointestinal symptoms have sometimes occurred (nausea, accelerated intestinal transit) but they regress spontaneously or after symptomatic treatment (2,3). 

Five patients with acute accidental poisoning with V. album rapidly developed nausea, vomiting, abdominal pain, hypotension, and bradycardia (26). In four cases the electrocardiogram showed sinus bradycardia and in one there was complete atrioventricular block with an ectopic atrial bradycardia and an intermittent idioventricular rhythm. Symptomatic treatment and/or atropine led to recovery within a few hours. 

Yilmaz E, Ertan B, Deniz T, et al. Histamine 1 receptor antagonist in symptomatic treatment of renal colic accompanied by nausea two birds with one stone Urology 2009 73(l) 32-36. 

Give symptomatic treatment for nausea, vomiting, diarrhoea, pain and sldn symptoms. 

There is no specific treatment for ingestion of ibotenic acid or muscimol rather, treatment is symptomatic and supportive. Anxiety, hysteria, or convulsions can be treated with sedatives, such as diazepam. This should be done cautiously, however, and with the lowest effective dose because animal studies revealed that respiratory arrest may occur. In severe cases, with prolonged nausea, vomiting, or diarrhea, monitoring of fluid and electrolyte status may be required. Recent cases of muscarine poisonings were reported by Benjamin (1992), and Tupalska-Wilczynska et al. (1997). 

GI irritants are the most common toxins in Boletes, particularly in red-spored and yellow-spored species. Muscarine is present in a few species, but too low to be significant. Symptoms are nausea, vomiting, and diarrhea. In more severe cases there may be muscle cramps and circulatory disturbance. Treatment is largely symptomatic, and recovery is usually complete one to two days after ingestion. 

Apart from disease entities, one recognizes a variety of symptoms that makes up a disease, like pain, fever, convulsions, cough, rhinitis, nausea, meteorism, diarrhea, headache, dyspnea, anorexia, insomnia, constipation, etc. All these symptoms could make up a symptom complex or syndrome, or may stand alone by themselves without being part of a disease entity. The term of symptomatic drug treatment usually refers to its use for the alleviation of symptoms, whether as a part of a syndrome or standing alone. 

Chlorpromazine is the prototype molecule of the series of substituted phenothiazine psychotherapeutic compounds. Chlorpromazine is used for the symptomatic management of schizophrenia and active acute psychoses. The drug is also used for the prevention and treatment of nausea and vomiting, and for the relief of restlessness and apprehension before surgery [1,2]. 

Thyrotropin stimulates iodine uptake, and this facilitates the diagnosis and treatment of recurrent disease or metastases in the follow-up of differentiated thyroid cancer. It is used as an alternative to thyroid hormone withdrawal, to avoid symptomatic hypothyroidism (1). Headache and nausea occur in 6-40% of patients after intramuscular administration, but are usually mild and transient (2,3). 

This toxic protein is contained in caster seeds but does not pass into the oil. Similar phytotoxins occur in croton seeds (Crotin) jequirity seeds (Abrin) the bark of the locust tree, Robinia pseudo-acacia (Robin) and in the seeds of some leguminous plants (Phasin). The last is but weakly toxic. Ricin is responsible for the toxic effects on eating castor seeds 5 or 6 of these are fatal to a child, 20 to adults, and 3 or 4 seeds may cause violent gastroenteritis with nausea, headache, persistent vomiting, colic, sometimes bloody diarrhea, thirst, emaciation, and great debility. The symptoms usually do not set in until after several days. More severe intoxications cause small frequent pulse, cold sweat, icterus, and convulsions. Death occurs in 6 to 8 d, from the convulsions or from exhaustion. The fatality rate is about 6%. This low fatality rate is due to the destruction of the poison in the alimentary canal. The treatment would be evacuant and symptomatic. Usually, 3 to 10 d are required to complete recovery. 

Adverse effects. Most patients tolerate oral iron therapy but 10-20% have symptoms that may be attributed to iron, generally gastrointestinal upset. These effects of oral iron include nausea, abdominal pain, and either constipation or diarrhoea. Upper GI effects appear to be dose-related and are best managed by ingestion of the tablet with or after food and/or reduction in the amount of iron content in each dose. This will prolong the necessary period of treatment. Diarrhoea or constipation can usually be treated symptomatically without a change in regimen. 

Different doses of ranitidine (150 mg bd and 300 mg bd for 8 weeks) have been compared in resolving heartburn in 271 patients with gastro-esophageal reflux disease who had been symptomatic after 6 weeks of therapy with ranitidine 150 mg bd (24). Less than 20% of the patients in either group had complete resolution of heartburn at 4 and 8 weeks there was no significant difference in the efficacy between the two treatment groups. At least one adverse event was reported by 38% of the patients in each group. They included sinusitis, nausea, abdominal pain, dyspepsia, constipation, and increased liver enzymes. 

Oral metoclopramide is indicated in the treatment of diabetic gastroparesis (10 mg 30 minutes before each meal and at bedtime for 2 to 8 weeks) and symptomatic gastroesophageal reflux (10 to 15 mg orally up to 4 times daily 30 minutes before each meal and at bedtime for 4 to 12 weeks). Furthermore, parenteral metoclopramide is indicated in prevention of nausea and vomiting associated with emetogenic cancer chemotherapy, for prophylaxis of postoperative nausea and vomiting when nasogastric suction is undesirable, and as a single dose to facilitate small-bowel intubation when the tube does not pass the pylorus with conventional maneuvers. 

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