Sodium acid citrate

Sodium Acid Citrate -do- Any red colour produced is not more intense than that produced by treating in the same manner 4 ml of a 0.005% sol. of oxalic acid (150 ppm). 

Disodium hydrogen citrate Sodium acid citrate CjttjNajO, 144-33-2 236.088 wh pow (w) 149 dec vs HjO... 

Sodium acid citrate 4574 Sulfobromophthalein sodium 9573 Tetraiodoethylene 9903... 

Solutions that contain sodium citrate/citric acid (Shohl s solution and Bicitra) provide 1 mEq/L (1 mmol/L) each of sodium and bicarbonate. Polycitra is a sodium/potassium citrate solution that provides 2 mEq/L (2 mmol/L) of bicarbonate, but contains 1 mEq/L (1 mmol/L) each of sodium and potassium, which can promote hyperkalemia in patients with severe CKD. The citrate portion of these preparations is metabolized in the liver to bicarbonate, while the citric acid portion is metabolized to C02 and water, increasing tolerability compared to sodium bicarbonate. Sodium retention is also decreased with these preparations. However, these products are liquid preparations, which may not be palatable to some patients. Citrate can also promote aluminum toxicity by augmenting aluminum absorption in the GI tract. 

Acid citrate dextrose solution-A (ACD-A) 22.0 g/L sodium citrate (Na3C6H507 ... 

To evaluate the presence of possible interferences, the following metabolites were tested at their physiological concentration bilirubin, sucrose, cholesterol, triglycerides, acetone, urea, uric acid, citric acid, L-ascorbic acid, citrate, pyruvate, haemoglobin, y-globulin, sodium pyruvate, NaCl, KC1, Ca2+ and EDTA. Urea, uric acid, L-ascorbic acid, NaCl, KC1 and Ca2+ generated a slight interference. 

Sodium acid pyrophosphate Sodium citrate Sodium diacetale Sodium gluconate... 

Acid Citrate Dextrose (ACD) Dissolve 2.1 g di-sodium hydrogen citrate and 2.5 g D-glucose in 50 mL of sterile distilled water, fdter (0.2 microns) and autoclave (at the relatively low temperature of 90°C, to prevent degradation of the glucose). Store at 4°C. 

Salts sodium dihydrogeno-citrate, sodium acid phosphate, sodium fumarate. 

I) Whatman No.l, sheet (14 x 6 inches), buffered by dipping in 5% solution of sodium dihydrogen citrate, blotting and drying at 25 for one hour. It can be stored indefinitely. A solvent composed of 4.8 g of citric acid in a mixture of 130 ml of water and 870 ml of n-butanol was used (103). 

Binding of iron by dietary fiber is strongly inhibited by ascorbic acid, citrate, cysteine, EDTA or phytate in concentrations as lew as 100 >uMols/Liter (A3). The inhibitors have the common property of being able to form soluble complexes with iron. The decarbox-ylic amino acids and their amides inhibit binding moderately as do lysine and histidine. Other amino acids either do not interfere with binding of iron fiber or do so only weakly. Calcium (as acetate) and phosphate act as moderate inhibitors. The detergents sodium lauryl sulfonate or cetyltrimethylammonium bromide had no effect on iron binding by fiber (A2). 

In patients with Stage 3 or higher CKD, the use of aUcalinizing salts, such as sodium bicarbonate or citrate/citric acid preparations, is useful to replenish depleted body bicarbonate stores. Sodium bicarbonate tablets are manufactured in 325- and 650-mg strengths (a 650-mg tablet contains 7.7 mEq sodium and 7.7 mEq bicarbonate). Shohl s solution and Bicitra contain 1 mEq/mL of sodium and the equivalent of 1 mEq/mL of bicarbonate as sodium citrate/citric acid. Citrate is metabolized in the liver to bicarbonate, and citric acid is metabolized to CO2 and water. Polycitra, which contains potassium citrate, (1 mEq/mL of sodium, 1 mEq/mL of potassium, and 2 mEq/mL of bicarbonate) should not be used in patients with severe CKD since hyperkalemia may result. 

Alkaloids S% Sodium dihydrogen citrate n-Butanol-acetic acid-HjO (12 3 5) n-Butanol-S% citric acid (9 1) 87 87... 

Methenamine and methenamine mandelate are only effective as urinary antisepties if the pH is about 5.5 or lower, when formaldehyde is released. This is normally achieved by giving urinary acidifiers such as ammonium chloride, ascorbic acid, or sodium acid phosphate. In the case of methenamine hippurate, the acidification of the urine is achieved by the presence of hippuric acid. The concurrent use of substances that raise the urinary pH such as acetazolamide, sodium bicarbonate, potassium or sodium citrate is clearly contraindicated. Potassium citrate mixture BPC has been shown to raise the pH by more than 1 at normal therapeutic doses, thereby making the urine sufficiently alkaline to interfere with the activation of methenamine to formaldehyde. Some antacids (containing magnesium, aluminium or calcium as well as sodium bicarbonate mentioned above) can also cause a significant rise in the pH of the urine. ... 

Ammonium citrate dibasic Ammonium formate Ammonium phosphate Betaine hydrochloride Diethylamine Dipropylenetriamine N-Hydroxysuccinic acid isopropanoiamine Lithium hydroxide N-Methylethanoiamine Sodium acid pyrophosphate Sodium metasilicate Sodium metasiiicate pentahydrate Sodium phosphate dibasic anhydrous Sodium phosphate dibasic dihydrate Trisodium citrate buffer component... 

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